You are viewing an incomplete version of our website. Please click to reload the website as full version.

Browse our CDKN1A (CDKN1A) ELISA Kits

Full name:
Cyclin-Dependent Kinase Inhibitor 1A ELISA Kits (CDKN1A)
On are 8 Cyclin-Dependent Kinase Inhibitor 1A (CDKN1A) ELISA Kits from 3 different suppliers available. Additionally we are shipping CDKN1A Antibodies (4) and CDKN1A Proteins (3) and many more products for this protein. A total of 15 CDKN1A products are currently listed.
CAP20, CDKI, Cdkn1, CIP1, MDA-6, mda6, P21, p21Cip1, p21WAF, SDI1, Waf1
list all ELISA KIts Gene Name GeneID UniProt
CDKN1A 1026 P38936
CDKN1A 12575 P39689

Show all species

Show all synonyms

More ELISA Kits for CDKN1A Interaction Partners

Human Cyclin-Dependent Kinase Inhibitor 1A (CDKN1A) interaction partners

  1. LRRC8A (show LRRC8A ELISA Kits) is an essential regulator of cisplatin induced p53 (show TP53 ELISA Kits) protein activity and its downstream signaling involving increased expression of p21Waf1/Cip1 and MDM2 (show MDM2 ELISA Kits), as well as activation of caspase-9 (show CASP9 ELISA Kits) and -3 in tumor cell lines.

  2. USP28 (show USP28 ELISA Kits)-53BP1 (show TP53BP1 ELISA Kits)-p53 (show TP53 ELISA Kits)-p21 signaling pathway is also required to arrest cell growth after a prolonged prometaphase.

  3. A new study describes a long noncoding RNA, DINO, transcribed from the CDKN1A promoter region that induces stabilization of p53 (show TP53 ELISA Kits) protein and promotes efficient activation of p53 (show TP53 ELISA Kits) target genes in response to DNA damage.

  4. Cables1 (show CABLES1 ELISA Kits)/p21 pathway has a strong effect on the induction of cell senescence and inhibition of cell growth, and acts as a novel regulatory mechanism in which p21 is probably one of several downstream effector molecules to mediate Cables1 (show CABLES1 ELISA Kits).

  5. These results show that ASPL (show ASPSCR1 ELISA Kits)-TFE3 (show TFE3 ELISA Kits) regulates cell cycle progression and induces cellular senescence by up-regulating p21 expression.

  6. our study provides evidence that up-regulation of p21 is mainly responsible for the suppressive effect of miR (show MLXIP ELISA Kits)-1180-5p on human bladder cancer cells and miR (show MLXIP ELISA Kits)-1180-5p can significantly inhibit tumorigenicity in vivo.

  7. Low CIP1 expression is associated with renal cell carcinoma.

  8. RNA-binding motif 5 silencing reduced the messenger RNA and protein expression of the p53 (show TP53 ELISA Kits) target gene p21. Our results suggest that RNA-binding motif 5 downregulation is involved in gastric cancer progression and that RNA-binding motif 5 behaves as a tumor suppressor gene in gastric cancer

  9. These findings suggest that normal p53 (show TP53 ELISA Kits) function is crucial in ATM (show ATM ELISA Kits)/p53 (show TP53 ELISA Kits)/p21 pathway activated by LDIR. The p53 (show TP53 ELISA Kits) status is the most probable reason leading to differential LDIR biological activities between breast tumor cells and normal breast cells

  10. PPARg (show PPARG ELISA Kits) signaling pathway may involve in the proliferation inhibition of evodiamine on K562 cells via inhibiting cylcin D1 and stimulating of p21.

Mouse (Murine) Cyclin-Dependent Kinase Inhibitor 1A (CDKN1A) interaction partners

  1. telomerase activity and cardiomyocyte telomere length decrease sharply in wild-type mouse hearts after birth, resulting in cardiomyocytes with dysfunctional telomeres and anaphase bridges and positive for the cell-cycle arrest protein p21.

