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Browse our ENOS Proteins (NOS3)

Full name:
Nitric Oxide Synthase 3 (Endothelial Cell) Proteins (NOS3)
On www.antibodies-online.com are 23 Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Proteins from 7 different suppliers available. Additionally we are shipping ENOS Antibodies (469) and ENOS Kits (92) and many more products for this protein. A total of 598 ENOS products are currently listed.
Synonyms:
2310065A03Rik, cNOS, EC-NOS, ecNOS, ENOS, NOS, Nos-3, NOS3, NOSIII
list all proteins Gene Name GeneID UniProt
NOS3 4846 P29474
NOS3 18127 P70313
NOS3 24600 Q62600

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Human Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. eNOS does not play an important role in this cerebrocortical blood flow redistribution after carotid artery occlusion.

  2. there were significant decreases in intercellular adhesion molecules 1 (ICAM1 (show ICAM1 Proteins)), ezrin (EZR (show EZR Proteins)), mitogen-activated protein kinase kinase 2 (MAP2K2 (show MAP2K2 Proteins)), and nitric oxide synthase 3 (NOS3 (show NANOS3 Proteins)) gene expressions in metabolic syndrome patients.

  3. Activation of eNOS by HDL (show HSD11B1 Proteins) is decreased in MetS (show ETV3 Proteins) patients before the appearance of diabetes mellitus and that sphingosine-1-phosphate depletion of HDL (show HSD11B1 Proteins) is the main factor responsible for this defect.

  4. The thrombotic predisposition to arterial ischemia of the lower limbs in type 2 diabetic patients is assessed by evaluating polymorphisms in the three major platelet membrane adhesion receptors and endothelial nitric oxide synthase.

  5. In conclusion, exenatide significantly improves coronary endothelial function in patients with newly diagnosed type 2 diabetes. The effect may be mediated through activation of AMPK (show PRKAA1 Proteins)/PI3K (show PIK3CA Proteins)-Akt (show AKT1 Proteins)/eNOS pathway via a GLP-1R (show GLP1R Proteins)/cAMP-dependent mechanism.

  6. The downregulation of beta-arrestins 1/2 in saphenous vein endothelial cells (SVECs) prevented the shear stress-induced rise in levels of phosphorylation of Akt (show AKT1 Proteins) and endothelial nitric oxide synthase (eNOS, Serine 1177).

  7. PGC-1alpha overexpression improves AngII-induced eNOS dysfunction via activated PI3K (show PIK3CA Proteins)/Akt (show AKT1 Proteins) pathway, impaired PP2A (show PPP2R4 Proteins) activity and reduced PP2A (show PPP2R4 Proteins)-A/eNOS association.

  8. eNOS G894T and T-786C SNPs were both significantly correlated with hypertension. Additionally, the T allele of G894T SNP and C allele of T-786C SNP may serve as potential biological markers for hypertension susceptibility in Asians. [Meta-Analysis}

  9. Results suggest a relation between different endothelial nitric oxide synthase geno-types and risk of acute rejection episodes in liver transplant recipients.

  10. The presence of the eNOS allele had a significantly negative impact on responsiveness to a selective alpha1 -blocker in benign prostatic hyperplasia related lower urinary tract symptoms treatment, suggesting that eNOS G894T gene polymorphism may be a genetic susceptibility factor for alpha1 -blocker efficacy in men with benign prostatic hyperplasia related lower urinary tract symptoms

Pig (Porcine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. NOS3 was lowest in kidneys removed from live pigs, greater in kidneys from pigs with brain death, and greatest in kidneys from pigs with cardiac arrest.

  2. Rapid atrial pacing increases ADMA and down-regulates eNOS expression in an ADMA-independent manner.

  3. Icariin and icariside II may increase the eNOS expression through activating EGF-EGFR (show EGFR Proteins) pathway in porcine aortic endothelial cells.

  4. Data suggest that pig sperm contain bNOS (show NOS1 Proteins), iNOS (show NOS2 Proteins), and eNOS; up-regulation of NOS by leptin (show LEP Proteins) during acrosome reaction and inhibition of acrosome reaction by inhibitors of nitric oxide synthases suggests these enzymes are involved in acrosome reaction.

