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Homozygous absence of SMN1 (show SMN1 ELISA Kits) exons 7 and 8, or exon 7 only, was found in 80% of childhood spinal muscular atrophy patients. Of those patients, 45% were also deleted for NAIP exon 5.
NAIP and survivin (show BIRC5 ELISA Kits) expressions were significantly reduced following varicocele induction when compared to sham animals whereas PDRN-treated rats showed an increase in NAIP and survivin (show BIRC5 ELISA Kits) levels.
The copy numbers and gene structures of NAIP genes were different in Chinese spinal muscular atrophy patients and healthy controls
results revealed that SMN2 (show SMN1 ELISA Kits) and NAIP copy numbers significantly influenced the age at onset, risk of death, and life expectancy in the spinal muscular atrophy patients and that the effect of SMN2 (show SMN1 ELISA Kits) was more significant
human Naip functions to activate the inflammasome in response to flagellin (show FliC ELISA Kits), similar to murine Naip5 (show BIRC2 ELISA Kits)/6.
Modulation of chemotherapeutic drug resistance in neuroblastoma (show ARHGEF16 ELISA Kits) SK-N-AS cells by the neural apoptosis inhibitory protein and miR (show MLXIP ELISA Kits)-520f.
Copy number variations of SMN2 (show SMN1 ELISA Kits) and NAIP genes in patients are related to spinal muscular atrophy clinical types (P < 0.05).
/NAIP1 and NAIP2/5 formed a large oligomeric complex with NLRC4 (show NLRC4 ELISA Kits) in the presence of corresponding bacterial ligands, and could support reconstitution of the NLRC4 (show NLRC4 ELISA Kits) inflammasome in a ligand-specific manner.
identified an intronic region of the NAIP gene responding to TEAD1 (show TEAD1 ELISA Kits)/YAP (show YAP1 ELISA Kits) activity, suggesting that regulation of NAIP by TEAD1 (show TEAD1 ELISA Kits)/YAP (show YAP1 ELISA Kits) is at the transcriptional level
the NAIP5 (show BIRC2 ELISA Kits)-NLRC4 (show NLRC4 ELISA Kits) inflammasome is induced by direct interactions with conserved N- and C-terminal regions of flagellin (show FliC ELISA Kits)
There is a close relationship between SMN2 (show SMN1 ELISA Kits), NAIP and H4F5 gene copy number and spinal muscular atrophy disease severity
This gene is part of a 500 kb inverted duplication on chromosome 5q13. This duplicated region contains at least four genes and repetitive elements which make it prone to rearrangements and deletions. The repetitiveness and complexity of the sequence have also caused difficulty in determining the organization of this genomic region. This copy of the gene is full length\; additional copies with truncations and internal deletions are also present in this region of chromosome 5q13. It is thought that this gene is a modifier of spinal muscular atrophy caused by mutations in a neighboring gene, SMN1. The protein encoded by this gene contains regions of homology to two baculovirus inhibitor of apoptosis proteins, and it is able to suppress apoptosis induced by various signals. Alternatively spliced transcript variants encoding distinct isoforms have been found for this gene.
baculoviral IAP repeat-containing 1
, baculoviral IAP repeat-containing protein 1
, neuronal apoptosis inhibitory protein
, nucleotide-binding oligomerization domain, leucine rich repeat and BIR domain containing 1
, psi neuronal apoptosis inhibitory protein
, NLR family, apoptosis inhibitory protein 2
, baculoviral IAP repeat-containing 1b