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anti-Human VEGF Antibodies:
anti-Mouse (Murine) VEGF Antibodies:
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Human Polyclonal VEGF Primary Antibody for IHC (p), WB - ABIN3044494
Jiang, Han, Li, Yang, Liu: Carboxymethyl chitosan represses tumor angiogenesis in vitro and in vivo. in Carbohydrate polymers 2015
Show all 62 Pubmed References
Human Polyclonal VEGF Primary Antibody for IHC (p), WB - ABIN3042324
Song, Yue, Li, Li, Zhao, Zhang: Study of the mechanism of sonodynamic therapy in a rat glioma model. in OncoTargets and therapy 2014
Show all 61 Pubmed References
Polyclonal VEGF Primary Antibody for IHC (p), WB - ABIN540566
Jin, Zhu, Sun, Mao, Xie, Greenberg: Vascular endothelial growth factor (VEGF) stimulates neurogenesis in vitro and in vivo. in Proceedings of the National Academy of Sciences of the United States of America 2002
Show all 4 Pubmed References
Guinea Pig Polyclonal VEGF Primary Antibody for IF (p), IHC (p) - ABIN707186
Wang, Liao, Wang, Deng, Yu: Transplantation of bone marrow stromal cells overexpressing human vascular endothelial growth factor 165 enhances tissue repair in a rat model of radiation-induced injury. in Chinese medical journal 2014
Show all 3 Pubmed References
Human Monoclonal VEGF Primary Antibody for Neut, ELISA - ABIN1003427
Kim, Li, Houck, Winer, Ferrara: The vascular endothelial growth factor proteins: identification of biologically relevant regions by neutralizing monoclonal antibodies. in Growth factors (Chur, Switzerland) 1992
Show all 2 Pubmed References
Human Polyclonal VEGF Primary Antibody for ELISA, WB - ABIN541854
Gu, Brännström, Jiang, Bergh, Wester: Vascular endothelial growth factor-A and -C protein up-regulation and early angiogenesis in a rat photothrombotic ring stroke model with spontaneous reperfusion. in Acta neuropathologica 2001
Show all 2 Pubmed References
Human Monoclonal VEGF Primary Antibody for FACS - ABIN4898939
Lee, Myers, Kim: Vascular endothelial growth factor drives autocrine epithelial cell proliferation and survival in chronic rhinosinusitis with nasal polyposis. in American journal of respiratory and critical care medicine 2009
Human Monoclonal VEGF Primary Antibody for CyTOF, FACS - ABIN4900874
Banerjee, Lin, Skinner, Schiffhauer, Peacock, Hicks, Redmond, Morrow, Huston, Shayne, Langstein, Miller-Graziano, Strickland, ODonoghue, De: Heat shock protein 27 differentiates tolerogenic macrophages that may support human breast cancer progression. in Cancer research 2011
Human Polyclonal VEGF Primary Antibody for IF (p), IHC (p) - ABIN675893
Ryzhov, Goldstein, Novitskiy, Blackburn, Biaggioni, Feoktistov: Role of A2B adenosine receptors in regulation of paracrine functions of stem cell antigen 1-positive cardiac stromal cells. in The Journal of pharmacology and experimental therapeutics 2012
Human Monoclonal VEGF Primary Antibody for PLA, ELISA - ABIN521322
Arai, Kawachi, Setiawan, Kobayashi: Hypoxia-selective growth inhibition of cancer cells by furospinosulin-1, a furanosesterterpene isolated from an Indonesian marine sponge. in ChemMedChem 2010
Vegfa signaling governs the formation of diverse arteries/veins by distinct cellular mechanisms in vertebrate vasculatures
vascular endothelial growth factor (show VEGF Antibodies) and Hedgehog (show SHH Antibodies) pathways have roles in the development of the superficial system of ocular vessel patterning in zebrafish
miR (show MYLIP Antibodies)-9 modulation of neuronal VEGF-A controls brain angiogenesis in vivo.
Timelapse analysis of individual cardiomyocyte trajectories reveals misdirected cells in zebrafish pdgfra (show PDGFRA Antibodies) mutants, suggesting that PDGF (show PDGFA Antibodies) signaling steers cardiomyocytes toward the midline during cardiac fusion. Intriguingly, the ligand pdgfaa is expressed in the endoderm medial to the pdgfra (show PDGFRA Antibodies)-expressing myocardial precursors.
This study demonstrates the involvement of VEGF signaling in regulating sustained liver injuries after acute alcohol exposure.
we determined that radial glia control this process via the Vegf decoy receptor sFlt1 (show FLT1 Antibodies): sflt1 (show FLT1 Antibodies) mutants exhibit the venous over-sprouting observed in radial glia-ablated larvae, and sFlt1 (show FLT1 Antibodies) overexpression rescues it. Genetic mosaic analyses show that sFlt1 (show FLT1 Antibodies) function in trunk endothelial cells can limit their over-sprouting.
