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Human BNP Protein expressed in Escherichia coli (E. coli) - ABIN987787
Lu, Fu, Chen, Lu, Yao, Shen, Gu: Multiplex detection of B-type natriuretic peptide, cardiac troponin I and C-reactive protein with photonic suspension array. in PLoS ONE 2012
Show all 2 Pubmed References
Mouse (Murine) BNP Protein expressed in Escherichia coli (E. coli) - ABIN1079003
Furrer, Eisele, Schmidt, Beer, Handschin: Paracrine cross-talk between skeletal muscle and macrophages in exercise by PGC-1α-controlled BNP. in Scientific reports 2017
CNP and BNP serve as oocyte maturation inhibitor to inhibit oocyte maturation in mammals
ANP and BNP gene expression differs considerably between cardiac chambers in the first 72 hours of life in healthy piglets, resembling the transition from fetal to neonate circulation. The cardiac gene expression does not explain plasma concentrations.
Up-regulation of BNP and a reduction in a ratio of sarcoplasmic reticulum calcium ATPase 2a in failing myocardium are observed in a porcine model of tachycardia.
Data show that all five molecules, BNP, ICAM-1 (show ICAM1 Proteins), TNF-alpha (show TNF Proteins), VCAM-1 (show VCAM1 Proteins) and IL-6 (show IL6 Proteins), quickly and reliably signaled adverse interactions.
identification and characterization of a novel alternatively spliced variant of porcine and human NPPB resulting from exon 2 skipping
Arial natriuretic factor (ANF (show HESX1 Proteins)) and N-terminal proANF may be better suited than brain natriuretic peptide as markers of cardiac preload during the development and treatment of acute heart failure.
Regional differences in BNP and CNP (show NPPC Proteins) expression were observed in myocardium with sustained left ventricular dyssynchronous contraction.
Data suggest that the negative inhibition of P2X3R activity by the BNP/NPR-A (show NPR1 Proteins) pathway results in a decreased P2X3R-mediated excitability of trigeminal neurons in wildetype cultures. In familial hemiplegic migraine type-1 model cultures, however, lack of efficient P2X3Rs downregulation contributes to the neuronal hyperexcitability phenotype.
we have validated a novel gene, Myh14, as a negative regulator of ISO-induced left ventricular mass hypertrophy in an in vivo mouse model and demonstrated the up-regulation of immediate early gene Myc, fetal gene Nppb, and fibrosis gene Lgals3 in ISO-treated Myh14 deficient hearts compared to controls.
P2X3 (show P2RX3 Proteins) receptors on mouse trigeminal ganglion neurons are subjected to contrasting modulation by inhibitory brain natriuretic peptide and facilitatory calcitonin gene-related peptide (show CALCA Proteins) that both operate via complex intracellular signaling.
mouse trigeminal neurons endogenous BNP acts on NPR-A (show NPR1 Proteins) receptors to determine constitutive depression of P2X3 (show P2RX3 Proteins) receptor function. Tonic inhibition of P2X3 (show P2RX3 Proteins) receptor activity by BNP/NPR-A (show NPR1 Proteins)/PKG (show PRKG1 Proteins) pathways occurs via two distinct mechanisms: P2X3 (show P2RX3 Proteins) serine phosphorylation and receptor redistribution to non-raft membrane compartments
The findings demonstrated a substantial role of mitochondrial dysfunction in mediating the downregulation of NKCC2 and ENaCalpha in obstructive kidney disease, possibly via iNOS-derived nitric oxide and BNP.
Data (including data from studies in knockout mice) suggest expression of ANP (show NPPA Proteins)/BNP/GC-A (show GUCA1A Proteins) (atrial natriuretic peptide (show NPPA Proteins), brain natriuretic peptide, and guanylyl cyclase-A (show NPR1 Proteins)) is important in embryonic neovascularization and organogenesis.
Goalpha plays a role in ANF sorting during intracellular vectorial transport and with the presence of a mechanism that preserves the molar relationship between cellular ANF and BNP.
The RNA-Seq demonstrates that NPPB which well activates natriuretic peptide receptor 1 (NPR1 (show NPR1 Proteins)), is well expressed in mouse dorsal root ganglian.
a slow modulatory action by BNP on TRPV1 (show TRPV1 Proteins) and P2X3 (show P2RX3 Proteins) receptors outlining the role of this peptide as a negative regulator of trigeminal sensory neuron excitability to nociceptive stimuli
Natriuretic peptides buffer renin (show REN Proteins)-dependent hypertension.
hormone produced primarily by the atrium and ventricle of the heart
brain natriuretic peptide
, gamma-brain natriuretic peptide
, natriuretic peptides B
, Brain natriuretic factor
, natriuretic peptide precursor B
, natriuretic peptide precursor type B
, Gamma-brain natriuretic peptide