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anti-Human NOD2 Antibodies:
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Human Monoclonal NOD2 Primary Antibody for FACS, ICC - ABIN151959
McDonald, Chen, Ollendorff, Ogura, Marchetto, Lécine, Borg, Nuñez: A role for Erbin in the regulation of Nod2-dependent NF-kappaB signaling. in The Journal of biological chemistry 2005
Show all 13 Pubmed References
Human Polyclonal NOD2 Primary Antibody for ICC, IF - ABIN153051
Till, Rosenstiel, Bräutigam, Sina, Jacobs, Oberg, Seegert, Chakraborty, Schreiber: A role for membrane-bound CD147 in NOD2-mediated recognition of bacterial cytoinvasion. in Journal of cell science 2008
Show all 5 Pubmed References
Human Polyclonal NOD2 Primary Antibody for ELISA, WB - ABIN251672
Keshav: Paneth cells: leukocyte-like mediators of innate immunity in the intestine. in Journal of leukocyte biology 2006
Show all 3 Pubmed References
Human Polyclonal NOD2 Primary Antibody for IHC (fro), IHC (p) - ABIN4340262
Lin, Song, Qin, Li, Li, Zhu, Zhang: Expression of nucleotide-binding oligomerization domain 2 in normal human dental pulp cells and dental pulp tissues. in Journal of endodontics 2009
Show all 3 Pubmed References
Human Polyclonal NOD2 Primary Antibody for IHC (fro), WB - ABIN2475899
Goolamali, Evered, Shuster: Thyroid disease and sebaceous function. in British medical journal 1976
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Human Polyclonal NOD2 Primary Antibody for ELISA, ICC - ABIN4340247
Rahman, Midtling, Svingen, Xiong, Bell, Tung, Smyrk, Egan, Faubion: The pathogen recognition receptor NOD2 regulates human FOXP3+ T cell survival. in Journal of immunology (Baltimore, Md. : 1950) 2010
Human Polyclonal NOD2 Primary Antibody for IP, WB - ABIN4340260
Chauhan, Marriott: Differential roles for NOD2 in osteoblast inflammatory immune responses to bacterial pathogens of bone tissue. in Journal of medical microbiology 2010
Human Monoclonal NOD2 Primary Antibody for FACS, ICC - ABIN4340264
Subramanian, Bergman, Henriques-Normark: Vitamin D Promotes Pneumococcal Killing and Modulates Inflammatory Responses in Primary Human Neutrophils. in Journal of innate immunity 2017
NOD2 is an important mediator in the viral uptake and inflammatory response during the pathogenesis of CVB3 myocarditis.
We identified 2 missense mutations at 16q12 in NOD2 (p.Ala110Thr and p.Arg311Trp), which encodes nucleotide-binding oligomerization domain protein 2. We further examined 94 genetically unrelated AgP patients by targeted sequencing of NOD2 and found that 2 patients among them also carried the p.Arg311Trp variant. Furthermore, we found 3 additional missense mutations in this gene (p.His370Tyr, p.Arg459Cys, and p.Ala868Thr)
Performed genetic analysis in patients with Crohn's disease (CD) to determine effect of NOD2/CARD15 polymorphisms on treatment response to adalimumab and infliximab.
The study of the correlation between genotype and phenotypic expression showed that CARD15 mutation is associated with ileocecal Crohn's disease, with fistulizing and stenosing behavior in Crohn's disease as well as with severe evolution
Ligand-driven triggering of TLR-3 (show TLR3 Antibodies), -4, NOD2, and DC-SIGN (show CD209 Antibodies), despite reducing viral replication, markedly increased the capacity of infected dendritic cells to stimulate HIV-specific cytotoxic T-cells.
This work suggests that Crohn's Disease-associated Nod2 variants could be stabilized in vivo with a molecular chaperone (show HSP90AA1 Antibodies).
From the results, the authors conclude that SNPs at the NOD2 and IL6 (show IL6 Antibodies) genes are associated with leprosy reactions as an outcome.
