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anti-Human C7 Antibodies:
anti-Mouse (Murine) C7 Antibodies:
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Human Polyclonal C7 Primary Antibody for FACS, IHC (p) - ABIN653210
Davila, Froeling, Tan, Bonnard, Boland, Snippe, Hibberd, Seielstad: New genetic associations detected in a host response study to hepatitis B vaccine. in Genes and immunity 2010
Show all 2 references for ABIN653210
Human Polyclonal C7 Primary Antibody for FACS, IHC (p) - ABIN951642
Kuijpers, Nguyen, Hopman, Nieuwenhuys, Dewald, Lankester, Roos, van der Ende, Fijen, de Boer: Complement factor 7 gene mutations in relation to meningococcal infection and clinical recurrence of meningococcal disease. in Molecular immunology 2010
Show all 2 references for ABIN951642
Human Polyclonal C7 Primary Antibody for IHC, IHC (p) - ABIN4299873
Kato, Nicholson, Neiman, Rantalainen, Holmes, Barrett, Uhlén, Nilsson, Spector, Schwenk: Variance decomposition of protein profiles from antibody arrays using a longitudinal twin model. in Proteome science 2011
The complement C7 rs6876739 CC genotypes and mannan-binding lectin (MBL2 (show MBL2 Antibodies)) gene polymorphisms of liver donors were significantly associated with bacterial infection in recipients.
Complement proteins C7 and CFH (show CFH Antibodies) control the stemness of liver cancer cells via LSF (show TFCP2 Antibodies)-1 pathway.
Borrelial CspA binds the human terminal complement components C7 and C9 and blocks assembly and membrane insertion of the terminal complement complex (TCC (show SFXN1 Antibodies)).
Complement C5b-9 induce a JNK (show MAPK8 Antibodies)/Bid (show BID Antibodies)-dependent and JNK (show MAPK8 Antibodies)-independent necrotic cell death.
To determine transcriptional regulation of the human complement component C7, a 1 kb promoter fragment was cloned and the transcription start site was determined. C7 is expressed by the hepatoma-derived cell line Hep-3B, but not by Hep-G2.
The interaction between the factor I domain of C7 and the C345C domain at the C terminus of the C5 alpha-chain (show FCGRT Antibodies) plays an essential role in complement membrane attack complex formation and complement lytic activity.
recurrence of fulminant meningococcal disease in a complement component C7-deficient patient
Cell cycle induction by C5b-9 in aortic endothelial cells is RGC-32 (show C13orf15 Antibodies) dependent and this is in part through regulation of Akt (show AKT1 Antibodies) and growth factor release.
C7 is a component of the complement system. It participates in the formation of Membrane Attack Complex (MAC). People with C7 deficiency are prone to bacterial infection.
complement component C7
, complement C7