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The protein encoded by PTH is a hormone secreted by parathyroid cells. Additionally we are shipping PTH Kits (73) and PTH Proteins (65) and many more products for this protein.
Showing 10 out of 403 products:
Human Monoclonal PTH Primary Antibody for ELISA, WB - ABIN1724741
Koshizuka, Ogata, Shiraki, Hosoi, Seichi, Takeshita, Nakamura, Kawaguchi: Distinct association of gene polymorphisms of estrogen receptor and vitamin D receptor with lumbar spondylosis in post-menopausal women. in European spine journal : official publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society 2006
Show all 2 references for ABIN1724741
Human Monoclonal PTH Primary Antibody for EIA, IHC (fro) - ABIN191965
Tampe, Broszio, Manneck, Missbichler, Blind, Müller, Schmidt-Gayk, Armbruster: Characterization of antibodies against human N-terminal parathyroid hormone by epitope mapping. in Journal of immunoassay 1992
Show all 2 references for ABIN191965
Human Monoclonal PTH Primary Antibody for EIA, IHC (p) - ABIN781709
Habener, Rosenblatt, Potts: Parathyroid hormone: biochemical aspects of biosynthesis, secretion, action, and metabolism. in Physiological reviews 1984
Human Monoclonal PTH Primary Antibody for EIA, IHC (fro) - ABIN191904
Mägerlein, Hock, Adermann, Müller-Beckmann, Neidlein, Forssmann, Stein: A new immunoenzymometric assay for bioactive N-terminal human parathyroid hormone fragments and its application in pharmacokinetic studies in dogs. in Arzneimittel-Forschung 1998
In this large community-based cohort, PTH levels, overall, were not independently associated with the risk of hypertension. However, we found some evidence that PTH may be associated with hypertension in blacks.
The objective of this article is to investigate the effect of renin (show REN Antibodies)-angiotensin system inhibitors (RASIs) on intact parathyroid hormone (iPTH) levels in continuous ambulatory peritoneal dialysis (CAPD) patients.
PTH does not contribute to the occurrence of metabolic components of obesity, but there is a positive correlation between 25(OH)D and HDL (show HSD11B1 Antibodies)-C.
Data suggest that effects of PTH on bone remodeling are mediated not only by osteoblasts/osteocytes but also by T-lymphocytes in bone marrow; T-lymphocytes regulate differentiation/life span of stromal cells and their responsiveness to PTH. [REVIEW]
Serum secreted i-PTH level might not be predictable by a total mass of parathyroid glands or by their blood supply.
A Homozygous [Cys25]PTH(1-84) Mutation That Impairs PTH/PTHrP Receptor Activation Defines a Novel Form of Hypoparathyroidism.
Study indicates significant association between specific PTH gene promoter region variants and altered levels of 25(OH)D and vitamin D deficiency among specific nationals.
Elevation of PTH, unlike vitamin D, is independently associated with Chronic Obstructive Pulmonary Disease (COPD (show ARCN1 Antibodies)) severity, and may be a better biomarker for COPD (show ARCN1 Antibodies).
Abnormal diurnal patterns of PTH are associated with sustained mild hypercalcemia in nondialyzed chronic kidney disease patients.
These data indicate that ephrinB2 (show EFNB2 Antibodies)/EphB4 (show EPHB4 Antibodies) signaling within the osteoblast lineage is required for late stages of osteoblast differentiation.
These data suggest that prostaglandin E2 acting via EP4R (show PTGER4 Antibodies) on bone marrow macrophages committed to the osteoblast cell lineage, stimulated secretion of a factor or factors that acted to suppress PTH-stimulated osteoblast differentiation.
These results suggest that in vivo PTH treatment increased in vitro osteoclastogenesis and resorption without altering the number of osteoclast precursors.
Osteoblast 2-deoxyglucose uptake and glycogen (show GYS2 Antibodies) synthesis were increased after exposure to low concentrations (0.1 nmol/l and above) of PTH.
Parathyroid hormone stimulated growth and decreased Col-X deposition via phosphotidylinositol-3,4,5 triphosphate kinase and mitogen activating protein kinase pathways in avian sterna.
likely involvement of the Sp family in regulating PTH gene expression through interactions with an Sp1 (show SP1 Antibodies) DNA element in the hormone's promoter.
alternative cis (show CISH Antibodies)-acting protein-binding elements may determine the regulation of PTH mRNA stability in response to changes in serum calcium and phosphate
a possible role for the Wnt (show WNT2 Antibodies) signaling pathway in PTH actions in bone
Data suggest that calcium-mediated destabilization of parathyroid hormone mRNA requires gene transcription and involves increases in cytosolic Ca.
These results indicate that PTH-mediated inhibition of renal phosphate transport involves phosphorylation of S77 of the NHERF-1 (show SLC9A3R1 Antibodies) PDZ (show INADL Antibodies) I domain and the dissociation of NHERF-1 (show SLC9A3R1 Antibodies)/Npt2a (show SLC34A1 Antibodies) complexes.
It was concluded that endogenously secreted PTH and GHR (show GHR Antibodies) signaling in bone are necessary to establish radial bone growth and optimize mineral acquisition during growth.
findings suggest that XLalphas (show GNAS Antibodies) enhances Gq/11 signaling to mediate the renal actions of PTH during early postnatal development.
bone adaptation during exercise is not only a function of dynamic loading, but also PTH release, and that PTH signaling contributes differently at the structural and tissue levels.
IGF1 (show IGF1 Antibodies) signaling plays a greater role in the skeletal actions of cPTH in the female mouse than in the male mouse, which may underlie the sex differences in the response to cPTH.
Downregulation of PTH receptor expression mediated by intracellular oxidant stress as the mechanisms in hyperlipidemia-induced PTH resistance.
Chondrogenesis induced by PTH in mesenchymal progenitor cells is mediated by Runx1 (show RUNX1 Antibodies), which involves the activation of PKA.
PTH is not required to upregulate renal expression of Cyp27b1 (show CYP27B1 Antibodies) during pregnancy or to stimulate recovery from loss of bone mineral content caused by lactation.
PTH deficiency itself impairs osteogenesis, osteoclastogenesis, and osteoclastic bone resorption, whereas subsequent upregulation of PTHrP (show PTHLH Antibodies) in osteogenic cells compensates by increasing bone accrual.
Induced formation of aberrant crypt foci (ACF (show A1CF Antibodies)) caused by azoxymethane (AOM (show COL2A1 Antibodies)) injection in 'rescued' CaSR (show CASR Antibodies)-/PTH- (C-/P-) double knockout colons compared with colons from control CaSR (show CASR Antibodies)+/PTH+ (C+/P+) mice to study the role of CaSR (show CASR Antibodies).
The protein encoded by this gene is a hormone secreted by parathyroid cells. This hormone elevates blood Ca2+ level by dissolving the salts in bone and preventing their renal excretion. Defects in this gene are a cause of familial isolated hypoparathyroidism (FIH).
, parathyroid hormone 1
, preproparathyroid hormone
, hypothalamic parathyroid hormone
, thyroid hormone
, probable peptidyl-tRNA hydrolase