Browse our anti-Myogenic Differentiation 1 (MYOD1) Antibodies

Horse (Equine) Myogenic Differentiation 1 (MYOD1) interaction partners

1. SRF and its cofactor MYOCD (show MYOCD Antibodies) likely contribute to the hypertrophy of peripheral airway smooth muscle observed in equine asthmatic airways, while the remodeling of the central airways is more static or involves different transcription factors.

2. Equine primary fibroblasts were transformed by lentiviral transduction of equine myogenic differentiation 1 into fusion-competent myoblasts.

Human Myogenic Differentiation 1 (MYOD1) interaction partners

1. We address this paradox using basic helix-loop-helix (bHLH) transcription factors ASCL1 (show ASCL1 Antibodies), ASCL2 (show ASCL2 Antibodies), and MYOD1, crucial mediators of lineage specification..Although the ASCL factors and MYOD1 have some distinct DNA motif preference, it is not sufficient to explain the extent of the differential binding. All three factors can bind inaccessible chromatin and induce changes in chromatin accessibility and H3K27ac

2. ACL (show ACLY Antibodies) regulates the net amount of acetyl groups available, leading to alterations in acetylation of H3(K9/14) and H3(K27 (show KRT27 Antibodies)) at the MYOD locus, thus increasing MYOD expression.

3. we found that MYOD transcription factor can upregulate miR (show MLXIP Antibodies)-223 expression by binding to an E-box region of the gga-miR (show MLXIP Antibodies)-223 gene promoter during avian myoblast differentiation. IGF2 and ZEB1 (show ZEB1 Antibodies) are two target genes of miR (show MLXIP Antibodies)-223

4. A high extent more than 25% of BRAF (show BRAF Antibodies)(V600E) alleles may be associated with disease outcome in PTC (show F9 Antibodies) patients.

5. we present the first report of MYOD1 (L122R) mutation in the largest cohort of 49 rhabdomyosarcomas reported so far, that are associated with a relatively aggressive clinical course

6. Analysis of human rhabdomyosarcoma revealed that MYF5 (show MYF5 Antibodies) and MYOD are mutually-exclusively expressed and each is required for sustained tumor growth.

7. Cell transdifferentiation of primary skin fibroblasts by forced expression of myogenic transcription factor MyoD was performed by quantitative analyses of gene expression and chromatin accessibility profiles.

8. The results strongly suggest that the combination of MYCL plus MYOD1 may promote direct conversion of human fibroblasts into functional myoblasts that could potentially be used for regenerative therapy for muscle diseases and congenital muscle defects.

9. Analysis of the chromatin status of Cdkn1c (show CDKN1C Antibodies) promoter and KvDMR1 in unresponsive compared to responsive cell types showed that their differential responsiveness to the MyoD-dependent induction of the gene does not involve just their methylation status but, rather, the differential H3 lysine 9 dimethylation at KvDMR1.

10. Data show that MeCP2 promotes gastric cancer (GC) cell proliferation via FOXF1 (show FOXF1 Antibodies)-mediated Wnt5a (show WNT5A Antibodies)/beta-Catenin (show CTNNB1 Antibodies) signaling pathway, and suppresses GC cell apoptosis through MYOD1-mediated Caspase-3 (show CASP3 Antibodies) signaling pathway.

Mouse (Murine) Myogenic Differentiation 1 (MYOD1) interaction partners

1. Linc-RAM (show FAM103A1 Antibodies) regulates expression of myogenic genes by directly binding MyoD, which in turn promotes the assembly of the MyoD-Baf60c (show SMARCD3 Antibodies)-Brg1 (show SMARCA4 Antibodies) complex on the regulatory elements of target genes.

2. ACL (show APOC4 Antibodies) regulates the net amount of acetyl groups available, leading to alterations in acetylation of H3(K9/14) and H3(K27 (show KRT27 Antibodies)) at the MYOD locus, thus increasing MYOD expression.

3. Studies indicate MyoD displays function to regulate determination of skeletal muscle progenitors [Review].

4. Muscle regulatory transcription factor MyoD regulates the expression of the pro-apoptotic Bcl2 (show BCL2 Antibodies) family member PUMA (show BBC3 Antibodies) by binding to the promoter region of PUMA (show BBC3 Antibodies). The increase in MyoD binding to the PUMA (show BBC3 Antibodies) promoter as a consequence of culture in differentiation media (DM) is diminished in myoblasts silenced for MyoD expression. In myoblasts silenced for MyoD expression, p53 (show TP53 Antibodies) binding to the PUMA (show BBC3 Antibodies) promoter is diminished in response

5. our results indicate that HDAC11 (show HDAC11 Antibodies) would suppress myoblast differentiation via regulation of MyoD-dependent transcription. These findings suggest that HDAC11 (show HDAC11 Antibodies) is a novel critical target for controlling myoblast differentiation.

