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CD24 and CD49f expressions of E14.5 mouse mammary anlagen cells define putative distribution of earlier embryonic mammary stem cell activities.
CD24 has a role in determining the epithelial phenotype of pancreatic cancer
While no obvious role was found for CD24 in the normal development and maintenance of the dopaminergic nigrostriatal system in mice, it may have a role in mediating the neuroprotective aspects of GDNF in this system.
CD24 cell surface expression may serve as a valuable biomarker in order to identify mammary tumors that will positively respond to targeted IGF1R therapies.
mice negative or positive for CD24 did not differ in terms of tumor initiation and burden in 3 mammary and prostate tumor models tested, except for Apc1572T/+ mice, in which lack of CD24 reduced the mammary tumor burden slightly but significantly.
Loss of CD24 in Mice Leads to Metabolic Dysfunctions and a Reduction in White Adipocyte Tissue.
CD24 controls breast cancer radiation response. Loss of CD24 expression leads to radiation resistance.
Delayed wound-healing in the absence of HSA/CD24 suggests that CD24 plays an important role in this process
Findings indicate that CD24(+) antigen cells play a role in tumor migration and metastasis and support Janus kinase 2 protein (JAK2) as a therapeutic target in ovarian cancer.
CD24 is a conserved marker for tracking divergent states in both reprogramming and standard pluripotent culture.
CD24 in non-immune cells might be crucialfor the guidance and recruitment of leukocytes.
CD24 expression negatively regulates the NF-kappaB pathway following experimental traumatic brain injury.
the deletion of CD24 in an HSP-driven model of autoimmunity led to the unexpected development of regulatory T cell and MDSC populations that augmented immune tolerance.
CD24 is shown in vitro and in vivo as being an important oncogene in the gut, and one that plays a critical role in the initiation and progression of carcinogenesis.
Siglec-G-CD24 axis, controls the severity of GVHD and suggest that enhancing this interaction may represent a novel strategy for mitigating GVHD.
CD24 was shown to regulate CD8(+) T cell activation through HMGB1-mediated engagement of T cell RAGE.
CD24 has a role in urothelial tumorigenesis and metastasis in male mice and is androgen regulated in mice and humans
CD24 is expressed in gastric parietal cells, where it modulates gastric responses to H. felis and gamma-radiation. CD24 also regulates susceptibility to apoptosis in the distal murine gastrointestinal tract.
CD24-based sorting in wild-type mice isolates a colonic epithelial fraction highly enriched in actively cycling and quiescent Lgr5 positive putative colonic epithelial stem cells.
Results suggest that LKB1 inactivation in the context of RAS activation facilitates metastasis by inducing an SFK-dependent expansion of a prometastatic, CD24(+) tumor subpopulation.
May have a specific role to play in early thymocyte development.
M1/69-J11D heat stable antigen
, X62 heat stable antigen
, cluster of differentiation 24
, heat stable antigen
, lymphocyte antigen 52
, signal transducer CD24