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anti-Human PLK3 Antibodies:
anti-Mouse (Murine) PLK3 Antibodies:
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Human Monoclonal PLK3 Primary Antibody for IP, WB - ABIN967544
Golsteyn, Schultz, Bartek, Ziemiecki, Ried, Nigg: Cell cycle analysis and chromosomal localization of human Plk1, a putative homologue of the mitotic kinases Drosophila polo and Saccharomyces cerevisiae Cdc5. in Journal of cell science 1994
Show all 4 Pubmed References
Human Polyclonal PLK3 Primary Antibody for ELISA, IHC - ABIN4346283
Wang, Beauchemin, Bertrand: Bcl-xL phosphorylation at Ser49 by polo kinase 3 during cell cycle progression and checkpoints. in Cellular signalling 2011
Human Polyclonal PLK3 Primary Antibody for WB - ABIN392602
Zhang, Shi, Paddon, Hampong, Dai, Pelech: B23/nucleophosmin serine 4 phosphorylation mediates mitotic functions of polo-like kinase 1. in The Journal of biological chemistry 2004
Data indicate that elevated levels of Polo-like kinase 3 (Plk3)and pT273 caspase-8 (show CASP8 Antibodies) are correlated with favorable clinical outcome in patients with anal squamous cell carcinoma (anal SCC (show CYP11A1 Antibodies)) treated with concomitant chemoradiotherapy (CRT (show SLC6A8 Antibodies)).
Knockout (KO) or knockdown of caspase-8 (show CASP8 Antibodies), CD95 (show FAS Antibodies) or FADD (show FADD Antibodies) prevents activation of Plk3 upon CD95 (show FAS Antibodies) stimulation, suggesting a requirement of a functional death-inducing signaling complex for Plk3 activation.
study revealed an interesting mutual regulation between Plk3 and SIAH2 (show SIAH2 Antibodies) and uncovered a regulatory network that functions to fine-tune the cellular hypoxic response.
the differential effects of hypoxic stress on Plk3 activity in Human Limbal Stem and HCE (show RNGTT Antibodies) cells. Instead of apoptosis, hypoxic stress suppresses Plk3 activity to protect limbal stem cells from death and to allow the process of Human Limbal Stem cell differentiation.
Data suggest that HOXA-AS2 (show ARSA Antibodies) could be an oncogene (show RAB1A Antibodies) for gastric cancer partly through suppressing P21 (show CDKN1A Antibodies), PLK3, and DDIT3 (show DDIT3 Antibodies) expression.
Plk3 is involved in the inhibition of cell proliferation and tumorigenesis, which may occur via interactions with p21 (show CDKN1A Antibodies).
PLK3 predominantly is expressed in Cholangiocarcinomas (CCA (show FBN2 Antibodies)) cells and that high PLK3 expression correlates with prolonged overall survival.
The role of Plk3 in oncogenesis: the aberrant expression of Plk3 was found in different types of tumors.
hyperosmotic stress-activated Plk3 elicited gammaH2AX (show H2AFX Antibodies).
Plk3 binds to CtIP (show RBBP8 Antibodies) phosphorylated at S327 via its Polo (show PLK1 Antibodies) box domains, which is necessary for robust damage-induced CtIP (show RBBP8 Antibodies) phosphorylation at S327 and subsequent CtIP (show RBBP8 Antibodies) phosphorylation at T847.
Polo-Like Kinase 3 Appears Dispensable for Normal Retinal Development Despite Robust Embryonic Expression
Calcium- and integrin-binding protein 1 (show CIB1 Antibodies) is involved in regulating endomitosis, perhaps through its interaction with Plk3
Plk3 functions as an essential component of the hypoxia regulatory pathway by direct phosphorylation of HIF-1alpha (show HIF1A Antibodies)
Plk3 as a new player in the regulation of the PI3K/PDK1 (show PDPK1 Antibodies)/Akt (show AKT1 Antibodies) signaling axis by phosphorylation and stabilization of PTEN (show PTEN Antibodies).
Data demonstrate that FGF-inducible kinase (Fnk) expression levels in transfected cells can be regulated by nuclear-cytoplasmic trafficking, ubiquitination, and proteosome-dependent degradation.
Plk3 localizes to the nucleolus and is involved in regulation of the G1/S phase transition.
Ectopic expression of the Plk3-kinase domain (Plk3-KD), but not its Polo (show PLK1 Antibodies)-box domain or a Plk3-KD mutant, suppressed the nuclear accumulation of HIF-1 alpha (show HIF1A Antibodies) induced by nickel or cobalt ions
The specificity of TTP (show ZFP36 Antibodies) for promoting the degradation of Plk3 was demonstrated by the unaltered decay of Plk3 mRNA in cell
Cytokine-inducible kinase is a putative serine/threonine kinase. CNK contains both a catalytic domain and a putative regulatory domain. It may play a role in regulation of cell cycle progression and tumorigenesis.
, serine/threonine-protein kinase PLK3
, FGF-inducible kinase
, polo-like kinase 3
, serine/threonine-protein kinase PLK3-like
, cytokine-inducible serine/threonine-protein kinase
, proliferation-related kinase
, cytokine inducible kinase
, serine-threonine kinase