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Human Monoclonal LCN2 Primary Antibody for ELISA, WB - ABIN1045904
Devarajan: Neutrophil gelatinase-associated lipocalin (NGAL): a new marker of kidney disease. in Scandinavian journal of clinical and laboratory investigation. Supplementum 2008
Show all 18 Pubmed References
Human Monoclonal LCN2 Primary Antibody for ELISA, WB - ABIN1045905
Makris, Markou, Evodia, Dimopoulou, Drakopoulos, Ntetsika, Rizos, Baltopoulos, Haliassos: Urinary neutrophil gelatinase-associated lipocalin (NGAL) as an early marker of acute kidney injury in critically ill multiple trauma patients. in Clinical chemistry and laboratory medicine : CCLM / FESCC 2009
Show all 18 Pubmed References
Rat (Rattus) Polyclonal LCN2 Primary Antibody for IHC (p), WB - ABIN966671
Begley, Robb, Rockman, Visvader, Bockamp, Chan, Green: Structure of the gene encoding the murine SCL protein. in Gene 1994
Show all 6 Pubmed References
Human Polyclonal LCN2 Primary Antibody for IHC, IHC (p) - ABIN4331126
Rathore, Berard, Redensek, Chierzi, Lopez-Vales, Santos, Akira, David: Lipocalin 2 plays an immunomodulatory role and has detrimental effects after spinal cord injury. in The Journal of neuroscience : the official journal of the Society for Neuroscience 2011
Show all 3 Pubmed References
Human Polyclonal LCN2 Primary Antibody for IF (p), IHC (p) - ABIN685627
Xing, Wang, Cheng, Montaner, Mandeville, Leung, van Leyen, Lok, Wang, Lo: Neuronal production of lipocalin-2 as a help-me signal for glial activation. in Stroke; a journal of cerebral circulation 2014
Show all 2 Pubmed References
Human Polyclonal LCN2 Primary Antibody for IHC (p), IHC - ABIN250360
Yu, Wu, Ping, Huang, Shen: NGAL can alternately mediate sunitinib resistance in renal cell carcinoma. in The Journal of urology 2014
Show all 2 Pubmed References
Mouse (Murine) Polyclonal LCN2 Primary Antibody for ELISA, WB - ABIN408671
Venkatesha, Hanai, Seth, Karumanchi, Sukhatme: Lipocalin 2 antagonizes the proangiogenic action of ras in transformed cells. in Molecular cancer research : MCR 2006
Rat (Rattus) Monoclonal LCN2 Primary Antibody for ELISA, ICC - ABIN451573
Sultan, Ahmad, Rave-Fränk, Malik, Hess, Christiansen, Cameron: Induction of Lipocalin2 in a Rat Model of Lung Irradiation. in International journal of molecular sciences 2016
Our findings collectively support a potential role of LCN2 in cancer cell invasion through the epithelial-to- mesenchymal transition pathway and suggest that LCN2 could be effectively utilized as a lymph node metastasis marker in cervical cancer.
We conclude that low BDNF (show BDNF Antibodies) and high LCN2 and NF-L (show NEFL Antibodies) levels are associated with Multiple Sclerosis (MS) pathogenesis, and high IGFBP1level is a biomarker for female MS only, suggesting different MS progression pathways between the sexes. LCN2 is a candidate predictor of response to natalizumab treatment, and NF-L (show NEFL Antibodies) is a candidate predictor of clinically isolated syndrome's (CIS (show CISH Antibodies)) conversion into MS.
Ascites NGAL level may be a biomarker of peritonitis in hospitalized patients with cirrhosis and an independent predictor of short-term in-hospital mortality.