High VEGFA expression is associated with Ectopic Proliferation and Retinal Dysplasia in Zebrafish Optic Pathway Tumors.
Rspo1 (show RSPO1 Antibodies) is required for hematopoietic stem cell specification through control of parallel signaling pathways controlling HSC (show FUT1 Antibodies) specification: Wnt16 (show WNT16 Antibodies)/DeltaC/DeltaD and Vegfa/Tgfbeta1 (show TGFB1 Antibodies)
Tmem2 (show TMEM2 Antibodies) regulates hyaluronic acid turnover to promote normal Vegf signaling during developmental angiogenesis.
low levels of Vegf signaling promote overall vascular endothelial differentiation and cell survival by upregulating etv2 (show ETV2 Antibodies) expression, while high levels of Vegf signaling promote arterial and inhibit venous specification.
VEGFA produced in the somites is required to initiate adult haemangioblast programming in the adjacent dorsal lateral plate mesoderm.
sequential, isoform-specific VEGFA signaling successively induces the endothelial, arterial, and hematopoietic stem cell programs in the dorsal aorta.
VEGF induces stromal cell migration or recruitment that are required for blood vessel formation.
increased VEGF(170) levels disturb Hand-1 (show HAND1 Antibodies) expression in the region required for normal heart morphogenesis
data for the first time demonstrate a calpain/PTP1B/VEGFR2 negative feedback loop in the regulation of VEGF-induced angiogenesis. Modulation of local PTP1B and/or calpain activities may prove beneficial in the treatment of impaired wound healing in diabetes.
Studied the effect of cyclic uniaxial stretch (20%, 1 Hz), with and without the stimulation of vascular endothelial growth factor (VEGF (show VEGF Antibodies)), on sprouting angiogenesis by employing a stretchable three-dimensional cell culture model.
Study shows that VEGFA mRNA in mammalian endothelial cells undergoes programmed translational readthrough (PTR (show PTCHD3 Antibodies)) generating VEGF-Ax, an isoform containing a unique 22-amino-acid C terminus extension.
Data indicate that superoxide dismutase (SOD) inhibited high glucose (HG)-induced expression of uPAR and VEGF in bovine retinal microvascular endothelial cell (REC).
Exposure of endothelial cells to VEGF, high glucose, or hydrogen peroxide up-regulated the XBP1 (show XBP1 Antibodies)/IRE1 alpha (show ERN1 Antibodies) and ATF6 (show ATF6 Antibodies) arms of the unfolded protein response compared with untreated cells.
Microvascular endothelial cells are more susceptible than aortic cells to advanced glycation end products-enhanced permeability and that AGE-enhanced permeability is dependent on VEGF expression induced by reactive oxygen species.
VEGF supports germ cell survival and sperm production in bulls.
analysis of polymorphisms of bovine VEGF gene and their associations with growth traits in Chinese cattle
VEGF mRNA expression at estrus was higher than at the early I, early II and late luteal stages (P<0.05), whereas VEGF protein content was greatest at the early I luteal stage and decreased thereafter
Alterations in the expression of VEGF-A and bFGF (show FGF2 Antibodies) systems suggest that angiogenic factors are involved in abnormal placental development in cloned gestations, contributing to impaired fetal development and poor survival rates.
VEGF, VEGFR2 (show KDR Antibodies) and GSTM1 (show GSTM1 Antibodies) polymorphisms in outcome of multiple myeloma patients treated with thalidomide-based regimens.
High VEGF expression is associated with hepatocellular carcinoma.
we found significant correlation with CD105 and survival rate of the patient. Similar correlation was found between histological grades and TNM (show ODZ1 Antibodies) staging in CD105. High expression was associated with low survival; whereas no significant correlation was found with expression of VEGF with survival, also with TNM (show ODZ1 Antibodies) staging and histological grading.
SNP rs2010963 located in the 5' untranslated region of the VEGFA gene can influence genetic susceptibility to primary varicose veins in Russians.
miR (show MLXIP Antibodies)-203 suppressed trophoblast cell proliferation, migration and invasion through the downregulation of VEGFA expression in the placenta samples. A correlation between miR (show MLXIP Antibodies)-203 and VEGFA was identified, which may contribute to the onset and/or progression of PE.
High VEGFA expression is associated with early-stage, but not in late-stage, laryngeal squamous cell carcinoma.
the results suggested that miR (show MLXIP Antibodies)-638 might perform tumor suppressive effects in Ewing sarcoma which might be mediated, at least partially, through suppressing the activity of VEGFA.
