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CDH13 genetic variants determine Chinese individuals' susceptibility to chronic obstructive pulmonary disease (COPD (show ARCN1 ELISA Kits)) and thus are efficient genetic biomarkers for early detection of COPD (show ARCN1 ELISA Kits).
Strong Functional Association of adipor2 (show ADIPOR2 ELISA Kits) and cdh13 with adipoq (show ADIPOQ ELISA Kits)
CDH13 Polymorphisms are Associated with Adiponectin (show ADIPOQ ELISA Kits) Levels and Metabolic Syndrome Traits Independently of Visceral Fat Mass.
CDH13 locus variants and adiponectin (show ADIPOQ ELISA Kits) levels are associated with circulating levels of cellular adhesion molecules and adiposity status in a differential manner that interacts with sex
Data demonstrate for the first time that SHP1 (show PTPN6 ELISA Kits) methylation has high specificity for diagnosis of endometrial carcinoma, while CDH13 promoter methylation plays a role in the earlier stage.
The present study identified a new genetic factor for CRC (show CALR ELISA Kits) risk and an interaction between CDH13 and APN (show ANPEP ELISA Kits) in CRC (show CALR ELISA Kits) risk. These genetic factors may be useful for predicting CRC (show CALR ELISA Kits) risk.
The downregulation of T-cadherin may contribute to gastric cancer progression, representing a useful biomarker for predicting the biological behavior and prognosis of gastric cancer.
Finnish prisoners, revealed that a monoamine oxidase A (MAOA (show MAOA ELISA Kits)) low-activity genotype (contributing to low dopamine turnover rate) as well as the CDH13 gene (coding for neuronal membrane adhesion protein) are associated with extremely violent behavior
The genetic polymorphisms at the CDH13 locus independently affect the adiponectin (show ADIPOQ ELISA Kits) levels, whereas the adiponectin (show ADIPOQ ELISA Kits) levels exhibit a suppressive effect on the association between CDH13 locus variants and various metabolic phenotypes and metabolic syndrome
The CDH13 rs11150556 CC genotype was associated with more hyperactive/impulsive symptoms in attention/deficit hyperactivity disorder.
CDH13 is a negative regulator of inhibitory synapses in the hippocampus, and provide insights into how CDH13 dysfunction may contribute to the excitatory/inhibitory imbalance observed in Attention Deficit Disorder with Hyperactivity.
These data show that both circulating and tissue-bound Adipo levels are dependent on Tcad and, in reverse, regulate tissue Tcad levels through a positive feedback loop.
These data highlight a previously unrecognized role for T-cadherin in limb revascularization and show that it is essential for mediating the vascular actions of adiponectin (show ADIPOQ ELISA Kits).
T-cad does not mediate the protective effects of adiponectin (show ADIPOQ ELISA Kits) in allergic airways responses in mice
T-cadherin is a component of insulin (show INS ELISA Kits) granules, suggesting that it contributes to the regulation of insulin (show INS ELISA Kits) secretion independently of direct interactions with adiponectin (show ADIPOQ ELISA Kits).
T-cad can exert pleiotropic effects on squamous cell carcinoma progression; up- or down-regulation of T-cad can promote tumour expansion in vivo.
T-cadherin protects from stress-induced pathological cardiac remodeling by binding APN (show ANPEP ELISA Kits) and activating its cardioprotective functions.
hyperexpression of T-cadherin in the B16F10 cells suppresses the proliferation of these cells in vitro and the growth of the tumor masses formed by melanoma cells on the chorioallantoic membrane and their neovascularization
Expression of T-cadherin in Basal keratinocytes of skin.
T-cadherin has a role in growth regulation involving p21(CIP1/WAF1 (show CDKN1A ELISA Kits)) expression and G2 arrest
This gene encodes a member of the cadherin superfamily. The encoded protein is localized to the surface of the cell membrane and is anchored by a GPI moiety, rather than by a transmembrane domain. The protein lacks the cytoplasmic domain characteristic of other cadherins, and so is not thought to be a cell-cell adhesion glycoprotein. This protein acts as a negative regulator of axon growth during neural differentiation. It also protects vascular endothelial cells from apoptosis due to oxidative stress, and is associated with resistance to atherosclerosis. The gene is hypermethylated in many types of cancer. Alternative splicing results in multiple transcript variants encoding different isoforms.
, H-cadherin (heart)
, cadherin 13, H-cadherin (heart)
, heart cadherin