Circulating NGAL and MMP-9 (show MMP9 Antibodies)/NGAL are associated with plaque vulnerability in patients with carotid artery stenosis. Statin treatment could contribute to plaque stabilization by reducing circulating NGAL and MMP-9 (show MMP9 Antibodies)/NGAL levels.
our findings indicated that LCN2 negatively modulated proliferation, EMT (show ITK Antibodies) and energy metabolism in colorectal cancer (CRC (show CALR Antibodies)) cells. Accordingly, LCN2 may be a candidate metastasis suppressor and potential therapeutic target in CRC (show CALR Antibodies)
High plasma NGAL correlates with high mortality and myocardial dysfunction in severe sepsis and septic shock.
The analysis showed higher urinary NGAL and urinary ATF3 (show ATF3 Antibodies) in patients with sepsis-AKI in comparison with patients with sepsis-non-AKI and healthy volunteers.
Knockout of Lcn2 attenuated tumor-associated lymphangiogenesis and breast tumor metastasis
Lipocalin-2 Promotes Endoplasmic Reticulum Stress and Proliferation by Augmenting Intracellular Iron in Human Pulmonary Arterial Smooth Muscle Cells
Acute kidney injury is more common with sepsis than non-infectious systemic inflammatory response syndrome. Plasma NGAL was diagnostic of AKI in both subgroups.
Overexpression of exogenous kidney-specific Ngal reduced cystic progression and prolonged the lifespan in polycystic kidney disease mice, and was associated with reductions in interstitial fibrosis and proliferation, and augmented apoptosis.
Either Lcn2 deficiency or anti-Lcn2 antibody blockade limits abdominal aortic aneurysm expansion in mice by decreasing neutrophil infiltration in the aorta.
Results suggest that blood brain barrier leakage occurs in white matter after subarachnoid hemorrhage (SAH (show ACSM3 Antibodies)) and that LCN2 contributes to SAH (show ACSM3 Antibodies)-induced blood brain barrier disruption.
LCN2 overexpression in bones modifies the bone marrow microenvironment via modulation of the expression of key secreted factors and cytokines, which in turn regulate the hematopoietic stem cell niche behavior enhancing both HSC (show FUT1 Antibodies) homing in young mice and erythrocytes production in older mice.
LCN2 modulated the secretion of proinflammatory cytokines in PSC of the PDAC tumor microenvironment, whereas downregulation of LCN2-specific receptor SLC22A17 (show SLC22A17 Antibodies) blocked these effects. Our results reveal how LCN2 acts in the tumor microenvironment links obesity, inflammation, and PDAC development.
results identify lipocalin 2 as a bone-derived hormone with metabolic regulatory effects, which suppresses appetite in a MC4R (show MC4R Antibodies)-dependent manner, and show that the control of appetite is an endocrine function of bone
Lcn2 can suppress the beneficial cardiac autophagic response to ischemia and that this contributes to enhanced ischemia-induced cell death and cardiac dysfunction.
Plasma levels of LCN2 measured in patients one week after ischemic stroke contribute to the prediction of clinical outcome at 90 days and reflect the systemic response to post-stroke infections
may potentiate the development of psoriasis via the enhancement of Th17- and antimicrobial peptide (show cAMP Antibodies)-mediated inflammation
we demonstrated the presence of high molecular weight (HMW) complexes (130, 170, and 220 kDa) containing MMP9 (show MMP9 Antibodies), TIMP1 (show TIMP1 Antibodies), and NGAL (also MMP2 (show MMP2 Antibodies) in 220 kDa complex) without proteolytic activity.
porcine p25alpha (show Tppp Antibodies) is heavily modified with a variety of modifications: phosphorylation, di- and trimethylation, citrullination and a HexNAc group.
mouse homolog plays a role in IL3 withdrawal-induced apoptosis
, lipocalin 2 (oncogene 24p3)
, 25 kDa alpha-2-microglobulin-related subunit of MMP-9
, migration-stimulating factor inhibitor
, neutrophil gelatinase-associated lipocalin
, oncogene 24p3
, siderocalin LCN2
, SV-40-induced 24P3 protein
, secreted inducible protein 24
, alpha-2-microglobulin-related protein
, alpha-2U globulin-related protein