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In vertebrates, the genes encoding the class of transcription factors called homeobox genes are found in clusters named A, B, C, and D on four separate chromosomes. Additionally we are shipping HOXA10 Antibodies (65) and HOXA10 Kits (8) and many more products for this protein.
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To summarize, significant differential methylation of HOXA10 and COMT (show COMT Proteins) promoter regions was found between the ectopic and eutopic endometrial tissues. This is the first study investigating the methylation of HOXA10 and COMT (show COMT Proteins) genes and their linkage to endometriosis in Chinese patients.
using both knockdown and knockout approaches we show that Hottip expression is required for activation of the 5' Hoxa genes (Hoxa13 (show HOXA13 Proteins) and Hoxa10/11) and for retaining Mll1 at the 5' end of Hoxa. Moreover, we demonstrate that artificially inducing Hottip expression is sufficient to activate the 5' Hoxa genes and that Hottip RNA binds to the 5' end of Hoxa
HOXA10 is overexpressed in oral squamous cell carcinoma (OSCC) and its expression is functionally associated with several important biological processes related to oral tumorigenesis, such as proliferation, migration and invasion.
The results demonstrated that mutation in HOXA10 gene contributes to the pathogenesis of cryptorchidism, but may not be a common cause.
The HOXA10 gene in women with endometriosis was hypomethylated compared to controls.
HOXA10 was expressed at a high level in the K562/ADM (show ADM Proteins) cells, and knockdown of HOXA10 enhances the sensitivity of the K562/ADM (show ADM Proteins) cells to cytotoxic killing by the therapeutic drug, ADR (show AKR1B1 Proteins), as a result of the increased intracellular accumulation of ADR (show AKR1B1 Proteins).
Regulated HOXA10 and HOXA11 (show HOXA11 Proteins) expression is necessary for endometrial receptivity; decreased HOXA10 or HOXA11 (show HOXA11 Proteins) expression leads to decreased implantation rates. Alternation of HOXA10 and HOXA11 (show HOXA11 Proteins) expression has been identified as a mechanism of the decreased implantation associated with endometriosis, polycystic ovarian syndrome, leiomyoma, polyps, adenomyosis, and hydrosalpinx.
The combinatory expression of HOXA10 and CD44 (show CD44 Proteins) was correlated with poor gastric cancer prognosis.
confirmed that HOXA10 promoted epithelialmesenchymal transition in ovarian cancer cells
Promoter activity of HOXA10 lies in 5.3-6.1 kb upstream of protein coding region.CTCF negatively regulates HOXA10 expression in breast cancer cells.CTCF flanks important promoter element of HOXA10.
study identified an E(2)/P(4) response element of the porcine HOXA10 gene for the first time
investigation of regulation of HOXA10 gene expression by estradiol and/or progesterone in porcine endometrium during estrous.
Homeobox A10 expression in the porcine endometrium is closely related to the implantation process and stimulated by conceptus products.
we suggest that proper regional decidualization and polyploidy development requires FoxM1 (show FOXM1 Proteins) signaling downstream of Hoxa10 and cyclin D3 (show CCND3 Proteins).
these studies demonstrate a previously undescribed role for HoxA10 in terminating emergency granulopoiesis, suggesting an important contribution by Hox (show MSH2 Proteins) proteins to the innate immune response.
Hoxa10 cooperates with Nkx2-5 (show NKX2-5 Proteins) to regulate the timing of cardiac mesoderm differentiation.
results show that reduced APC activity is sufficient to induce formation of epithelial inclusion cysts and support ovarian endometrioid adenocarcinoma development and suggest that induced HOXA10 expression and loss of PTEN are key mechanisms driving endometrioid histotype differentiation and progression
a molecular mechanisms through which increased expression of HoxA10 increases Cdx4 expression by direct CDX4 activation and by Fgf2 (show FGF2 Proteins)-induced beta-catenin (show CTNNB1 Proteins) activity. This results in Cdx4-induced HoxA10-expression, creating a positive feedback mechanism
Setbp1 (show SETBP1 Proteins) promotes the self-renewal of murine myeloid progenitors via activation of Hoxa9 (show HOXA9 Proteins) and Hoxa10.
found that increased Fgf2 (show FGF2 Proteins) production by HoxA10-overexpressing myeloid progenitor cells induced a phosphoinositol 3-kinase-dependent increase in beta-catenin (show CTNNB1 Proteins) protein
Results suggest that maternal obesity impairs fetal skeletal development through down-regulation of the HoxA10 gene, which may lead to an increase in the prevalence of low bone mass in the offspring later in life.
Data show that the extra-embryonic function of Hoxa10, -11, and -13 genes stems from their specific expression in the allantois, an extra-embryonic hallmark of amniote vertebrates.
PBX1 (show PBX1 Proteins) plays a central role in attenuating the activity of HOXA10 as an activator of osteoblast-related genes.
In vertebrates, the genes encoding the class of transcription factors called homeobox genes are found in clusters named A, B, C, and D on four separate chromosomes. Expression of these proteins is spatially and temporally regulated during embryonic development. This gene is part of the A cluster on chromosome 7 and encodes a DNA-binding transcription factor that may regulate gene expression, morphogenesis, and differentiation. More specifically, it may function in fertility, embryo viability, and regulation of hematopoietic lineage commitment. Alternatively spliced transcript variants have been described. Read-through transcription also exists between this gene and the downstream homeobox A9 (HOXA9) gene.
, homeobox A10, isoform 1
, homeobox protein Hox-A10-like
, homeo box A10
, homeobox protein 1H
, homeobox protein HOXA10
, homeobox protein Hox-1.8
, homeobox protein Hox-1H
, homeobox protein Hox-A10
, homeobox protein A10