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Human Hepcidin ELISA Kit for Sandwich ELISA - ABIN415120
Cheng, Sun, Jiang, Tang, Jiao: Hepcidin expression in patients with acute leukaemia. in European journal of clinical investigation 2012
Show all 16 Pubmed References
Human Hepcidin ELISA Kit for Competition ELISA - ABIN2761946
Sany, Elsawy, Elshahawy: Hepcidin and regulation of iron homeostasis in maintenance hemodialysis patients. in Saudi journal of kidney diseases and transplantation : an official publication of the Saudi Center for Organ Transplantation, Saudi Arabia 2014
Show all 9 Pubmed References
Human Hepcidin ELISA Kit for Sandwich ELISA - ABIN457010
Jiang, Sun, Tang, Xu, Jiao: Hepcidin expression and iron parameters change in Type 2 diabetic patients. in Diabetes research and clinical practice 2011
Show all 8 Pubmed References
Mouse (Murine) Hepcidin ELISA Kit for Sandwich ELISA - ABIN415686
Shin, Chung, Joe, Pae, Chang, Cho, Ryter, Chung: Pretreatment with CO-releasing molecules suppresses hepcidin expression during inflammation and endoplasmic reticulum stress through inhibition of the STAT3 and CREBH pathways. in Blood 2012
Show all 6 Pubmed References
Rat (Rattus) Hepcidin ELISA Kit for Sandwich ELISA - ABIN416434
Huang, Chuang, Yang, Huang, Wu, Chen: Cholestasis downregulate hepcidin expression through inhibiting IL-6-induced phosphorylation of signal transducer and activator of transcription 3 signaling. in Laboratory investigation; a journal of technical methods and pathology 2009
Show all 5 Pubmed References
Mouse (Murine) Hepcidin ELISA Kit for Competition ELISA - ABIN2761947
Vecchi, Montosi, Garuti, Corradini, Sabelli, Canali, Pietrangelo: Gluconeogenic signals regulate iron homeostasis via hepcidin in mice. in Gastroenterology 2014
Show all 2 Pubmed References
Pig (Porcine) Hepcidin ELISA Kit for Competition ELISA - ABIN2761945
Pu, Guo, Liu, Xiong, Wang, Du: Iron Supplementation Attenuates the Inflammatory Status of Anemic Piglets by Regulating Hepcidin. in Biological trace element research 2015
Show all 2 Pubmed References
Rat (Rattus) Hepcidin ELISA Kit for Sandwich ELISA - ABIN578992
Wang, Du, Duan, Guo, Sun, Liu: Inhibiting heme oxygenase-1 attenuates rat liver fibrosis by removing iron accumulation. in World journal of gastroenterology : WJG 2013
This study shows that hepcidin knockdown in zebrafish using morpholinos leads to iron overload.
The data also show that the antibacterial activity of hepcidin-2 depends upon the disulfide bridges.
data support an alternative mechanism for hepcidin regulation during zebrafish embryonic development, which is independent of hemojuvelin (show HFE2 ELISA Kits).
Hepcidin expression is regulated by a transferrin-a (show Tf ELISA Kits)-dependent pathway in the zebrafish embryo.
study followed the dynamics of hepcidin-mediated ferroportin (show SLC40A1 ELISA Kits) internalization; also showed that the novel p.D84E mutation, associated with the classical form of ferroportin (show SLC40A1 ELISA Kits) disease, is both iron transport defective and hepcidin insensitive
During regulation of hepcidin synthesis, multiple promoter elements in the HAMP gene respond to variable signaling pathways corresponding to different extracellular situations.
Our findings indicate that the HAMP-P -582A>G polymorphism (rs10421768) is associated with susceptibility to extrapulmonary TB, but not pulmonary TB. CD14 (show NDUFA2 ELISA Kits)+ monocytes from individuals with the rs10421768 GG genotype secreted significantly less hepcidin in response to M. tuberculosis lipoarabinomannan compared with cells from individuals with either the AA or AG genotypes.
Expression of Hepcidin and Ferroportin (show SLC40A1 ELISA Kits) in the Placenta, and Ferritin (show FTL ELISA Kits) and Transferrin Receptor 1 (show TFR ELISA Kits) Levels in Maternal and Umbilical Cord Blood in Pregnant Women with and without Gestational Diabetes
this study shows that hepcidin is involved in the pathogenesis of sepsis-induced acute kidney injury
The hepcidin plays a role in the pathogenesis and progression of kidney injury via modulation of iron-mediated oxidant injury.
The levels of hepcidin were higher, while those of iron, transferrin (show Tf ELISA Kits), and sTfR (show TFRC ELISA Kits) were lower in children with cardiometabolic risk factors.
Main pathways of systemic and genetic regulation of hepcidin, as well as its influence on the disorders related to iron metabolism. Review.
