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Glial Fibrillary Acidic Protein (GFAP) ELISA Kits

GFAP encodes one of the major intermediate filament proteins of mature astrocytes. Additionally we are shipping GFAP Antibodies (707) and GFAP Proteins (31) and many more products for this protein.

list all ELISA KIts Gene Name GeneID UniProt
GFAP 2670 P14136
GFAP 24387 P47819
GFAP 14580 P03995
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Top GFAP ELISA Kits at

Showing 10 out of 63 products:

Catalog No. Reactivity Sensitivity Range Images Quantity Supplier Delivery Price Details
Rat 5.7 pg/mL 62.5-4000 pg/mL 96 Tests Log in to see 9 to 11 Days
Human 0.156 ng/mL 0.625-40 ng/mL   96 Tests Log in to see 9 to 11 Days
Mouse 0.71 pg/mL 7.81-500 pg/mL 96 Tests Log in to see 9 to 11 Days
Cow 12.2 pg/mL 31.25-2000 pg/mL 96 Tests Log in to see 9 to 11 Days
Rat 55 pg/mL 4.12-3000 pg/mL 96 Tests Log in to see 9 to 11 Days
Pig 0.05 ng/mL 0.1-20 ng/mL   96 Tests Log in to see 9 to 11 Days
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Guinea Pig
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Top referenced GFAP ELISA Kits

  1. Rat (Rattus) GFAP ELISA Kit for Sandwich ELISA - ABIN416142 : Tskitishvili, Nisolle, Munaut, Pequeux, Gerard, Noel, Foidart: Estetrol attenuates neonatal hypoxic-ischemic brain injury. in Experimental neurology 2014 (PubMed)
    Show all 5 references for ABIN416142

  2. Human GFAP ELISA Kit for Sandwich ELISA - ABIN365729 : Akdemir, Yardan, Kati, Duran, Alacam, Yavuz, Okuyucu: The role of S100B protein, neuron-specific enolase, and glial fibrillary acidic protein in the evaluation of hypoxic brain injury in acute carbon monoxide poisoning. in Human & experimental toxicology 2014 (PubMed)
    Show all 2 references for ABIN365729

More ELISA Kits for GFAP Interaction Partners

Rhesus Monkey Glial Fibrillary Acidic Protein (GFAP) interaction partners

  1. There was significantly more GFAP immunoreactivity in the prefrontal cortex and hippocampus of aged animals compared to adult or middle-aged animals.

Human Glial Fibrillary Acidic Protein (GFAP) interaction partners

  1. This study demonstrated that GFAP as a promising biomarker to distinguish ischemic stroke from intracerebral hemorrhage.

  2. The levels of GFAP in Alzheimer's disease, dementia with Lewy bodies, and frontotemporal lobar degeneration patients were significantly higher than those in the healthy control subjects.

  3. GFAP is significantly associated with outcome, but it does not add predictive power to commonly used prognostic variables in a population of patients with TBI of varying severities.

  4. Neither duplications nor deletions of GFAP were found, suggesting that GFAP coding-region rearrangements may not be involved in Alexander disease or Alexanderrelated leukoencephalopathies.

  5. The data suggest that human vitreous body GFAP is a protein biomarker for glial activation in response to retinal pathologies.

  6. Studied diagnostic Value of Serum Levels of GFAP, pNF-H, and NSE (show ENO2 ELISA Kits) Compared With Clinical Findings in Severity Assessment of Human Traumatic Spinal Cord Injury.

  7. GFAP peaks early during haemorrhagic brain lesions (at significantly higher levels), and late in ischaemic events, whereas antibodies against NR2 RNMDA have significantly higher levels during ischemic stroke at all time-points.

  8. There was an absence of GFAP in astrocytes during early fetal spinal cord development until 9 months of gestation , and the appearance of GFAP-positive reactivity was later than that of neurons.

  9. These data imply that a tight regulation of histone acetylation in astrocytes is essential, because dysregulation of gene expression causes the aggregation of GFAP, a hallmark of human diseases like Alexander's disease.

  10. Identification of a novel nonsense mutation in the rod domain of GFAP that is associated with Alexander disease.

Pig (Porcine) Glial Fibrillary Acidic Protein (GFAP) interaction partners

  1. Isolation of an evolutionary conserved novel GFAP isoform, GFAPkappa, produced by alternative splicing and polyadenylation of the 3'-region of the human GFAP pre-mRNA is described.

