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Rat (Rattus) NOX1 ELISA Kit for Sandwich ELISA - ABIN431571
El-Naga: Pre-treatment with cardamonin protects against cisplatin-induced nephrotoxicity in rats: impact on NOX-1, inflammation and apoptosis. in Toxicology and applied pharmacology 2013
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Human NOX1 ELISA Kit for Sandwich ELISA - ABIN417371
Abdelmageed, El-Naga, El-Demerdash, Elmazar: Indole-3- carbinol enhances sorafenib cytotoxicity in hepatocellular carcinoma cells: A mechanistic study. in Scientific reports 2016
Rat (Rattus) NOX1 ELISA Kit for Sandwich ELISA - ABIN1081786
El-Naga, Mahran: Indole-3-carbinol protects against cisplatin-induced acute nephrotoxicity: role of calcitonin gene-related peptide and insulin-like growth factor-1. in Scientific reports 2016
the anti-proliferative and pro-apoptotic effect of cambogin on breast adenocarcinoma is mediated via inducing NOX1-dependent ROS (show ROS1 ELISA Kits) production and the dissociation of ASK1 (show MAP3K5 ELISA Kits) and Trx1 (show MLL ELISA Kits)
Transcriptional regulation of NOX genes expression in human breast adenocarcinoma cells is modulated by adaptor protein CIN85 (show SH3KBP1 ELISA Kits).
the transition-state substrate analogue inhibitor of Prdx6 phospholipase A2 activity (MJ-33) was shown to suppress Nox1 activity, suggesting Nox1 activity is regulated by the phospholipase activity of Prdx6. Finally, wild type Prdx6, but not lipase or peroxidase mutant forms, supports Nox1-mediated cell migration in the HCT-116 colon epithelial cell model of wound closure
Cells redox environment mediated by NOX1 isozymes activation down-regulates BRCA1 expression and promotes DNA homologous recombination repair in cancer.
LRRC8A (show LRRC8A ELISA Kits) channels support TNFalpha (show TNF ELISA Kits)-induced superoxide production by Nox1 which is required for receptor endocytosis.
These results are consistent with the hypothesis that antioxidants or NOX1/4 inhibition may be useful in blocking profibrotic effects of TGFbeta (show TGFB1 ELISA Kits) on dermal and gingival fibroblasts and warrant consideration for further development as potential antifibrotic agents
We demonstrated that rapid deletion of p22phox (show CYBA ELISA Kits) is possible and that the activity of Nox1 and Nox4 (show NOX4 ELISA Kits) but not Nox5 (show NOX5 ELISA Kits) exclusively depends on p22phox (show CYBA ELISA Kits).
PAK4 (show PAK4 ELISA Kits) downregulation decreased PPARgamma (show PPARG ELISA Kits)-mediated Nox1 expression and suppressed EMT (show ITK ELISA Kits) in IR-treated glioma cells.
Nox1-SHH (show SHH ELISA Kits)-Grem1 (show GREM1 ELISA Kits) signaling axis in pulmonary vascular endothelium that is likely to contribute to Pulmonary hypertension.
5-HT1B receptor-dependent cellular Src (show SRC ELISA Kits)-related kinase-Nox1-pathways contribute to vascular remodeling in pulmonary arterial hypertension.
our data identify EBP50 (show SLC9A3R1 ELISA Kits) as a previously unidentified regulator of Nox1 and support that it promotes Nox1 activity by binding p47(phox) This interaction is pivotal for agonist-induced smooth muscle ROS (show ROS1 ELISA Kits), hypertrophy, and vasoconstriction and has implications for ROS (show ROS1 ELISA Kits)-mediated physiological and pathophysiological processes.
We examined the pattern of NOX expression in spinal cords of patients and mouse models of ALS and analyzed the impact of genetic deletion of the NOX1 and 2 isoforms as well as pharmacological NOX inhibition in the SOD1 (show SOD1 ELISA Kits)(G93A) ALS mouse model.In contrast to previous publications, survival of SOD1 (show SOD1 ELISA Kits)(G93A) mice was not modified upon breeding with constitutive NOX1 and NOX2 (show CYBB ELISA Kits) deficient mice.
