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anti-Human S100A9 Antibodies:
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Human Polyclonal S100A9 Primary Antibody for ICC, IF - ABIN409938
Sumi, Shimizu, Himeno: Involvement of Nrf2 activation in the upregulation of S100A9 by exposure to inorganic arsenite. in International journal of molecular medicine 2012
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Human Monoclonal S100A9 Primary Antibody for ELISA, WB - ABIN948434
Li, Riau, Setiawan, Mehta, Ti, Tong, Tan, Beuerman: S100A expression in normal corneal-limbal epithelial cells and ocular surface squamous cell carcinoma tissue. in Molecular vision 2011
Show all 2 Pubmed References
Human Polyclonal S100A9 Primary Antibody for WB - ABIN519967
Bianchi, Niemiec, Siler, Urban, Reichenbach: Restoration of anti-Aspergillus defense by neutrophil extracellular traps in human chronic granulomatous disease after gene therapy is calprotectin-dependent. in The Journal of allergy and clinical immunology 2011
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Human Monoclonal S100A9 Primary Antibody for EIA, FACS - ABIN111888
Brandtzaeg, Jones, Flavell, Fagerhol: Mac 387 antibody and detection of formalin resistant myelomonocytic L1 antigen. in Journal of clinical pathology 1989
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Human Monoclonal S100A9 Primary Antibody for EIA, FACS - ABIN111889
Delabie, de Wolf-Peeters, van den Oord, Desmet: Differential expression of the calcium-binding proteins MRP8 and MRP14 in granulomatous conditions: an immunohistochemical study. in Clinical and experimental immunology 1990
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Human Polyclonal S100A9 Primary Antibody for IHC (p), IHC - ABIN449656
McCormick, Rahimi, Bobryshev, Gaus, Zreiqat, Cai, Lord, Geczy: S100A8 and S100A9 in human arterial wall. Implications for atherogenesis. in The Journal of biological chemistry 2005
Show all 2 Pubmed References
Human Monoclonal S100A9 Primary Antibody for EIA, FACS - ABIN238498
Mortezai, Harder, Schnabel, Moors, Gauly, Schlüter, Wagener, Buck: Tandem affinity depletion: a combination of affinity fractionation and immunoaffinity depletion allows the detection of low-abundance components in the complex proteomes of body fluids. in Journal of proteome research 2010
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Human Monoclonal S100A9 Primary Antibody for ELISA, WB - ABIN519968
Feng, Lee, Chang, Chan, Kuo, Lin, Chung, Kuo, Yu, Lin, Wang, Chou, Huang, Kuo: CD14(+)S100A9(+) monocytic myeloid-derived suppressor cells and their clinical relevance in non-small cell lung cancer. in American journal of respiratory and critical care medicine 2012
Human Monoclonal S100A9 Primary Antibody for IA, FACS - ABIN2192037
Robinson, Tessier, Poulsom, Hogg: The S100 family heterodimer, MRP-8/14, binds with high affinity to heparin and heparan sulfate glycosaminoglycans on endothelial cells. in The Journal of biological chemistry 2002
Authors found that downregulation of S100A9 significantly reduced the expression of NF-kappaB (show NFKB1 Antibodies), phosphorylation of NF-kappaB (show NFKB1 Antibodies) and Bcl-2 (show BCL2 Antibodies), as well as the expression of MMP7 (show MMP7 Antibodies) and MMP2 (show MMP2 Antibodies). Restoration of NF-kappaB (show NFKB1 Antibodies) expression sufficiently reversed the inhibitory effects on cell proliferation and invasion induced by S100A9 downregulation in vitro and in vivo.
Urinary NGAL (show LCN2 Antibodies) is a good predictor of mortality in established pediatric acute kidney injury (AKI) of heterogeneous etiology and outperforms calprotectin (show S100A8 Antibodies) and KIM-1 (show HAVCR1 Antibodies). Urinary calprotectin (show S100A8 Antibodies) and KIM-1 (show HAVCR1 Antibodies) predict renal replacement therapy requirement in pediatric AKI.
Identify proteins that interact with calgranulin B to suppress the proliferation of colon cancer cells.
This study demonstrated that S100A9 was detected in the serums of Alzheimer disease patients
Expression levels of S100A9 and Tenascin-C (show TNC Antibodies) were correlated with TNM (show ODZ1 Antibodies) stages and metastasis in colorectal cancer.
In Guatemalan preschoolers, the median fecal calprotectin (show S100A8 Antibodies) value was 58 mg/kg, with a mean of 98 +/- 136 mg/kg and a range from 10 to 950 mg/kg; 61% of values were above the manufacturer's cut-off for elevated concentration and 51% exceeded an age-adjusted criterion. The central tendency (mean or median) and distribution were generally shifted to the right in relation to comparable reports from children across the world.