Adenocarcinomas showed significantly higher staining scores of both VEGF and alphaSMA (show ACTA2 Antibodies) than squamous cell carcinomas did. In 42 cases of high CD31 (show HBA1 Antibodies) score, five-year survival rate (87%) of patients with lung cancer showing mature tumor vessels was significantly better than that (69%) of patients with immature tumor vessels
Single nucleotide polymorphism of VEGF-A is associated with relapse in gastroenteropancreatic neuroendocrine neoplasms.
MALAT1 has an in port ant role in the onset of osteolysis via its ability to induce RANKL (show TNFSF11 Antibodies) expression and inhibit the effect of miR225p.
These data provide a new pathological perspective on cerebellar astrogliosis in Niemann-Pick type C disease and suggest the importance of VEGF as a therapeutic target for this disease.
leukocyte domiciled midkine (show MDK Antibodies) mediates increased plasma levels of VEGFA relevant for upregulation of endothelial nitric oxide synthase (show NOS3 Antibodies) 1 (show NOS Antibodies) and 3
Mesenchymal stem cells secrete VEGF which in turn mediates the differentiation of endothelial progenitor cells into endothelial cells.
This study showed that the quantity of VEGF in the glioma microenvironment seems to be crucial for the participation of microglia/macrophages on tumor progression.
AK131850 directly competed miR (show MLXIP Antibodies)-93-5p in N-OC and M-OC through sponge, thereby increasing VEGFa transcription, expression and secretion through derepressing of miR (show MLXIP Antibodies)-93-5p on VEGFa.
miR203 expression may be upregulated by IL17 (show IL17A Antibodies) stimulation, and miR203 is a positive regulator of IL17induced VEGF secretion.
NF-kappaBmiR15abFGF/VEGFA axis contributes to the impaired angiogenic capacity of bone marrowmesenchymal stem cells in high fat dietfed mice.
Results support the idea that excess heparin binding epidermal growth factor-like growth factor (HB-EGF (show HBEGF Antibodies)) leads to a significant elevation of vascular endothelial growth factor (VEGF (show VEGF Antibodies)) and ventricular dilatation. These data suggest a potential pathophysiological mechanism that elevated HB-EGF (show HBEGF Antibodies) can elicit VEGF induction and hydrocephalus.
Over-expression of VEGF-A165b is protective against proteinuria in a mouse model with progressive depletion of all endogenous VEGF-A splice isoforms from the kidney.
The present data suggest that Ischemic preconditioning transiently increases plasma VEGF levels by downregulating miR (show MLXIP Antibodies)-762 and miR (show MLXIP Antibodies)-3072-5p in CD34 (show CD34 Antibodies)-positive BM cells, leading to protection against organ ischemia.
It was concluded that VEGF and factor VIII are important growth factors associated with fetal development in pigs and are identified in all uterine segments.
results are consistent with a participation of (VEFG) in the regulation of the dynamics of oviductal fluid secretion and the oviduct contractibility
Here we demonstrate that VEGF-165 mediates MSC (show MSC Antibodies) differentiation into ECs via VEGFR-2 (show KDR Antibodies)-dependent induction of Sox18 (show SOX18 Antibodies), which ultimately coordinates the transcriptional upregulation of specific markers of the EC phenotype.
VEGF was significantly downregulated 7 days after cryotherapy of the sclera and stayed at that level until day 14. It returned to baseline by day 21.
VEGF production, blood vessel network and follicle remodeling are impaired by the antiprogesterone RU486.
findings suggest that the augmented neovascular response seen with VEGF administration was through the VEGF-induced upregulation of Notch (show NOTCH1 Antibodies) signaling.
interleukin-1beta-induced vascular endothelial growth factor (show VEGF Antibodies) in airway smooth muscle cells
Upregulation of VEGF during hypoxia in chondrocyte is mediated partially through HIF-1alpha (show HIF1A Antibodies).
data shows that members of the VEGF-VEGFR (show KDR Antibodies) system are temporally and spatially well localized for playing key roles during umbilical cord formation and its intensive growth observed after day 75 of pregnancy
cardiac-specific and hypoxia-induced coexpression of VEGF and Ang1 (show ANGPT1 Antibodies) improves the perfusion and function of porcine MI heart through the induction of angiogenesis and cardiomyocyte proliferation
Peripheral Blood-Derived Mesenchymal Stromal Cells Promote Angiogenesis via Paracrine Stimulation of Vascular Endothelial Growth Factor (show VEGF Antibodies) Secretion
TNF (show TNF Antibodies) may up-regulate VEGF and stimulate angiogenesis in the mare (show C16orf35 Antibodies) early corpus luteum.
