Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species
Mouse (Murine) APOC1 ELISA Kit for Sandwich ELISA - ABIN415689
Moxon, Liu, Moran, Crossman, Krishna, Yonglitthipagon, Emeto, Morris, Padula, Mulvenna, Rush, Golledge: Proteomic and genomic analyses suggest the association of apolipoprotein C1 with abdominal aortic aneurysm. in Proteomics. Clinical applications 2014
apoC-I and apoC-III (show APOC3 ELISA Kits) inhibit lipolysis by displacing LPL (show LPL ELISA Kits) from lipid emulsion particles. We also propose a role for these apolipoproteins in the irreversible inactivation of LPL (show LPL ELISA Kits) by factors such as angptl4 (show ANGPTL4 ELISA Kits).
These findings indicated that variants in TOMM40 (show TOMM40 ELISA Kits)/APOE (show APOE ELISA Kits)/APOC1 region might be associated with human longevity. Further studies are needed to identify the causal genetic variants influencing human longevity.
These results suggest that ApoC-I peptides may be a potential diagnostic biomarker and therapeutic approach for breast cancer.
APOC1 SNP is associated with the A beta (show SUCLA2 ELISA Kits)-42 levels in CSF (show CSF2 ELISA Kits).
This sbioinformatics analysis explored the shared genetic etiology underlying Type 2 Diabetes and Alzheimer's Disease on SNP level, gene level, and pathway level. Six SNPs on APOC1 gene.
The endogenous retroviral promoters (LTRs) of the human endothelin B receptor (EDNRB (show EDNRB ELISA Kits)) and apolipoprotein C1 (APOC1) genes have high sequence identity but differ in activity and tissue specificity.
The ability of apoC1 to inhibit CETP (show CETP ELISA Kits) activity is impaired in patients with diabetes. Glycation of apoC1 leads to a change in its electrostatic properties that might account, at least in part, for a loss of constitutive CETP (show CETP ELISA Kits) inhibition and an increase in plasma CETP (show CETP ELISA Kits) activity in patients with diabetes.
APOE (show APOE ELISA Kits) e4 allele status is associated with dementia and severity of Alzheimer's disease pathologic features in Parkinson disease.
Data indicate that apolipoprotein C-I (APOC1) rs11568822 polymorphism was associated with increased Alzheimer's disease (AD) risk in Caucasians, Asians and Caribbean Hispanics, but not in African Americans.
Helical domains that mediate lipid solubilization and ABCA1 (show ABCA1 ELISA Kits)-specific cholesterol efflux in apolipoproteins C-I and A-II
apoC-I and apoC-III (show APOC3 ELISA Kits) inhibit lipolysis by displacing LPL (show LCP1 ELISA Kits) from lipid emulsion particles. We also propose a role for these apolipoproteins in the irreversible inactivation of LPL (show LCP1 ELISA Kits) by factors such as angptl4 (show ANGPTL4 ELISA Kits).
Apolipoprotein C-I was significantly increased in obese mice plasma.
The absence of ApoC-I results in impaired memory functions, which is, together with previous data, suggestive of an important, bell-shaped gene-dose dependent role for ApoC-I in appropriate brain functioning
Data show that the stimulating effect of apoCI on the lipopolysaccharide (LPS (show TLR4 ELISA Kits))response resembles that of LPS-binding protein (LBP (show LBP ELISA Kits)) and depends on CD14 (show CD14 ELISA Kits)/ Toll-like receptor 4 (show TLR4 ELISA Kits) signaling.
present observations provide direct support for a potent specific inhibition of CETP (show CETP ELISA Kits) by plasma apoCI in vivo
The apoC-I content of lipoprotein remnants may serve as an early marker of coronary artery disease risk.
TR4 (show NR2C2 ELISA Kits) can also regulate apolipoprotein E (show APOE ELISA Kits), C-I, and C-II gene expression via the TR4 (show NR2C2 ELISA Kits) response element within the hepatic control region
apoC-I is a potent inhibitor of LPL (show LPL ELISA Kits)-mediated triglyceride lipolysis
irrespective of receptor-mediated remnant clearance by the liver, liver-specific expression of recombinant human apoCI causes hypertriglyceridemia in the absence of the VLDLr (show VLDLR ELISA Kits) and apoCIII (show APOC3 ELISA Kits) in mice
Endogenous apoC-I increases hyperlipidemia in apoE (show APOE ELISA Kits)-knockout mice by stimulating VLDL production and inhibiting LPL (show LPL ELISA Kits).
Systemic apoCI increases atherosclerosis, probably by inducing hyperlipidemia, in the absence of ApoE (show APOE ELISA Kits)
Cholesteryl ester transfer protein (show CETP ELISA Kits) is the sole major determinant of cholesteryl ester transfer in normolipidemic rabbit plasma as a result of the inability of rabbit apoCI to change HDL (show HSD11B1 ELISA Kits) electronegativity.
The protein encoded by this gene is a member of the apolipoprotein C1 family. This gene is expressed primarily in the liver, and it is activated when monocytes differentiate into macrophages. A pseudogene of this gene is located 4 kb downstream in the same orientation, on the same chromosome. This gene is mapped to chromosome 19, where it resides within a apolipoprotein gene cluster. Alternatively spliced transcript variants have been found for this gene, but the biological validity of some variants has not been determined.
, Apolipoprotein C-I
, apolipoprotein C1
, liver regeneration-related protein LRRG04