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Chylomicron remnants and very low density lipoprotein (VLDL) remnants are rapidly removed from the circulation by receptor-mediated endocytosis in the liver. Additionally we are shipping and many more products for this protein.
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The results suggest that education and female gender, not APOE (show APOE ELISA Kits) genotype, may be important as independent strong predictive factors for disease progression in patients with early-onset Alzheimer's disease.
C2238/alphaANP downregulates ApoE (show APOE ELISA Kits) in vascular smooth muscle cells through type C natriuretic peptide receptor-dependent activation of Egr-1 (show EGR1 ELISA Kits) and the consequent upregulation of miR199a.
Sporadic Abeta (show APP ELISA Kits) accumulation may be partly associated with increased amyloidogenic APP (show APP ELISA Kits) production, especially in APOE (show APOE ELISA Kits) varepsilon4-negative subjects.
The APOE epsilon2 allele increases the risk of Frontotemporal Dementia in ALS patients.
Single-nucleotide polymorphisms in APOE (show APOE ELISA Kits) gene is associated with Alzheimer's disease.
Strong association between ApoE (show APOE ELISA Kits) e4 allele and AD.
Higher peak velocity-reactivity in APOE (show APOE ELISA Kits) epsilon4 carriers suggests vascular compensation for deleterious effects of this known risk allele for Alzheimer's disease and stroke
APOE (show APOE ELISA Kits)-epsilon4 carriers showed an inability to effectively modulate posteromedial cortex during scene, but not face and object, working memory and perception.
These results demonstrate that the impact of cerebrovascular risk on cortical thickness integrity may vary by the presence or absence of the APOE (show APOE ELISA Kits)-4 allele, a well-known risk factor for Alzheimer's disease.
Our results suggest that APOE (show APOE ELISA Kits)-E4 allele status may be marginally associated with better hearing thresholds in older adults.
Chylomicron remnants and very low density lipoprotein (VLDL) remnants are rapidly removed from the circulation by receptor-mediated endocytosis in the liver. Apolipoprotein E, a main apoprotein of the chylomicron, binds to a specific receptor on liver cells and peripheral cells. ApoE is essential for the normal catabolism of triglyceride-rich lipoprotein constituents. The APOE gene is mapped to chromosome 19 in a cluster with APOC1 and APOC2. Defects in apolipoprotein E result in familial dysbetalipoproteinemia, or type III hyperlipoproteinemia (HLP III), in which increased plasma cholesterol and triglycerides are the consequence of impaired clearance of chylomicron and VLDL remnants.
, apolipoprotein E3