Mouse (Murine) Glial Fibrillary Acidic Protein (GFAP) interaction partners

  1. Study provides a mechanistic link between the GFAP mutations/overexpression and the symptoms in those affected with Type II Alexander disease

  2. Study described GFAP-expressing non-myelinating Schwann cells in the lung, validated a transgenic mouse line that drives expression of cre under a GFAP promoter

  3. findings thus show that the inability to produce GFAP and Vim (show VIM ELISA Kits) affects normal retinal physiology and that the effect of IF deficiency on retinal cell survival differs, depending on the underlying pathologic condition

  4. CUL4B (show CUL4B ELISA Kits) as a negative regulator of GFAP expression during neural development.

  5. Astrocytes deficient of GFAP and vimentin (show VIM ELISA Kits) showed decreased Notch (show NOTCH1 ELISA Kits) signal sending competence and altered expression of Notch (show NOTCH1 ELISA Kits) signaling pathway-related genes

  6. Absence of GFAP, or both GFAP and vimentin (show VIM ELISA Kits), alters Alzheimer's disease-induced changes in gene expression profile of astrocytes, showing a compensation of the decrease of neuronal support genes and a trend for a higher inflammatory expression profile

  7. immunohistochemistry revealed increased numbers of GFAP/BDNF (show BDNF ELISA Kits) double-positive cells, which agrees with the observed changes in the culture system

  8. Data indicate that glial fibrillary acidic protein (GFAP) was up-regulated in satellite glial cells (SGCs) in dorsal root ganglia 14 days after streptozotocin injection.

  9. Findings demonstrate that ENT1 (show SLC29A1 ELISA Kits) regulates GFAP expression and possibly astrocyte function

  10. Data suggest that prenatal alterations in expression of various fetal brain proteins (including down-regulation of Gfap) are associated with aberrant behavioral characteristics of transgenic mice that model autism-like behavior.

Zebrafish Glial Fibrillary Acidic Protein (GFAP) interaction partners

  1. The distribution of GFAP immunoreactivity implies that enteric glia are widespread in the fish gastrointestinal tract.

  2. Generation of transgenic zebrafish that express green fluorescent protein (GFP) in glial cells driven by the zebrafish glial fibrillary acidic protein (GFAP) regulatory elements.

  3. Cells expressing the two reporters display radial glial morphology, colocalize with the NSC marker Sox2 (show SOX2 ELISA Kits), undergo proliferation, and are capable of self-renewal within the matrix of distinct thickness in the telencephalon.

GFAP Antigen Profile

Antigen Summary

This gene encodes one of the major intermediate filament proteins of mature astrocytes. It is used as a marker to distinguish astrocytes from other glial cells during development. Mutations in this gene cause Alexander disease, a rare disorder of astrocytes in the central nervous system. Alternative splicing results in multiple transcript variants encoding distinct isoforms.

Gene names and symbols associated with GFAP

  • glial fibrillary acidic protein (GFAP) antibody
  • glial fibrillary acidic protein (LOC100136168) antibody
  • glial fibrillary acidic protein (gfap) antibody
  • glial fibrillary acidic protein (Gfap) antibody
  • AI836096 antibody
  • cb345 antibody
  • etID36982.3 antibody
  • GFAP antibody
  • gfapl antibody
  • wu:fb34h11 antibody
  • wu:fk42c12 antibody
  • xx:af506734 antibody
  • zgc:110485 antibody

Protein level used designations for GFAP

glial fibrillary acidic protein , glial fibrillary acidic protein alpha , intermediate filament , intermediate filament protein , zrf-1 antigen

712941 Macaca mulatta
454741 Pan troglodytes
100136168 Oncorhynchus mykiss
100534495 Oreochromis niloticus
2670 Homo sapiens
24387 Rattus norvegicus
100720409 Cavia porcellus
101081938 Felis catus
396562 Sus scrofa
281189 Bos taurus
14580 Mus musculus
100352532 Oryctolagus cuniculus
419969 Gallus gallus
480495 Canis lupus familiaris
443220 Ovis aries
30646 Danio rerio
100172719 Pongo abelii
100033970 Equus caballus
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