After acoustic trauma, vascular endothelial growth factor (show VEGF Antibodies) was up-regulated both in the cochlea and vestibule. Expression in both structures suggests a reparative role with potentially therapeutic implications.
Studied the role of T help 17 cells (Th17) and STAT3 (show STAT3 Antibodies)-VEGF pathway in pathogenesis of psoriasis.
MiR (show MYLIP Antibodies)-145 silencing promotes bone repair of avascular necrosis of femoral head (ANFH (show COL2A1 Antibodies)) via upregulating VEGF, bFGF (show FGF2 Antibodies) and inhibiting the bone cells apoptosis through Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) pathway
ghrelin (show GHRL Antibodies) can inhibit intraplaque angiogenesis and promote plaque stability by down-regulating VEGF and VEGFR2 (show KDR Antibodies) expression, inhibiting the plaque content of macrophages, and reducing MCP-1 (show CCL2 Antibodies) expression at an advanced stage of atherosclerosis in rabbits
VEGF mRNA abundance was present at low levels at 16 h post-ovulation and remained low at 72 h, but the level increased at 144 h in uterus.
correlation was observed between CT perfusion parameters (BF, BV, PS, and MAV (show KLHL2 Antibodies)) and expression of VEGF and MVD (show MVD Antibodies) in tumor tissue
Physiologic ischemic training stimulates VEGF-mediated mobilization of endothelial progenitor cells as well as angiogenesis.
Suggest supplemental oxygen inhibits HIF-1alpha (show HIF1A Antibodies)/VEGF signaling to reduce smooth muscle proliferation in rabbit arteriovenous fistula model.
Therefore, it was reasonable to speculate that the increased expression of VEGF and MMP-9 (show MMP9 Antibodies) in residual hepatic tumor cells and tumor angiogenesis post-embolization would be responsible for the increased metastatic potentiality and invasiveness.
VEGF expression peaked at 8 weeks after meniscus transplantation
Studied the biocompatibility and neovascularization of the PLGA nanospheres wrapped with vascular endothelial growth factor (VEGF (show VEGF Antibodies)).
VEGF induces TGF-beta1 (show TGFB1 Antibodies) expression and myofibroblast transformation after glaucoma surgery.
Data suggest that microRNA-126 plays role in diabetic retinopathy; here, choroid-retinal endothelial cells exposed to high glucose exhibit down-regulation of microRNA-126 and up-regulation of VEGFA and PIK3R2; overexpression of microRNA-126 down-regulates expression of VEGFA and PIK3R2. (VEGFA = vascular endothelial growth factor A; PIK3R2 = class Ia phosphoinositol-3 kinase regulatory subunit 2 (show CCT2 Antibodies))
VEGF plays an important role in choroid-retinal endothelial cell proliferation and tube formation.
These findings suggest that FBLN5 (show FBLN5 Antibodies) may interfere with choroidal neovascularization by downregulating VEGF, CXCR4 (show CXCR4 Antibodies), and TGFB1 (show TGFB1 Antibodies) expression and inhibiting choroidl endothelial cell proliferation.
Apelin (show APLN Antibodies) may play a role in the development of central retinal vein occlusion (CRVO). Apelin (show APLN Antibodies) has a unique upstream signaling pathway, independent of the VEGF pathway.
Dll4 (show DLL4 Antibodies) play an important role in choroidal neovascularization (CNV) angiogenesis, which appears to be regulated by HIF-1alpha (show HIF1A Antibodies) and VEGF during the progression of CNV under hypoxic conditions.
A functional network involving VEGF, IGF2, and MMP9 (show MMP9 Antibodies) in early placental trophoblast cells and maternal endometrium appears to be important for normal placentation.
This gene is a member of the PDGF/VEGF growth factor family and encodes a protein that is often found as a disulfide linked homodimer. This protein is a glycosylated mitogen that specifically acts on endothelial cells and has various effects, including mediating increased vascular permeability, inducing angiogenesis, vasculogenesis and endothelial cell growth, promoting cell migration, and inhibiting apoptosis. Elevated levels of this protein is linked to POEMS syndrome, also known as Crow-Fukase syndrome. Mutations in this gene have been associated with proliferative and nonproliferative diabetic retinopathy. Alternatively spliced transcript variants, encoding either freely secreted or cell-associated isoforms, have been characterized. There is also evidence for the use of non-AUG (CUG) translation initiation sites upstream of, and in-frame with the first AUG, leading to additional isoforms.
, vascular endothelial growth factor A
, vascular endothelial growth factor A-A
, vascular endothelial growth factor A-a
, vascular endothelial growth factor 164
, vascular permeability factor
, vascular endothelial growth factor 188
, angiogenic factor
, Vascular permeability factor
, endothelial growth factor-164
, vascular endothelial growth factor VEGF