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anti-Mouse (Murine) Renin Antibodies:
anti-Rat (Rattus) Renin Antibodies:
anti-Human Renin Antibodies:
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Human Polyclonal Renin Primary Antibody for IF (p), IHC (p) - ABIN715436
Wanka, Staar, Lutze, Peters, Hildebrandt, Beck, Bäumgen, Albers, Krieg, Zimmermann, Sczodrok, Schäfer, Hoffmann, Peters: Anti-necrotic and cardioprotective effects of a cytosolic renin isoform under ischemia-related conditions. in Journal of molecular medicine (Berlin, Germany) 2015
Show all 2 references for ABIN715436
Mouse (Murine) Polyclonal Renin Primary Antibody for EIA, IHC (p) - ABIN264819
Carilli, Wallace, Smith, Wong, Lewicki: Semi-preparative purification of recombinant human renin and prorenin. in Journal of chromatography 1989
Show all 2 references for ABIN264819
Human Monoclonal Renin Primary Antibody for ELISA, WB - ABIN562625
Salhan, Husain, Subrati, Goyal, Singh, Rai, Malhotra, Singhal: HIV-induced kidney cell injury: role of ROS-induced downregulated vitamin D receptor. in American journal of physiology. Renal physiology 2012
Human Polyclonal Renin Primary Antibody for EIA, WB - ABIN497962
Lavoie, Liu, Bianco, Beltz, Johnson, Sigmund: Evidence supporting a functional role for intracellular renin in the brain. in Hypertension 2006
Human Polyclonal Renin Primary Antibody for ELISA, WB - ABIN1535288
Hardman, Hort, Catanzaro, Tellam, Baxter, Morris, Shine: Primary structure of the human renin gene. in DNA (Mary Ann Liebert, Inc.) 1985
Results show negative regulation of HDAC1 (show HDAC1 Antibodies) by a Cul3 (show CUL3 Antibodies)-REN (show REN1 Antibodies) E3 ubiquitin ligase (show MUL1 Antibodies) complex.
REN is upregulated by neurogenic signals (retinoic acid, EGF, and NGF) in embryonal stem (ES) cells and neural progenitor cell lines in association with neurotypic differentiation.
REN (show REN1 Antibodies) protein enhances caspase-3 (show CASP3 Antibodies) activation and antagonizes the Shh (show SHH Antibodies) pathway suggesting that this gene may represent a restraint of Shh (show SHH Antibodies) signaling.
This study demonstrates the efficacy of aldosterone/direct renin concentration ratio as a screening test for primary aldosteronism.
The dominant model (CC vs. CT+TT) of rs1894111 polymorphism in the ADRBK1 gene might be associated with low-renin hypertension in Han Chinese.
Data show that the optimum time for beta-adrenoreceptor antagonists (beta-blockers) withdrawal was 2 weeks when using direct renin concentration (DRC (show FECH Antibodies)) and 3 weeks for plasma renin activity (PRA (show S100A6 Antibodies)).
Active renin was not significantly different between hypertensive patients with and without left ventricular hypertrophy.
The present study provides evidence that the rs1464816 polymorphism in REN is associated with CKD progression in ADPKD.
Plasma renin and Aldosterone to renin ratio but not aldosterone are independently associated with presence of carotid plaques in coronary artery disease patients.
concentrations of serum aldosterone, plasma direct renin, and plasma renin activity were measured after a night's rest and again after walking for two hours.
Increased plasma renin, AT and VEGF might play a role in the onset or development of superficial infantile hemangiomas.
Elevated levels of circulating (pro)renin were observed in pre-eclampsia patients
The results suggest that variants in the enhancer region of the human renin gene have an effect on the expression levels of renin in renal tissue; this observation is in good accordance with the results of the transcriptional assay.
Renin catalyzes the first step in the activation pathway of angiotensinogen--a cascade that can result in aldosterone release,vasoconstriction, and increase in blood pressure. Renin, an aspartyl protease, cleaves angiotensinogen to form angiotensin I, which is converted to angiotensin II by angiotensin I converting enzyme, an important regulator of blood pressure and electrolyte balance. Transcript variants that encode different protein isoforms and that arise from alternative splicing and the use of alternative promoters have been described, but their full-length nature has not been determined. Mutations in this gene have been shown to cause familial hyperproreninemia.
, BTB/POZ domain-containing protein KCTD11
, retinoic acid, EGF, and NGF upregulated
, renin 1 structural
, angiotensin-forming enzyme
, renin precursor, renal