  2. Consistent with the latter, Arid1a (show ARID1A ELISA Kits) reexpression in tumor cells led to increased p21 (Cdkn1a) expression and dramatic accumulation of cells in G2 phase of the cell cycle. These results also indicate a potential opportunity for therapeutic intervention in ARID1A (show ARID1A ELISA Kits)-deficient human breast cancer subtypes that retain one intact copy of the gene and also maintain wild-type TRP53 (show TP53 ELISA Kits) activity

  3. We discovered that FXR1P (show FXR1 ELISA Kits)-deficient aNSCs have altered expression of a select number of cell-cycle genes, and we identified the mRNA of cyclin-dependent kinase inhibitor 1A (Cdkn1a, p21) as a direct target of FXR1P (show FXR1 ELISA Kits).

  4. These results demonstrate that CQ and AQ increase the expression level of p21 and inhibit T cell proliferation and the development of IFN-gamma (show IFNG ELISA Kits)-producing Th1 (show HAND1 ELISA Kits) cells, thereby revealing beneficial roles in treating a wide range of chronic inflammatory diseases mediated by inflammatory T cells.

  5. increases the number of ALP-positive colonies after iPSC induction and decreases expression levels of Eomes (show EOMES ELISA Kits) and p21 mRNAs. Based on these observations, we propose that the CCR4 (show CCR4 ELISA Kits)-NOT deadenylase activity contributes to iPSC induction.

  6. this study shows that p21 is upregulated in T effector cells but not natural regulatory T cells after treatment with azacitidine, which protects from graft-versus-host Disease

  7. lincRNA-p21 overexpression dramatically inhibited acetylation of H3 and H4 at the Thy-1 (show THY1 ELISA Kits) promoter and Thy-1 (show THY1 ELISA Kits) expression levels in HLF1 (show HLF ELISA Kits) cells. Finally, lincRNA-p21 interference rescued LPS (show TLR4 ELISA Kits)-induced increase of lung and BAL collagen contents. LincRNA-p21 could lead to pulmonary fibrosis in ARDS by inhibition of the expression of Thy-1 (show THY1 ELISA Kits).

  8. p21 upregulation in hair follicle stem cells is associated with telogen retention in aged mice

  9. MiR-17-92 functions as a hepatocyte proliferation stimulator and acts in an estrogen-dependent manner. The loss of this miRNA results in increases in p21 and Pten expression and therefore impairs liver regeneration in female mice.

  10. we observed that miR (show MLXIP ELISA Kits)-378 modulates the oscillation amplitudes of Cdkn1a in the control of cell cycle and Por (show POR ELISA Kits) in the regulation of oxidation reduction by forming partnership with different circadian transcription factors

CDKN1A Antigen Profile

Antigen Summary

This gene encodes a potent cyclin-dependent kinase inhibitor. The encoded protein binds to and inhibits the activity of cyclin-CDK2 or -CDK4 complexes, and thus functions as a regulator of cell cycle progression at G1. The expression of this gene is tightly controlled by the tumor suppressor protein p53, through which this protein mediates the p53-dependent cell cycle G1 phase arrest in response to a variety of stress stimuli. This protein can interact with proliferating cell nuclear antigen (PCNA), a DNA polymerase accessory factor, and plays a regulatory role in S phase DNA replication and DNA damage repair. This protein was reported to be specifically cleaved by CASP3-like caspases, which thus leads to a dramatic activation of CDK2, and may be instrumental in the execution of apoptosis following caspase activation. Multiple alternatively spliced variants have been found for this gene.

Alternative names and synonyms associated with CDKN1A

  • cyclin-dependent kinase inhibitor 1A (p21, Cip1) (CDKN1A) Elisa Kit
  • cyclin-dependent kinase inhibitor 1A (P21) (Cdkn1a) Elisa Kit
  • CAP20 Elisa Kit
  • CDKI Elisa Kit
  • Cdkn1 Elisa Kit
  • CIP1 Elisa Kit
  • MDA-6 Elisa Kit
  • mda6 Elisa Kit
  • P21 Elisa Kit
  • p21Cip1 Elisa Kit
  • p21WAF Elisa Kit
  • SDI1 Elisa Kit
  • Waf1 Elisa Kit

Protein level used designations for CDKN1A

CDK-interacting protein 1 , CDK-interaction protein 1 , DNA synthesis inhibitor , cyclin-dependent kinase inhibitor 1 , melanoma differentiation associated protein 6 , wild-type p53-activated fragment 1 , melanoma differentiation-associated protein , p21

1026 Homo sapiens
12575 Mus musculus
Selected quality suppliers for CDKN1A (CDKN1A) ELISA Kits
Did you look for something else?