6. The data showed that Mettl3 (show METTL3 Antibodies) is required for MyoD mRNA expression in proliferative myoblasts.

7. Gm7325, as a novel MyoD-target gene, is specifically induced in activated satellite cells, and may have an important role in skeletal myogenesis.

8. The ELF (show SPTBN1 Antibodies)-EMFs did not affect C2C12 myoblast viability or proliferation rate. Conversely, at ELF (show SPTBN1 Antibodies)-EMF intensity in the mT range, the myogenic process was accelerated, through increased expression of MyoD, myogenin (show MYOG Antibodies), and connexin 43 (show GJA1 Antibodies)

9. In this study, we identified ubiquitin-specific protease 4 (USP4 (show USP4 Antibodies)), one of deubiquitinating enzymes, as a suppressor of MRFs by demonstrating that a knockdown of USP4 (show USP4 Antibodies) enhances myogenesis by controlling MyoD and the level of myogenesis marker proteins in C2C12 cells... we propose that USP4 (show USP4 Antibodies) is a key player in myogenic differentiation; it controls myogenic regulatory factors in a catalytic-independent manner

10. MyoD regulates the oxidative metabolic capacity of adult skeletal muscle;ChIP-seq analysis identified MyoD binding on the PGC (show PGC Antibodies)-1b, but not PGC (show PGC Antibodies)-1a, gene locus;MyoD cooperates with alternative NF-kappaB (show NFKB1 Antibodies) to regulate PGC (show PGC Antibodies)-1b transcription; MyoD and RelB (show RELB Antibodies) co-occupy many other genes involved in aerobic respiration

Pig (Porcine) Myogenic Differentiation 1 (MYOD1) interaction partners

1. an Enhancer box and a binding site for a cooperative co-activator of MyoD are present in the promoter region of porcine PPARgamma (show PPARG Antibodies).

2. Of the eight adult pig tissue types that were tested, the expression of Myf5 (show MYF5 Antibodies) and MyoD1 was highest in the muscle tissue.

3. Single nucleotide polymorphisms in the MYOD1 and GDF8 (show MSTN Antibodies) genes are associated with genetic transcription during myogenesis in pigs.

4. Therefore, this study demonstrated that the different regulatory adipogenic roles of MSTN (show MSTN Antibodies) in ADSCs and MSCs act by differentially regulating PPARgamma (show PPARG Antibodies) and MyoD expression.

5. Therefore, the g.489C>T and g.1264C>A SNPs in MYOD1 may be meaningful DNA markers that can be used for improving important porcine economic traits.

6. The total expression profile of MyoD and Pax7 (show PAX7 Antibodies) genes suggests that higher muscularity in Pietrain pigs is associated with the presence of a greater number of active satellite stem cells compared to other breeds.

7. Exons and promoters are amplified and sequenced in the 5'UTR (show UTS2R Antibodies) region of this gene.

8. Relative MYOD1 expression was not different, but MYOG (show MYOG Antibodies) expression was higher in the (ligated-tube)crowded group embryos.

9. MYOD1 intron 1 DdeI polymorphism was not significantly associated with any meat quality traits tested

Cow (Bovine) Myogenic Differentiation 1 (MYOD1) interaction partners

1. Transcript abundance for the muscle regulatory gene MYOD1 was lower in animals with more tender beef.

2. Bos taurus MYF5 (show MYF5 Antibodies) activates MYF5 (show MYF5 Antibodies) and MYOD1 expression in cultured fibroblasts.

3. results suggest that MyoD and Myf5 (show MYF5 Antibodies) influence the MyHC (show MYH13 Antibodies) isoform expression, although the effects are not decisive in specifying the phenotypes of adult muscles

4. n conclusion, hypoxia stimulates the proliferation of satellite cells and promotes their myogenic differentiation with MyoD playing an important role

Zebrafish Myogenic Differentiation 1 (MYOD1) interaction partners

1. Irxl1/Mkx (show MKX Antibodies) can repress myoD expression through direct binding to its promoter and may thus play a negative regulatory role in muscle differentiation.

2. Myod in turn up-regulates cdkn1c (show CDKN1C Antibodies), thereby providing a positive feedback loop that switches myogenic cells to terminal differentiation

3. Myf5 (show MYF5 Antibodies) and Myod function independently during cranial myogenesis.