Data suggest hepcidin is the master regulator of systemic iron homeostasis; hepcidin levels are suppressed when erythropoiesis is stimulated; the erythroid-derived hormone erythroferrone appears to be a convincing candidate for link between increased erythropoiesis and hepcidin suppression. [REVIEW]
Data suggest that proinflammatory cytokine interleukin-1beta (IL1B (show IL1B ELISA Kits)) up-regulates hepcidin expression in hepatocytes; inflammation induces IL1B (show IL1B ELISA Kits) production in Kupffer cells and hepatocytes; IL1B (show IL1B ELISA Kits) up-regulates CEBPD (CCAAT/enhancer binding protein delta (show CEBPD ELISA Kits)) expression in hepatocytes; up-regulation of CEBPD (show CEBPD ELISA Kits) expression up-regulates hepcidin transcription.
The lack of effect of erythropoietin (show EPO ELISA Kits) on hepcidin expression in mask mice can not be explained by changes in erythroferrone synthesis, as splenic erythroferrone content increased after erythropoietin (show EPO ELISA Kits) administration in both C57BL/6 and mask mice.
these results characterise a new model of rapidly inducible hepcidin disruption, and demonstrate the critical contribution of hepcidin to the hypoferraemia of inflammation
Hepatic gene expression of hepcidin is regulated in beta-thalassemia by ATOH8.
Endogenous hepcidin and its agonist mediate resistance to selected infections by clearing non-transferrin (show Tf ELISA Kits)-bound iron.
The data demonstrate that endothelial cells are the predominant source of BMP6 (show BMP6 ELISA Kits) in the liver and support a model in which endothelial cells BMP6 (show BMP6 ELISA Kits) has paracrine actions on hepatocyte hemojuvelin (show HFE2 ELISA Kits) to regulate hepcidin transcription and maintain systemic iron homeostasis.
Smad1 (show SMAD1 ELISA Kits) and Smad5 (show SMAD5 ELISA Kits) have overlapping functions to govern hepcidin transcription. Moreover, erythropoietin (show EPO ELISA Kits) and erythroferrone target Smad1 (show SMAD1 ELISA Kits)/5 signaling and require Smad1 (show SMAD1 ELISA Kits)/5 to suppress hepcidin expression.
Genetic inactivation of hepatic angiocrine Bmp2 (show BMP2 ELISA Kits) signaling in Stab2 (show STAB2 ELISA Kits)-Cre mice caused massive iron overload in the liver and increased serum iron levels and iron deposition in several organs similar to classic hereditary hemochromatosis (show HFE ELISA Kits); these changes were mediated by decreased hepatic expression of hepcidin, a key regulator of iron homeostasis.
These studies indicate that drug-like minihepcidins have a potential as future therapeutics for untransfused beta-thalassemia and polycythemia vera (show IGF2BP3 ELISA Kits).
the function of matriptase-2 (show TMPRSS6 ELISA Kits) is dominant over that of ERFE and is essential in facilitating hepcidin suppression by attenuating the BMP-SMAD (show SMAD1 ELISA Kits) signaling.
hepcidin induction by endoplasmic reticulum stress involves the central SMAD1 (show SMAD1 ELISA Kits)/5/8 pathway
this study demonstrates that urine hepcidin-25 concentrations strongly correlate with hepatic hepcidin mRNA abundance, plasma hepcidin-25 levels, iron transferrin (show Tf ELISA Kits) saturation and non-heme liver iron levels.
Data suugest that hepcidin might had antiinflammatory function and is a candidate regulator of the cross-talk between iron regulation and inflammation.
report the full-length cDNA sequences of porcine hepcidin and liver-expressed antimicrobial peptide-2 (LEAP-2 (show LEAP2 ELISA Kits))
Hepcidin peptide is up-regulated by iron and bacterial components in the trout liver.
The product encoded by this gene is involved in the maintenance of iron homeostasis, and it is necessary for the regulation of iron storage in macrophages, and for intestinal iron absorption. The preproprotein is post-translationally cleaved into mature peptides of 20, 22 and 25 amino acids, and these active peptides are rich in cysteines, which form intramolecular bonds that stabilize their beta-sheet structures. These peptides exhibit antimicrobial activity. Mutations in this gene cause hemochromatosis type 2B, also known as juvenile hemochromatosis, a disease caused by severe iron overload that results in cardiomyopathy, cirrhosis, and endocrine failure.
hepcidin antimicrobial peptide
, antimicrobial peptide
, iron regulatory peptide
, preprohepcidin 1
, liver-expressed antimicrobial peptide 1
, putative liver tumor regressor
, hepcidin antimicrobial peptide 1
, antimicrobial peptide hepcidin
, putative hepcidin antibacterial peptide