Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Interleukin 17A (IL17A)is a proinflammatory cytokine secreted by activated T-lymphocytes. Additionally we are shipping IL17RA Proteins (32) and IL17RA Kits (11) and many more products for this protein.
Showing 10 out of 191 products:
Human Monoclonal IL17RA Primary Antibody for FACS, ICFC - ABIN2657694
Moseley, Haudenschild, Rose, Reddi: Interleukin-17 family and IL-17 receptors. in Cytokine & growth factor reviews 2003
Show all 5 references for ABIN2657694
Human Polyclonal IL17RA Primary Antibody for EIA, WB - ABIN493067
Fossiez, Djossou, Chomarat, Flores-Romo, Ait-Yahia, Maat, Pin, Garrone, Garcia, Saeland, Blanchard, Gaillard, Das Mahapatra, Rouvier, Golstein, Banchereau, Lebecque: T cell interleukin-17 induces stromal cells to produce proinflammatory and hematopoietic cytokines. in The Journal of experimental medicine 1996
Show all 3 references for ABIN493067
Mouse (Murine) Polyclonal IL17RA Primary Antibody for EIA, WB - ABIN615904
Gaffen, Kramer, Yu, Shen: The IL-17 cytokine family. in Vitamins and hormones 2006
Mouse (Murine) Monoclonal IL17RA Primary Antibody for FACS - ABIN2678059
Toy, Kugler, Wolfson, Vanden Bos, Gurgel, Derry, Tocker, Peschon: Cutting edge: interleukin 17 signals through a heteromeric receptor complex. in Journal of immunology (Baltimore, Md. : 1950) 2006
High IL-17RA expression is associated with Intrahepatic Cholangiocarcinoma.
Identified the IL17RA rs4819554 SNP as a risk factor for psoriasis.
IL-17 receptor A and adenosine deaminase 2 (show CECR1 Antibodies) deficiency due to deletion mutations was found in siblings with chronic mucocutaneous candidiasis and chronic systemic inflammation.
In vitro, CSE stimulation significantly increased IL-17F and IL-17R in 16HBE (2.5%) and A549 (5%) while IL-17A and IL-17F in PBMC (10%). IL-17A and CSE stimulation, rather than CSE or rhIL-17A alone, increased proliferation in 16HBE and apoptosis in A549
The data indicate that the genetic variants of rs2275913 in IL-17A, rs763780 in IL-17F, and rs4819554 in IL-17RA may not play a role in the pathogenesis of preeclampsia in Chinese Han women.
IL17F (show IL17F Antibodies) and IL17RA polymorphisms modulate susceptibility to cerebral malaria(CM) and provide evidence that IL-17F (show IL17F Antibodies) protects against CM.
Increased expression of IL-17RA plays an important role in gastric cancer progression, migration and prognosis.
Both genetic variants of IL-17R (rs882643 and rs2241049) were associated with PGD (show PGD Antibodies). This confirms a genetic predisposition toward PGD (show PGD Antibodies) and suggests a role of IL-17 (show IL17A Antibodies) in driving neutrophilia in PGD (show PGD Antibodies).
The presence of IL7R (show IL7R Antibodies) polymorphisms seems to be related to severe liver disease in HIV/hepatitis c virus coinfected patients.
expression of immunoreactivity for IL-17A (show IL17A Antibodies), IL-17RA, IL-17E (show IL25 Antibodies), and IL-17F (show IL17F Antibodies) was significantly elevated in prostatic tissue from benign prostatic hyperplasia and prostate cancer compared with that in controls
IL-17RA in non hematopoietic cells regulates recruitment of neutrophils to the lung during the adaptive phase of the immune response.
Bronchial epithelial TLR2 (show TLR2 Antibodies)/IL17R signaling crosstalk is necessary for lung IL17 (show IL17A Antibodies) homeostasis and is compromised by morphine.
IL-17 (show IL17A Antibodies) signaling through IL-17RA is an important driver of lung inflammation and airway hyperresponsiveness.
results suggest that the p38alpha (show MAPK14 Antibodies)-MKP-1 (show DUSP1 Antibodies) signaling axis links IL-17R signaling in tissue-resident cells to autoimmune inflammation dependent on infiltrating T(H)17 cells.
There were no significant differences in clinical scores and numbers of infiltrating eosinophils among IL-25KO, TSLPR (show CRLF2 Antibodies) KO, and wild-type mice.
IL-17RA signaling is critical for the development of renal pathology in autoimmune glomerulonephritis.IL-17 enhanced the production of proinflammatory cytokines and chemokines from tubular epithelial cells.
Il17ra appears to be a novel modulator of monocyte phenotype and possible therapeutic target in renal fibrosis
IL-17 (show IL17A Antibodies) expression is higher in C57BL/6 versus BALB/c cornea after infection and that the latter group has more MerTK (show MERTK Antibodies)+ cells.
It was concluded that TLR signaling can directly stimulate the expression of IL-17RA, but not IL-17RC (show IL17RC Antibodies), in neuroglial cells, which functionally respond to IL-17A (show IL17A Antibodies) by secreting chemokines, and accelerating CD4 (show CD4 Antibodies) cell migration.
Absence of IL-17RA leads to a Th2-like phenotype characterized by IL-4 (show IL4 Antibodies) production and suggests that IL-17RA signaling plays a critical role in the regulation of IL-4 (show IL4 Antibodies) in collagen-induced arthritis and development of autoimmune inflammation of the joint.
Interleukin 17A (IL17A)is a proinflammatory cytokine secreted by activated T-lymphocytes. It is a potent inducer of the maturation of CD34-positive hematopoietic precursors into neutrophils. The protein encoded by this gene (interleukin 17A receptor\; IL17RA) is a ubiquitous type I membrane glycoprotein that binds with low affinity to interleukin 17A. Interleukin 17A and its receptor play a pathogenic role in many inflammatory and autoimmune diseases such as rheumatoid arthritis. Like other cytokine receptors, this receptor likely has a multimeric structure.
IL-17 receptor A
, interleukin-17 receptor A