Both Nox1 and Duox2 (show DUOX1 ELISA Kits) induce exfoliation of crypt epithelium, but only Nox1 induces apoptosis. NOX1 and DUOX2 (show DUOX1 ELISA Kits) may be potential therapeutic targets for treating ileocolitis in human patients suffering inflammatory bowel disease (IBD).
we used the Ang II (show AGT ELISA Kits) infused hph-1 (show EGLN2 ELISA Kits) mice to examine the roles of NOX isoforms in the development of AAA (show AAAS ELISA Kits). We generated double mutants of hph-1 (show EGLN2 ELISA Kits)-NOX1, hph-1 (show EGLN2 ELISA Kits)-NOX2 (show CYBB ELISA Kits), hph-1 (show EGLN2 ELISA Kits)-p47phox, and hph-1 (show EGLN2 ELISA Kits)-NOX4 (show NOX4 ELISA Kits)
C-kit (show KIT ELISA Kits)-positive hematopoietic stem/progenitor cells expressed significantly higher of Nox1 and catalase (show CAT ELISA Kits), but less of lactoperoxidase (show LPO ELISA Kits) than in matured mononuclear cells.
Nox1 deletion reduces oxidant load and restores microvascular health in obese mice.
Data suggests that ROS (show ROS1 ELISA Kits) produced during primitive endoderm differentiation is dependent in part on increased NOX1 and NOX4 (show NOX4 ELISA Kits) levels, which is under the control of GATA6 (show GATA6 ELISA Kits). Furthermore, these results suggest that the combined activity of multiple NOX proteins is necessary for the differentiation of F9 cells to primitive endoderm.
Gp91phox (show CYBB ELISA Kits) NADPH oxidase modulates litter size by up-regulating mucin1 (show MUC1 ELISA Kits) expression in the uterus of mice.
NOX4 (show NOX4 ELISA Kits)- and NOX1-derived ROS (show ROS1 ELISA Kits) contribute to atherosclerosis in the aortic sinus of diabetic ApoE (show APOE ELISA Kits) knockout mice.
NADPH oxidase plays an important role in proMMP-2 expression and activation and MMP-2 (show MMP2 ELISA Kits) mediated SMC (show DYM ELISA Kits) proliferation occurs through the involvement of Spm (show NPC1 ELISA Kits)-Cer (show CBLN1 ELISA Kits)-S1P (show MBTPS1 ELISA Kits) signaling axis under ANG II (show AGT ELISA Kits) stimulation of PASMCs
Differential Roles of Protein Complexes NOX1-NOXO1 and NOX2-p47phox in Mediating Endothelial Redox Responses to Oscillatory and Unidirectional Laminar Shear Stress.
The current study was designed to determine mechanisms underlying 20-hydroxyeicosatetraenoic acid -stimulated nitric oxide (NO) release, and particularly the role of NADPH oxidase, reactive oxygen species, and PI3-kinase (show PIK3CA ELISA Kits) in stimulated NO release.
NEP (show MME ELISA Kits) expression is down-regulated in vascular endothelial cells by physiological laminar shear, possibly via a mechanotransduction mechanism involving NADPH oxidase-induced reactive oxygen species production.
The nox1 expression in zebrafish during early nervous system development from 12 to 48 hours post fertilization.
Over inhibition of the NADPH oxidase by the NADPH (show NQO1 ELISA Kits) Inhibitor DPI (show DSP ELISA Kits) may reduce the cell even the tissue in the progress of healing after the injury, in zebrafish liver cells.
This gene encodes a member of the NADPH oxidase family of enzymes responsible for the catalytic one-electron transfer of oxygen to generate superoxide or hydrogen peroxide. Alternatively spliced transcript variants encoding multiple isoforms have been observed for this gene.
NADH/NADPH mitogenic oxidase subunit P65-MOX
, NADPH oxidase homolog-1
, mitogenic oxidase (pyridine nucleotide-dependent superoxide-generating)
, mitogenic oxidase 1
, NADH/NADPH mitogenic oxidase subunit p65-mox
, GP91 phox homolog
, NADPH oxidase 1 alpha
, NADPH oxidase-1
, predicted NADPH oxidase-1
, NADPH oxidase 1