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Four SNPs in IFNGR2 (show IFNGR2 Proteins), IL12RB1 (show IL12RB1 Proteins), IL12RB2 (show IL12RB2 Proteins), and IL23R were found to be associated with the MAP infection status of the resource population.
these findings suggest that the variants +2199 A/C IL-23R and -197 G/A IL-17A (show IL17A Proteins) could contribute to rheumatoid arthritis development in the studied population
Data show that interleukin-23 receptor (IL-23R) single nucleotide polymorphism (SNP) rs11465817 contributes to the risk of recurrent oral ulceration (ROU) in Chinese.
Findings indicate that IL17A (show IL17A Proteins) -197 G/A and IL23R H3Q are not associated with susceptibility to MM. However, IL-17 (show IL17A Proteins) and IL-23R polymorphisms may affect severity, bone lesions, and extra-medullary disease in patients with MM.
This study provides evidence for three alcohol-induced ONFH susceptibility genes (NOS3 (show NANOS3 Proteins), ABCB1 (show ABCB1 Proteins) and IL23R) in Chinese males and polymorphisms of them may be associated with alcohol-induced ONFH risk.
genetic association studies in population in southwest China: Data suggest that SNPs in STAT4 (show STAT4 Proteins) (rs7574865), IL23R (rs11209032), and STAT3 (show STAT3 Proteins) (rs744166) are associated with occurrence, severity, and immunosuppressive therapy outcomes of aplastic anemia in the population studied. (STAT (show STAT1 Proteins) = signal transducer and activator of transcription (show STAT1 Proteins)) [article includes Meta-Analysis]
Study provides a comprehensive examination of the available evidence for the association between polymorphisms in the IL-23R gene and ulcerative colitis (UC). The meta-analysis suggests that IL-23R gene polymorphisms are associated with UC susceptibility, especially in Caucasians.
Data indicate that the interleukin-23 receptor (IL-23R) SNPs rs11209026, p.Arg381Gln; rs41313262 p.Val362Ile were not associated with susceptibility to inflammatory bowel disease (IBD) in Chinese Han population.
Interleukin-23 receptor cytokine-binding homology region balances the ratio of Th17/Th9/Treg cells in collagen-induced arthritis.
our study provides the evidence that functional IL-23R rs1884444 G/T and IL-17F (show IL17F Proteins) rs763780 A/G polymorphisms may be a new genetic susceptibility factor to SLE, especially in the Polish population.
IL23R rs10889677 and IL17A (show IL17A Proteins) rs2275913 were not associated with the susceptibility to Necrotizing enterocolitis (NEC). In conclusion, data suggest that a variant of IL17F (show IL17F Proteins) (rs763780) may contribute to the development of NEC.
Results suggest that the use of vectors carrying Soluble Interleukin-23 Receptor (sIL-23R) to block the interleukin-23/IL-23R interaction may be a new therapeutic strategy for the treatment of multiple sclerosis.
RBPJ (show RBPJ Proteins) binds and trans-activates the Il23r promoter and induces IL-23R expression and represses anti-inflammatory IL-10 (show IL10 Proteins) production in Th17 cells.
Epithelial IL-23R signaling enables protective IL-22 (show IL22 Proteins) responses in experimental colitis.
Study highlights the importance of IL-23R expression level and the instability of Foxp3 (show FOXP3 Proteins)+ regulatory T cells in the development of inflammatory bowel diseases.
Differential splicing generates antagonistic soluble IL-23R (sIL-23R) variants, which might limit IL-23-mediated immune responses. Here, ectodomain shedding of IL-23R was identified as an alternative pathway for the generation of sIL-23R.
Estrogen and progesterone decrease let-7f microRNA expression and increase IL-23/IL-23 (show IL23A Proteins) receptor signaling and IL-17A (show IL17A Proteins) production in patients with severe asthma.
Data show the contribution of IL-23/IL-23 (show IL23A Proteins) receptor and IL-7/IL-7 (show IL7 Proteins) receptor signaling in Th17 and Th1 (show HAND1 Proteins) cell dynamics during experimental autoimmune encephalomyelitis (EAE).
Study provides evidence that IL-23 (show IL23A Proteins)/IL-23R plays a critical role in brain ischemic injury
IL-23 (show IL23A Proteins) plus T-cell receptor signalling results in significant upregulation of IL-23 receptor expressed predominantly on CD4 (show CD4 Proteins)(hi)CD8 (show CD8A Proteins)(hi)CD3 (show CD3E Proteins)(+)alphabetaTCR(+) thymocytes, and leads to RORgammat-dependent apoptosis.
Homeostatic IL-23 receptor signaling limits Th17 response through IL-22 (show IL22 Proteins)-mediated containment of commensal microbiota.
The protein encoded by this gene is a subunit of the receptor for IL23A/IL23. This protein pairs with the receptor molecule IL12RB1/IL12Rbeta1, and both are required for IL23A signaling. This protein associates constitutively with Janus kinase 2 (JAK2), and also binds to transcription activator STAT3 in a ligand-dependent manner.
, interleukin 23 receptor
, interleukin-23 receptor-like
, IL-23 receptor
, interleukin 23 receptor-like