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Human Monoclonal TFR Primary Antibody for IF, WB - ABIN968749
McClelland, Kühn, Ruddle: The human transferrin receptor gene: genomic organization, and the complete primary structure of the receptor deduced from a cDNA sequence. in Cell 1985
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Human Monoclonal TFR Primary Antibody for IF, WB - ABIN968748
Seiser, Posch, Thompson, Kühn: Effect of transcription inhibitors on the iron-dependent degradation of transferrin receptor mRNA. in The Journal of biological chemistry 1996
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Human Polyclonal TFR Primary Antibody for ICC, IF - ABIN4358760
Ta, Keller, Haltmeier, Saka, Schmied, Opazo, Tinnefeld, Munk, Hell: Mapping molecules in scanning far-field fluorescence nanoscopy. in Nature communications 2015
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Human Monoclonal TFR Primary Antibody for ELISA, FACS - ABIN4358817
Ng: Iron, microbiota and colorectal cancer. in Wiener medizinische Wochenschrift (1946) 2016
Human Polyclonal TFR Primary Antibody for ELISA, WB - ABIN4358779
Jian, Yang, Huang: Src regulates Tyr(20) phosphorylation of transferrin receptor-1 and potentiates breast cancer cell survival. in The Journal of biological chemistry 2011
Simian immunodeficiency virus Nef-induced inhibition of TfR (show TFRC Antibodies) endocytosis leads to the reduction of Transferrin (show Tf Antibodies) uptake and intracellular iron concentration and is accompanied by attenuated lentiviral replication in macrophages.
combination of desferal with oxaliplatin can overcome cervical cancer oxaliplatin resistance through the regulation of hCtr1 (show SLC31A1 Antibodies) and TfR1 (show TFRC Antibodies)
Data suggest that hematologic parameters in children consuming lacto-ovo vegetarian diets are comparable with those of control children, but ferritin (show FTL Antibodies) levels are lower; inclusion of novel serum biomarkers, soluble transferrin receptor and hepcidin (show HAMP Antibodies), in nutritional assessment can better detect subclinical iron deficiency in children following vegetarian diet. This study was conducted in Poland with children ages 4.5-9 years.
results suggest that lower Hepcidin-25, as well as higher sTfR and sTfR/Hepcidin-25 ratio were significant predictors of favorable hemoglobin response within a month after IV administration of ferric carboxymaltose in patients with CKD
TfR1 (show TFRC Antibodies) was highly expressed in glioblastomas and associated with shorter survival in the whole cohort, but not in the individual malignancy grades
Expression of Hepcidin (show HAMP Antibodies) and Ferroportin (show SLC40A1 Antibodies) in the Placenta, and Ferritin (show FTL Antibodies) and Transferrin Receptor 1 Levels in Maternal and Umbilical Cord Blood in Pregnant Women with and without Gestational Diabetes
Transferrin (show Tf Antibodies) facilitates the formation of DNA double-strand breaks (DNA-DSBs) via transferrin receptor TfR1 but not TfR2 (show TFR2 Antibodies).
Human melanoma cells are able to up-regulate TFRC (show TFRC Antibodies) expression using hyaluronan/CD44 (show CD44 Antibodies) signaling.
high expression of transferrin receptor-1 resulting in iron uptake contributes to increase in the labile iron pool which plays roles in cholangiocarcinoma progression with aggressive clinical outcomes.
EGFR (show EGFR Antibodies) regulates iron homeostasis to promote non-small cell lung cancer growth through redistribution of transferrin receptor 1.
TFR1 (show TFRC Antibodies) is overexpressed in adult T-cell leukemia/lymphoma caells .
Knock-out of TFR1 (show TFRC Antibodies) in Purkinje cells reduces mGlu1 (show GRM1 Antibodies) expression at synapses and impairs motor coordination.
Tfrc deletion dramatically suppressed both transforming growth factor-beta (TGF-beta) and bone morphogenetic protein (BMP) signaling in cranial neural crest cell-derived mandibular tissues.
decreasing TfR1 (show TFRC Antibodies) expression during beta-thalassemic erythropoiesis, either directly via induced haploinsufficiency or via exogenous apotransferrin, decreases ineffective erythropoiesis and provides an endogenous mechanism to upregulate hepcidin (show HAMP Antibodies), leading to sustained iron-restricted erythropoiesis and preventing systemic iron overload in beta-thalassemic mice
the value of transferrin receptors (TfRs)/cell reveals a 100-fold increase in the number of TfRs per bEnd3 cells compared to human umbilical vein cells.
our study reveals that TFR (show TFRC Antibodies) functions as a novel regulator to control AMPA (show GRIA3 Antibodies) trafficking efficiency and synaptic plasticity
We found that iron assimilation via Tfr1 (show TFRC Antibodies) was critical for skeletal muscle metabolism, and that iron deficiency in muscle led to dramatic changes, not only in muscle, but also in adipose tissue and liver.
Mice lacking Tfr1 (show TFRC Antibodies) in the heart died in the second week of life and had cardiomegaly, poor cardiac function, failure of mitochondrial respiration, and ineffective mitophagy.
Transferrin Receptor 1 Facilitates Poliovirus Permeation of Mouse Brain Capillary Endothelial Cells.
Erythrocytic iron deficiency enhances susceptibility to Plasmodium chabaudi infection in mice carrying a missense mutation in tfr1 (show TFRC Antibodies).
Tfr1 (show TFRC Antibodies) has a role in homeostatic maintenance of the intestinal epithelium, acting through a role that is independent of its iron-uptake function
This gene encodes a cell surface receptor necessary for cellular iron uptake by the process of receptor-mediated endocytosis. This receptor is required for erythropoiesis and neurologic development. Mice that are deficient in this receptor show impaired erythroid development and abnormal iron homeostasis.
, transferrin receptor protein 1
, transferrin receptor 1
, mammary tumor virus receptor 1