  5. Periodic acceleration (pGz) acutely increases endothelial and neuronal nitric oxide synthase (show NOS1 Proteins) expression in endomyocardium of normal swine.

  6. Data suggest that angiotensin II regulates nNOS and eNOS expression and NOS activity in afferent arterioles of the developing kidney via angiotensin 1 and 2 receptors.

  7. Endothelial nitric oxide synthase mRNA expression was elevated in gestational day 50 intrauterine growth retardation placenta and areola compared to gd50 control.

  8. Oligonol prevented the impairment of eNOS activity induced by high glucose through reversing altered eNOS phosphorylation status.

  9. Exercise training significantly increased total eNOS and phosphorylated levels of eNOS (pSer(1179)) in collateral-dependent arteries of experimental minipigs.

  10. Data show that wall shear stress increases with a decrease in artery diameter; eNOS protein contents decrease with an increase in diameter.

Cow (Bovine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. TNFalpha (show TNF Proteins)reduces eNOS activity in bovine endothelial cells through serine 116 phosphorylation and Pin1 binding.

  2. Pomanox supplementation hinders hyperlipemia-induced coronary endothelial dysfunction by activating the Akt (show AKT1 Proteins)/endothelial nitric oxide-synthase pathway and favorably counteracting vascular inflammation and oxidative damage.

  3. Signals that activate and phosphorylate eNOS are transmitted through distinct membrane domains in endothelial cells. Cholesterol domains, but not individual caveolae, mediate HDL stimulation of eNOS. VEGF and shear stress may act through caveolae.

  4. eNOS serine 1179 phosphorylation, in addition to enhancing NO production, also profoundly affects superoxide generation

  5. In addition to the heme center of eNOS oxygenase domain, we confirmed another O2.- generation site in the eNOS reductase domain and characterized its regulatory properties.

  6. A dimer-destabilized mutant of bovine eNOS where cysteine 101 was replaced by alanine, cloned and introduced into an eNOS-deficient mouse strain and that results provide the first in vivo evidence that eNOS-dependent oxidative stress is unlikely to be an initial cause of impaired endothelium-dependent vasodilation and/or a pathologic factor promoting intimal hyperplasia.

  7. Data show that resveratrol (Res) reversed caveolin-1 (Cav-1 (show CAV1 Proteins))/endothelial nitric oxide synthase (eNOS) expressions in high glucose cultured bovine aortic endothelial cells (BAECs).

  8. Fluorescence decays of fluorescently labeled CaM (show KRIT1 Proteins) bound to eNOS reveal four distinct conformational states and single-molecule fluorescence trajectories show multiple fluorescence states with transitions between states

  9. Endothelial nitric oxide synthase is regulated by ERK (show MAPK1 Proteins) phosphorylation at Ser602.

  10. key regulatory role of CaM (show KRIT1 Proteins) involves the stabilization of structural intermediates and precise positioning of the pivot for the FMN (show FMN1 Proteins) domain tethered shuttling motion to accommodate efficient and rapid electron transfer in the homodimer of eNOS.

Rabbit Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. VEGFR2 (show KDR Proteins) activation was not affected by Slit2 (show SLIT2 Proteins), but eNOS phosphorylation was diminished

  2. Data suggest distinct localizations of eNOS at the spiral arteries/arterial sinuses and iNOS (show NOS2 Proteins) along the radial arteries in the developing placenta.

  3. Pulmonary ischaemia-reperfusion up-regulates inducible nitric oxide synthesis and/activity, which coincides with reduced endothelial nitric oxide synthase activity.

  4. eNOS dysregulation may be a central mechanism of impaired cardioprotection during hyperglycemia.

  5. Quercetin inhibited myocardial ischemia-reperfusion-induced NOS3 mRNA and protein expression.

Mouse (Murine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. these data suggest that CD NOS3 may be involved in the diuretic response to a water load in a sex-specific manner; the mechanism of this effect remains to be determined.

