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SATB1 reprograms the expression of tumor growth- and metastasis-associated genes to promote tumorigenesis and functionally overlaps with Wnt (show WNT2 ELISA Kits) signaling critical for colorectal cancer tumorigenesis.
SATB1 as a Dual Regulator of Anti-Apoptotic BCL2 (show BCL2 ELISA Kits) and Pro-Apoptotic NOXA (show PMAIP1 ELISA Kits) Genes
SATB1 protein expression showed an increasing trend in advancing stages of breast cancer development.
replicative oncolytic adenovirus armed with SATB1 shRNA exhibits effective antitumor effect in human prostate cancer. Our study provides the basis for the development of ZD55-SATB1 for the treatment of prostate cancer
Immunohistochemical expression of SATB1 and SATB2 (show SATB2 ELISA Kits) was analysed in tissue microarrays with primary tumours and a subset of paired lymph node metastases from 175 patients operated with pancreaticoduodenectomy for periampullary adenocarcinoma.
expression of SATB1 may increase the size of the BCSC population via the activation of the Notch (show NOTCH1 ELISA Kits) signaling pathway and by increasing expression levels of Snail1 (show SNAI1 ELISA Kits) and Twist1 (show TWIST1 ELISA Kits).
SATB1 and HER2 (show ERBB2 ELISA Kits) expression correlated with poorly differentiated breast cancer and indicated an unfavorable prognosis.
SATB1 is overexpressed in pancreatic cancer, promoting cancer cell proliferation and invasion through the activation of MYC (show MYC ELISA Kits).
Up-regulation of histidine-rich calcium binding protein (show HRC ELISA Kits) promotes tumor metastasis in hepatocellular carcinoma and is mediated by SATB1.
results suggest that SATB1 plays a crucial role in the progression of bladder cancer by regulating genes controlling EMT (show ITK ELISA Kits) processes
These results suggest that SATB1 plays an essential role in establishment of immune tolerance.
Data show that RNA binding protein HuD and special adenine-thymine (AT)-rich DNA-binding protein 1 (SATB1) form a positive regulatory loop that enhances NeuroD1 protein transcription and subsequent neuronal differentiation.
Fluorosed mouse ameloblasts have increased SATB1 retention and Galphaq (show GNAQ ELISA Kits) activity.
The results indicated that the expression of SATB1 in both mRNA and protein levels was significantly decreased after cells transferred with siRNA sequence for 48 h, the proliferation of MF9 cells was significantly inhibited.
SATB1 binds to the ASE (show ARSE ELISA Kits) and Rag promoters, facilitating inclusion of Rag2 (show RAG2 ELISA Kits) in the chromatin hub and the loading of RNA polymerase II to both the Rag1 (show RAG1 ELISA Kits) and Rag2 (show RAG2 ELISA Kits) promoters.
SATB1 is indispensable for lymphocyte differentiation and stem cell development. (Review)
The ubiquitin-like domain-CUT repeat-like tandem of SATB1 are required for DNA binding.
Satb1 is a regulator that promotes hematopoietic stem cells quiescence and represses lineage commitment.
SATB1 as a critical regulator of interneuron maturation and terminal differentiation in the mammalian cortex.
These data suggest that Satb1 is required for medial ganglionic eminence-derived interneuron differentiation, connectivity, and survival.
This gene encodes a matrix protein which binds nuclear matrix and scaffold-associating DNAs through a unique nuclear architecture. The protein recruits chromatin-remodeling factors in order to regulate chromatin structure and gene expression. Multiple transcript variants encoding different isoforms have been found for this gene.
SATB homeobox 1
, special AT-rich sequence binding protein 1
, AT-rich binding protein-1
, DNA-binding protein SATB1-like
, DNA-binding protein SATB1
, special AT-rich sequence binding protein 1 (binds to nuclear matrix/scaffold-associating DNA)
, special AT-rich sequence-binding protein 1