Browse our anti-FZD3 (FZD3) Antibodies

Human Frizzled Family Receptor 3 (FZD3) interaction partners

1. schizophrenia may be related to some polymorphisms of gene FZD3 that are in stronger linkage disequilibrium to Chinese than to the other populations studied (Meta-Analysis)

2. marked reduction in the prominence of TUJ1 bundles in number, thickness, and length. Our results showed that deregulation of the planar cell polarity genes CELSR3 (show CELSR3 Antibodies) and FZD3 might disrupt the enteric innervation pattern

3. seven-transmembrane domain receptors Celsr3 (show CELSR3 Antibodies) and Fzd3, in particular, control the development of most longitudinal tracts in the central nervous system. [Review]

4. Our analysis showed no significant association between the rs2241802 polymorphism in FZD3 gene and neural tube defects

5. DNA methylation (show HELLS Antibodies) aberrations rather than polymorphisms of FZD3 gene increase the risk of spina bifida in a high-risk region for neural tube defects.

6. Wnt3a (show WNT3A Antibodies)/Frizzled-3 signaling plays important role in regulating the proliferation and differentiation of neural crest cells and various developmental stages of melanocyte precursors.

7. Aberrant methylation modification of the FZD3 gene increases the risk of congenital hydrocephalus (show FOXC1 Antibodies) by altering chromatin structure and disturbing gene expression.

8. FZD3 signaling sensitized peripheral sensory neurons in pain hypersensitivity.

9. clinical significance of frizzled homolog 3 protein in colorectal cancer patients

10. Genetic variants of the FZD3 gene may affect susceptibility to schizophrenia in Chinese Han and Va populations.

Xenopus laevis Frizzled Family Receptor 3 (FZD3) interaction partners

1. loss of Fzd3 induces severe malformations of the developing eye and this defect is phenocopied by loss of the activated leukocyte cell adhesion molecule (Alcam (show ALCAM Antibodies)).

Mouse (Murine) Frizzled Family Receptor 3 (FZD3) interaction partners

1. Results suggest that Celsr3 (show CELSR3 Antibodies) and Fzd3 orchestrate the formation of a scaffold of pioneer neurons and their axons during embryogenesis. This scaffold extends from prethalamus to ventral telencephalon and subcortex, and steers reciprocal corticothalamic fibers.

2. Frizzled3 is required to shape the pattern of RBC (show CACNA1C Antibodies) somas and dendrites, and the structural and functional connectivity between rods and RBCs. Our results highlight novel functions for Fzd3 in regulating retinal development.

3. Temporal and spatial expression profiles of Frizzled 3 in the ovary during the estrous cycle has been reported.

4. Celsr3 (show CELSR3 Antibodies) and Fzd3 enable immature neurons to respond to Wnt7 (show WNT7B Antibodies), upregulate Jag1 (show JAG1 Antibodies) and thereby facilitate feedback signals that tune the timing of neural progenitor cell fate decisions via Notch (show NOTCH1 Antibodies) activation.

5. This study demonstrated that Frizzled3 Controls Axonal Polarity and Intermediate Target Entry during Striatal Pathway Development.

6. In Fz3(-/-) limbs, dorsal axons stall at a precise location in the nerve plexus, and, in contrast to the phenotypes of several other axon path-finding mutants, Fz3(-/-) dorsal axons do not reroute to other trajectories

7. Wnt3a (show WNT3A Antibodies)/Frizzled-3 signaling plays important role in regulating the proliferation and differentiation of neural crest cells and various developmental stages of melanocyte precursors.

8. Fz3 and Fz6 (show FZD6 Antibodies) have partly interchangeable roles in tissue polarity signaling for epithelial orientation and axon growth and guidance

9. The first signal, controlled by cadherin, EGF (show EGF Antibodies)-like, laminin G-like, seven-pass, G-type receptor (Celsr) 2 (show CELSR2 Antibodies), Celsr3 (show CELSR3 Antibodies), Frizzled3 (Fzd3) and Van Gogh (show VANGL2 Antibodies) like2 (Vangl2 (show VANGL2 Antibodies)) organizes multicilia in individual cells (single-cell polarity)

10. Celsr2 (show CELSR2 Antibodies)-3 and Fzd3 regulate axonal navigation in the forebrain by using mechanisms different from classical epithelial core planar cell polarity, and require interacting partners other than Vangl1 (show Vangl1 Antibodies)-2 that remain to be identified.