Kinesin Family Member 17 Proteins (KIF17)

Transports vesicles containing N-methyl-D-aspartate (NMDA) receptor 2B along microtubules.. Additionally we are shipping KIF17 Antibodies (25) and many more products for this protein.

list all proteins Gene Name GeneID UniProt
KIF17 57576 Q9P2E2
KIF17 16559 Q99PW8
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Top KIF17 Proteins at

Showing 3 out of 3 products:

Catalog No. Origin Source Conjugate Images Quantity Supplier Delivery Price Details
Insect Cells Human His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Log in to see 70 Days
Insect Cells Mouse His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Log in to see 70 Days
HOST_Wheat germ Human GST tag 10 μg Log in to see 11 to 12 Days

KIF17 Proteins by Origin and Source

Origin Expressed in Conjugate
Human ,
Mouse (Murine)

More Proteins for Kinesin Family Member 17 (KIF17) Interaction Partners

Human Kinesin Family Member 17 (KIF17) interaction partners

  1. The first evidence of an interaction between septins and a nonmitotic kinesin is provided and it is suggested that SEPT9 (show SEPT9 Proteins) modulates the interactions of KIF17 with membrane cargo.

  2. KIF17 can modify RhoA-GTPase signaling to influence junctional actin and the stability of the apical junctional complex of epithelial cells.

  3. Expression of KIF17 in schizophrenic postmortem brains was significantly lower than controls. Both genotypic distribution and allelic frequency of rs2296225 polymorphism were significantly different between the chronic schizophrenia subjects and controls.

  4. although EB1 (show MAPRE2 Proteins) and KIF17-Tail may coordinate KIF17 catalytic activity, our data reveal a novel and direct role for KIF17 in regulating MT dynamics.

  5. This study suggested that disruption of KIF17, although rare, could result in a schizophrenia phenotype and emphasize the possible involvement of rare de novo mutations in this disorder.

  6. Depletion of KIF17 from cells growing in three-dimensional matrices results in aberrant epithelial cysts that fail to generate a single central lumen and to polarize apical markers.

  7. Data show that the homodimeric kinesin-2 (show KIF2A Proteins) motor KIF17 is kept in an inactive state in the absence of cargo, and define two molecular mechanisms that contribute to autoinhibition of KIF17.

  8. The intense placental expression of KIFC1 (show KIFC1 Proteins) in syncytiotrophoblast and KIF17 in vascular endothelium suggests that both proteins might be important in a cargo-transport system. KIFC1 (show KIFC1 Proteins) and KIF17 expression are increased of both in preeclamptia and diabetes.

  9. KIF17b serves as a molecular motor (show MYO1E Proteins) component of a TB-RBP (show TSN Proteins)-mouse ribonucleoprotein complex transporting a group of specific CREM (show CREM Proteins)-regulated mRNAs.

Mouse (Murine) Kinesin Family Member 17 (KIF17) interaction partners

  1. Our findings have uncovered a hitherto unknown effector of Rab23 (show RAB23 Proteins) and demonstrate how Rab23 (show RAB23 Proteins) could mediate the transport of Kif17 to the primary cilium

  2. D1-type dopaminergic receptors are delivered to cilia by a mechanism requiring the receptor cytoplasmic tail, the intraflagellar transport complex-B (IFT-B), and ciliary kinesin KIF17. This targeting mechanism critically depends on Rab23 (show RAB23 Proteins).

  3. KIF17 contributed to the development of bone cancer pain in the spinal cord through NR2B (show GRIN2B Proteins) transport and mLin10 may also play a role in pain development.

  4. findings indicated that CaMKII (show CAMK2G Proteins)-mediated KIF17/NR2B (show GRIN2B Proteins) trafficking may contribute to bone cancer pain, and inhibition of CaMKII (show CAMK2G Proteins) may be a useful alternative or adjunct therapy for relieving cancer pain

  5. prenatal stress significantly decreased the hippocampal KIF17 and NR2B (show GRIN2B Proteins) level in offspring at postnatal stages of 3 weeks and 9 weeks, which may be related to deficits of spatial cognition caused by prenatal stress.

  6. The results of this study suggested that phosphorylation-based regulation of NMDA receptor transport is critical for learning and memory in vivo and regulate by kif17.

  7. This study demonistrated that KIF17 is fundamental for memory and learning via differential support of synaptic NR2A (show GRIN2A Proteins)/2B levels.

  8. NRF-1 (show NRF1 Proteins) co-regulates oxidative enzymes that generate energy and neurochemicals that consume energy related to glutamatergic neurotransmission, such as KIF17, NR1 (show GRIN1 Proteins), and NR2B (show GRIN2B Proteins).

  9. KIF17 contributes to neuronal events required for learning and memory by trafficking fundamental N-methyl-d-aspartate-type glutamate (show GRIN1 Proteins) receptor, NR2A (show GRIN2A Proteins).

  10. ACT selectively associates with KIF17b, a kinesin highly expressed in male germ cells; The ACT-KIF17b interaction is restricted to specific stages of spermatogenesis and directly determines the intracellular localization of ACT [KIF17b]

KIF17 Protein Profile

Protein Summary

Transports vesicles containing N-methyl-D-aspartate (NMDA) receptor 2B along microtubules.

Gene names and symbols associated with KIF17

  • kinesin family member 17 (LOC100341628)
  • kinesin family member 17 (KIF17)
  • kinesin family member 17 (Kif17)
  • 5930435E01Rik protein
  • AW492270 protein
  • KIF3X protein
  • Kif17b protein
  • KLP-2 protein
  • mKIAA1405 protein
  • OSM-3 protein

Protein level used designations for KIF17

KIF17 variant protein , KIF3-related motor protein , kinesin-like protein KIF17 , N-4 kinesin , kinesin 17

100341628 Oryctolagus cuniculus
57576 Homo sapiens
16559 Mus musculus
100688382 Canis lupus familiaris
530435 Bos taurus
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