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The protein encoded by CSF3 is a cytokine that controls the production, differentiation, and function of granulocytes. Additionally we are shipping CSF3 Proteins (116) and CSF3 Kits (109) and many more products for this protein.
Showing 10 out of 398 products:
Human Monoclonal CSF3 Primary Antibody for FACS - ABIN2661167
Demetri, Griffin: Granulocyte colony-stimulating factor and its receptor. in Blood 1991
Show all 6 references for ABIN2661167
Human Polyclonal CSF3 Primary Antibody for WB - ABIN1881232
Takagi, Nakamura, Miwa, Adachi, Fujioka, Haruki, Taniguchi: A case of G-CSF-producing invasive apical cancer resected following preoperative adjuvant therapy. in The Thoracic and cardiovascular surgeon 2010
Show all 2 references for ABIN1881232
Cow (Bovine) Polyclonal CSF3 Primary Antibody for IHC, ELISA - ABIN1582209
Biscetti, Pecorini, Straface, Arena, Stigliano, Rutella, Locatelli, Angelini, Ghirlanda, Flex: Cilostazol promotes angiogenesis after peripheral ischemia through a VEGF-dependent mechanism. in International journal of cardiology 2013
Human Polyclonal CSF3 Primary Antibody for IF (p), IHC (p) - ABIN736521
Kameyama, Udagawa, Hoshi, Toukairin, Arai, Nogami: The mRNA expressions and immunohistochemistry of factors involved in angiogenesis and lymphangiogenesis in the early stage of rat skin incision wounds. in Legal medicine (Tokyo, Japan) 2015
data provide novel information about the regulation of neutrophil migration in zebrafish, positioning Gcsf-Chr19 as a key signal during the course of an inflammatory process triggered by severe damage
Data indicate that Gcsf is required for the specification and proliferation of hematopoietic stem cells, suggesting that Gcsf represents an ancestral cytokine responsible for the broad support of hematopoietic stem and progenitor cells (HSPCs).
SB203580 increases G-CSF expression in macrophages by increasing the stability of G-CSF mRNA via its 3'UTR (show UTS2R Antibodies), and the effect was not due to its inhibition of p38 MAPK (show MAPK14 Antibodies) activity.
Results suggested that G-CSF plays an important role in preventing colitis, likely through populating immune regulatory macrophages in the intestine.
findings provide convincing evidence that monophosphoryl lipid A-induced G-CSF facilitates early expansion, mobilization, and recruitment of neutrophils to the site of infection after burn injury
Overexpression of VEGF (show VEGFA Antibodies) may compensate for the G-CSF deficit through preservation of cellular components, including blood vessels, in the postinfarction heart.
G-CSF supports long-term muscle regeneration in mouse models of muscular dystrophy.
constitutive activation of the NF-kappaB (show NFKB1 Antibodies) pathway in CAIX (show CA9 Antibodies)-depleted cells restored G-CSF secretion
Exposure of Sca-1(+) cells to G-CSF in the culture medium for 72 h induced time-dependent but self-limiting cell cycle acceleration with a restricted effect on the CSC proliferation.
these results suggest that the G-CSF pathway regulates the production of autoantibodies in murine models of lupus.
G-CSF acts in a cell intrinsic manner to expand multipotent progenitors to increase production of tumor-derived Ly6G+ neutrophils.
Endothelial cell(EC)-intrinsic MYD88 (show MYD88 Antibodies) signaling and subsequent G-CSF production by ECs is required for myeloid progenitor lineage skewing toward granulocyte-macrophage progenitors, increased colony-forming unit granulocyte activity.
tumor G-CSF expression is an indicator of an extremely poor prognosis in cervical cancer patients that are treated with chemotherapy.
G-CSF stimulates beta1 integrin expression and Swan (show RBM12 Antibodies) 71 cell migration by activating PI3K (show PIK3CA Antibodies) and MAPK (show MAPK1 Antibodies) signaling pathways.
these data suggest that G-CSF may contribute to tumor growth and decrease the antitumor effect of radiotherapy, possibly by promoting vascularization in cancer lesions.
in vitro chemotaxis assays and an in vivo transplantation model for chemoattraction confirmed that UCX((R)) are chemotactic to CD34 (show CD34 Antibodies)(-)/CD45 (show PTPRC Antibodies)(-) BM-MSCs via a cell-specific mobilization mechanism mediated by G-CSF.
Case Report: undifferentiated colon carcinoma producing G-CSF.
increases MMP-2 (show MMP2 Antibodies) activity and VEGF (show VEGFA Antibodies) secretion in trophoblasts through activation of PI3K (show PIK3CA Antibodies)/Akt (show AKT1 Antibodies) and Erk (show EPHB2 Antibodies) signaling pathways
Elevated IL-8 (show IL8 Antibodies) and G-CSF may be involved in the pathophysiology of narcolepsy.
G-CSF- or GM-CSF (show CSF2 Antibodies)-secreting cancers, albeit not very common are, however, among the most rapidly advancing ones due to a cytokine-mediated immune suppression and angiogenesis. [Review]
GM-CSF (show CSF2 Antibodies), through its stimulatory function on macrophages, may promote aneurysm progression.
G-CSF constrains cancer to grow and progress by, respectively, supporting the survival of sympathetic nerve fibers in 6-hydroxydopamine-sympathectomized mice.
Data suggest granulocyte-colony stimulating factor (G-CSF) as a possible candidate for agents in which neutrophils can provide protection.
The protein encoded by this gene is a cytokine that controls the production, differentiation, and function of granulocytes. The active protein is found extracellularly. Alternatively spliced transcript variants have been described for this gene.
colony stimulating factor 3 (granulocyte)
, granulocyte colony-stimulating factor
, granulocyte colony stimulating factor
, Granulocyte colony-stimulating factor