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Human Monoclonal C3B Primary Antibody for IHC (fro), WB - ABIN2477797
Hallström, Nordström, Tan, Manolov, Lambris, Isenman, Zipfel, Blom, Riesbeck: Immune evasion of Moraxella catarrhalis involves ubiquitous surface protein A-dependent C3d binding. in Journal of immunology (Baltimore, Md. : 1950) 2011
Our study shows C3 to be a relatively strong susceptibility gene for advanced-type-AMD (show AMD1 Antibodies) (exudative-and-geographic-atrophy) in an Iranian population.
BBB (show ALMS1 Antibodies) disruption is present in ACS (show PLA2G15 Antibodies), and elevated levels of IL-6 (show IL6 Antibodies) and C3 in CSF (show CSF2 Antibodies) in diffuse NPSLE
This study uncovers the origin of the effect of ionic strength on C3d-CR2 interaction and deepens the understanding of the molecular mechanism of their interaction, which is valuable for the design of vaccines and small molecule inhibitors.
Studies indicate that the complement response lie the active fragments, C3a (show C3 Antibodies) and C5a, acting through their specific receptors, C3aR (show C3AR1 Antibodies), C5aR1 (show C5AR1 Antibodies) and C5aR2 to direct the cellular response to inflammation.
exposure of neural stem cells to neutrophil-synthesized concentrations of C1q and C3a (show C3 Antibodies) promoted astrogliogenesis and cell migrationtion.
THP (show UMOD Antibodies) appears to participate directly in complement inactivation by its ability to act as a cofactor for C3b degradation.
Decreased C5a expression is associated with increased inflammation in cystic fibrosis (show S100A8 Antibodies).
C3d (show C3 Antibodies)-positive glomerular staining is an independent risk factor for the development of end-stage renal disease in ANCA-associated renal vasculitis.
Factor I binds C3b-Factor H (show CFH Antibodies) between Factor H (show CFH Antibodies) domains 2 and 3 and a reoriented C3b C-terminal domain and docks onto the first scissile bond, while stabilizing its catalytic domain for proteolytic activity.
The VEGF (show VEGFA Antibodies) haplotype TGA (show TBX1 Antibodies) could be used as a marker for poor visual prognosis in Tunisian patients with neovascular AMD (show AMD1 Antibodies) treated with bevacizumab.
Complement component C3 plays a central role in the activation of complement system. Its activation is required for both classical and alternative complement activation pathways. People with C3 deficiency are susceptible to bacterial infection.
C3 and PZP-like alpha-2-macroglobulin domain-containing protein 1
, C3a anaphylatoxin
, acylation-stimulating protein cleavage product
, complement C3
, complement component C3
, complement component C3a
, complement component C3b