  2. CAV1 (show CAV1 Proteins) KO mice have elevated IOP and reduced conventional outflow facility when compared with WT mice. CAV2 (show CAV2 Proteins) expression was absent in CAV1 (show CAV1 Proteins) KO mice, but we observed increased expressions of eNOS

  3. TNF-alpha (show TNF Proteins) does not regulate eNOS activity in murine endothelial cells through serine 116 phosphorylation and Pin1 (show PIN1 Proteins) binding.

  4. The eNOS expressed in smooth muscle cells in FAs (show FAS Proteins) attenuates alpha-adrenergic vasoconstriction.

  5. Thioredoxin (show TXN Proteins)-mediated deglutathionylation of eNOS in the coronary artery in vivo protected against reperfusion injury, even in the presence of normal levels of glutathione.

  6. Eph (show EPHA1 Proteins)-B4 stimulates eNOS phosphorylation in vitro and may mediate vein graft adaptation by regulation of eNOS activity in vivo.

  7. Loss of endothelial NO plays an important role in the generation of p25 (show CDK5R1 Proteins) and resulting tau phosphorylation in neuronal tissue. Endothelial nitric oxide synthase (eNOS)-deficient (eNOS-/-) mice display increased levels of p25 (show CDK5R1 Proteins), an aberrant activator of cyclin-dependent kinase 5 (show CDK5 Proteins), which is one of the primary kinases responsible for tau hyperphosphorylation, and a statistically higher p25/p35 (show CDK5R1 Proteins) ratio.

  8. Pulse pressure finely regulates eNOS, controlling cerebral artery reactivity.

  9. TNF-alpha (show TNF Proteins) induces vascular insulin (show INS Proteins) resistance by mechanisms that involve positive modulation of PTEN and inhibition of Akt (show AKT1 Proteins)/eNOS/NO signaling.

  10. Beta2AR (show ADRB2 Proteins) overexpression enhances endothelial progenitor cells (EPC (show TCF21 Proteins)) functions in vitro and enhances the vascular repair abilities of EPCs in vivo via the beta2AR (show ADRB2 Proteins)/Akt (show AKT1 Proteins)/eNOS pathway.

Guinea Pig Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. The present evidence indicated that the customary HBOT protocol may increase constitutive NOS expression.

ENOS (NOS3) Protein Profile

Protein Summary

Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. Nitric oxide is synthesized from L-arginine by nitric oxide synthases. Variations in this gene are associated with susceptibility to coronary spasm. Multiple transcript variants encoding different isoforms have been found for this gene.

Alternative names and synonyms associated with ENOS (NOS3)

  • nitric oxide synthase 3 (endothelial cell) (NOS3)
  • nitric oxide synthase 3 (endothelial cell) (nos3)
  • endothelial nitric oxide synthase (23B)
  • nitric oxide synthase 3, endothelial cell (Nos3)
  • 2310065A03Rik protein
  • cNOS protein
  • EC-NOS protein
  • ecNOS protein
  • ENOS protein
  • NOS protein
  • Nos-3 protein
  • NOS3 protein
  • NOSIII protein

Protein level used designations for NOS3

nitric oxide synthase 3 (endothelial cell) , nitric oxide synthase, endothelial-like , EC-NOS , NOS type III , NOSIII , cNOS , constitutive NOS , endothelial NOS , nitric oxide synthase, endothelial , eNOS , endothelial nitric oxide synthase , endothelial nitric oxide synthase NOS3 , endothelial nitric oxide synthase 3

GENE ID SPECIES
714231 Macaca mulatta
100018035 Monodelphis domestica
100486016 Xenopus (Silurana) tropicalis
443077 Ovis aries
4846 Homo sapiens
403784 Canis lupus familiaris
397557 Sus scrofa
287024 Bos taurus
443073 Ovis aries
100063339 Equus caballus
100009498 Oryctolagus cuniculus
18127 Mus musculus
24600 Rattus norvegicus
100135577 Cavia porcellus
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