NOD2 rs3135500 and IL12B (show IL12B Antibodies) rs1368439 SNPs were not genetic risk factors for colorectal cancer in the studied Iranian population.
Variants of NOD2/CARD15 and TLR4 (show TLR4 Antibodies) do not confer susceptibility to inflammatory bowel disease, but appear to determine Crohn's disease phenotypes in this southeastern Brazilian population.
IFNgamma positively affects NOD2-mediated signaling in human conventional dendritic cells, in a manner considerably dependent on XIAP (show XIAP Antibodies) and partially dependent on mTOR (show FRAP1 Antibodies).
Results show that the simultaneous absence of Nod1 (show NOD1 Antibodies) and Nod2 is associated with accelerated T cell death upon alloantigen encounter, suggesting these proteins might provide new targets to ameliorate T cell responses in a variety of inflammatory states, including those associated with bone marrow or solid organ transplantation.
These data suggest that NOD2 contributes to bone loss in estrogen deficiency by elevating reactive oxygen species levels in OCs.
this study shows that the effect of the gut (show GUSB Antibodies) microbiota on bone is dependent on NOD1 (show NOD1 Antibodies) and NOD2 signaling
this study shows that microbial sensing by Nod2 is an important mechanism to regulate small intestinal mucosal damage following acute T cell activation
results demonstrate that the recognition of bacterial products by NOD2 inside the PLNs contributes to T1D development, establishing a new putative target for intervention during the early stages of the disease.
These results indicate glycine enhances muscle protein mass under an inflammatory condition. The beneficial roles of glycine on the muscle are closely associated with maintaining Akt (show AKT1 Antibodies)-mTOR (show FRAP1 Antibodies)-FOXO1 (show FOXO1 Antibodies) signaling and suppressing the activation of TLR4 (show TLR4 Antibodies) and/or NOD2 signaling pathways.
this study shows that innate immunity activation by muramyl peptides is mediated via an interaction between YB1 (show YBX1 Antibodies) and NOD2
this study shows that NOD2 is important for osteoclast differentiation and inflammatory bone resorption in vivo and also for the macrophage response to Gram-negative bacteria
this study reveals that LRRK2 (show LRRK2 Antibodies) is a new positive regulator of Rip2 (show ARHGDIG Antibodies) and promotes inflammatory cytokine induction through the Nod1 (show NOD1 Antibodies)/2-Rip2 (show ARHGDIG Antibodies) pathway.
Data indicate six single-nucleotide polymorphisms (SNPs) in the caspase recruitment domain 15 protein (CARD15) gene associated with susceptibility to bovine tuberculosis (BTB) in Chinese Holstein cows.
Data indicate that the Nod2 signaling adaptor protein (NOD2) G-->A at position 1594 bp plays a critical role in increasing 305-day milk yields.
Genetic variation and putative regulatory regions in bovine CARD15.
A significant association was found between two polymorphisms of CARD15 and paratuberculosis status; cows with the heterozygous genotype were 3.35 times more likely to be infected than cows with the reference genotype.
This gene is a member of the Nod1/Apaf-1 family and encodes a protein with two caspase recruitment (CARD) domains and six leucine-rich repeats (LRRs). The protein is primarily expressed in the peripheral blood leukocytes. It plays a role in the immune response to intracellular bacterial lipopolysaccharides (LPS) by recognizing the muramyl dipeptide (MDP) derived from them and activating the NFKB protein. Mutations in this gene have been associated with Crohn disease and Blau syndrome.
caspase recruitment domain 15 protein
, NLR family, CARD domain containing 2
, NOD-like receptor C2
, caspase recruitment domain family, member 15
, caspase recruitment domain protein 15
, caspase recruitment domain-containing protein 15
, inflammatory bowel disease protein 1
, nucleotide-binding oligomerization domain 2
, nucleotide-binding oligomerization domain, leucine rich repeat and CARD domain containing 2
, nucleotide-binding oligomerization domain-containing protein 2