Low S100A9 expression is associated with head and neck neoplasms.
Report role of fecal calprotectin (show S100A8 Antibodies) assay in the diagnosis and management of inflammatory bowel disease.
S100A8 (show S100A8 Antibodies) and S100A9 aggravate Coxsackievirus B3-induced myocarditis and might serve as therapeutic targets in inflammatory cardiomyopathies.
Calprotectin (show S100A8 Antibodies) had the highest association with ultrasound synovitis and predicted treatment response. It may thus be considered as a marker for evaluating inflammation and responsiveness in patients with rheumatoid arthritis on biologic disease-modifying anti-rheumatic drug treatment.
Data suggest that up-regulation of S100A8 (show S100A8 Antibodies) and S100A9 is a key component of early endometrial response to uterine involution in the post-partum period and to prevent chronic endometritis/uterine inflammation; up-regulation can be influenced by diet.
Study verified porcine calprotectin (S100A8 (show S100A8 Antibodies)/A9) expression at the protein level in multiple Haemophilus parasuis infected tissues and explored their molecular characterization.
this study shows that S100A9 inhibits B lymphopoiesis by acting on myeloid cells and promoting the release of inflammatory molecules, including IL-1beta (show IL1B Antibodies)
data suggest that S100A8 (show S100A8 Antibodies)/A9 acts directly on BV-2 microglial cells via binding to TLR4 (show TLR4 Antibodies) and RAGE (show AGER Antibodies) on the membrane and then stimulates the secretion of proinflammatory cytokines through ERK (show EPHB2 Antibodies) and JNK (show MAPK8 Antibodies)-mediated NF-kappaB (show NFKB1 Antibodies) activity in BV-2 microglial cells.
Like S100A8 (show S100A8 Antibodies), S100A9 and S100A8 (show S100A8 Antibodies)/A9 reduced neutrophil influx in acute lung injury provoked by lipopolysaccharide (LPS (show TLR4 Antibodies)) challenge.
findings reveal unexpected gene expression differences between WT and KO mice at a young age (in the absence of physiological stress), and address the hypothesis that Mrp8 (show S100A8 Antibodies) and Mrp14 accumulation promotes age-related inflammation
An excessive expansion of monocytic-myeloid-derived suppressor cells in S100A9-/- neonates compared to wild-type neonates. This strong baseline expansion was associated with hyperinflammatory responses during endotoxemia and fatal septic courses
Data support a role of S100A9 to initiate and amplify the neutrophilic inflammation in asthma, possibly via inducing IL-1beta (show IL1B Antibodies), IL-17 (show IL17A Antibodies) and IFN-gamma (show IFNG Antibodies).
The findings show that reduced PTP1B (show PTPN1 Antibodies) responses contribute to disease symptoms in part by enhancing S100A9 expression during viral-associated chronic obstructive pulmonary disease exacerbations.
Neutrophil-derived S100A8 (show S100A8 Antibodies)/A9 promotes thrombocytosis in diabetic mice
S100A9 knockdown almost completely abrogated the effects of IRF7 (show IRF7 Antibodies) deletion on granulocytic myeloid-derived suppressor cells development and tumor metastasis.
Perinatal alarmins S100A8 (show S100A8 Antibodies) and S100A9 specifically altered MyD88 (show MYD88 Antibodies)-dependent proinflammatory gene programs. S100 programming prevented hyperinflammatory responses without impairing pathogen defense. TRIF (show RNF138 Antibodies)-adaptor-dependent regulatory genes remained unaffected by perinatal S100 programming and responded strongly to lipopolysaccharide, but were barely expressed.
Calprotectin (show S100A8 Antibodies) expression in laminar epidermis occurs after extravasation of leukocytes indicating that leukocyte emigration may be an initiating factor in laminar epithelial stress and inflammation in black walnut extract-induced laminitis.
The protein encoded by this gene is a member of the S100 family of proteins containing 2 EF-hand calcium-binding motifs. S100 proteins are localized in the cytoplasm and/or nucleus of a wide range of cells, and involved in the regulation of a number of cellular processes such as cell cycle progression and differentiation. S100 genes include at least 13 members which are located as a cluster on chromosome 1q21. This protein may function in the inhibition of casein kinase and altered expression of this protein is associated with the disease cystic fibrosis.
, S100 calcium-binding protein A9 (calgranulin B)
, calgranulin B
, calprotectin L1H subunit
, leukocyte L1 complex heavy chain
, migration inhibitory factor-related protein 14
, protein S100-A9
, S100 calcium binding protein A9 (calgranulin B)
, intracellular calcium-binding protein (MRP14)
, myeloid-related protein 14
, protein MRP-126
, S100 calcium binding protein A9
, S100 calcium-binding protein A9
, neutrophil cytosolic 23 kDa protein
, RNA-binding region containing protein 2-like