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Human IL7 ELISA Kit for Sandwich ELISA - ABIN921098
Kröncke, Loppnow, Flad, Gerdes: Human follicular dendritic cells and vascular cells produce interleukin-7: a potential role for interleukin-7 in the germinal center reaction. in European journal of immunology 1996
Show all 3 references for ABIN921098
Rat (Rattus) IL7 ELISA Kit for Sandwich ELISA - ABIN1115680
Dai, Wu, Li, Chen, Chen, Hu, Shen: Effects and mechanisms of Geniposide on rats with adjuvant arthritis. in International immunopharmacology 2014
Mouse (Murine) IL7 ELISA Kit for Sandwich ELISA - ABIN921099
Liu, Leung, Wang, Tan, Wang, Guo, Fang, Zhao, Wan, Qin, Lu, Li, Pan, Song, Liu, Hong, Lu, Zhang: Crucial role of interleukin-7 in T helper type 17 survival and expansion in autoimmune disease. in Nature medicine 2010
increased IL-7 protein production in situ (lung tissue) from non-human primates who received rBCG vaccination, which was associated with increased survival.
The observations suggest that IL-7 may play a role in the pathogenesis of Graves' disease and may be associated with its clinical activity.
IL-7 and SCF (show KITLG ELISA Kits) are elevated for a prolonged period after double umbilical cord blood transplantation and persistently high levels of these cytokines may correlate with worse clinical outcomes.
Common gamma-chain (show IL2RG ELISA Kits) cytokines IL-2 (show IL2 ELISA Kits), IL-7, and IL-15 (show IL15 ELISA Kits) prevent differentiation of naive T cells in vitro and limit activation of primed T cells in the absence of antigenic stimulus, which can contribute to the formation of cytokine imbalance.
IL7 was expressed primarily in the infundibulum and s (show CD8A ELISA Kits)uprabulb (show CD4 ELISA Kits)of the hair follicle. IL7 expression was increased in cutaneous T-cell lymphoma.
this study identified that IL-7, as well as the Akt (show AKT1 ELISA Kits)/ mTOR (show FRAP1 ELISA Kits) signaling pathway, effectively modulates human Double-Negative T Cell- mediated suppression of allogeneic T cell responses.
IL-7/IL-17 (show IL17A ELISA Kits) axis mediates chronic pelvic pain in experimental autoimmune prostatitis and in patients.
In view of the important role IL-7 plays in lymphocyte proliferation, homeostasis and survival, down regulation of CD127 (show IL7R ELISA Kits) by Tat (show TAT ELISA Kits) likely plays a central role in immune dysregulation and CD4 (show CD4 ELISA Kits) T-cell decline
decreased IL-7 in peripheral blood, maybe, is a consequence of the negative feedback of the pro-inflammatory function in ITP (show ITPA ELISA Kits) patients.
Septic patients showed the lowest levels of IL-7. Patients with severe sepsis reached levels of IL-7 higher than those observed in the groups of uncomplicated sepsis and septic shock.
Interferon-alpha (show IFNA ELISA Kits) inhibits CD4 (show CD4 ELISA Kits) T cell responses to interleukin-7 and interleukin-2 (show IL2 ELISA Kits) and selectively interferes with Akt (show AKT1 ELISA Kits) signaling.
continuous IL7 signaling was not required for tumor regression, although LIP of naive CD8 (show CD8A ELISA Kits)+ T cells is usually regulated by IL7
IL7 represses the follicular helper T cell gene program.
This study uncovers the metabolic mechanisms by which IL-7 tailors the metabolism of memory T cells to promote their longevity and fast response to rechallenge.
IL-7 reduced IRAK-M (show IRAK3 ELISA Kits) expression and attenuated immune tolerance induced by either LPS (show TLR4 ELISA Kits) or CpGA
Poly I:C induces IL-7 production, early inflammatory responses, and characteristic pathologies of SS-like dacryoadenitis in non-autoimmune-prone C57BL/6 mice.
Data show that interleukin 7 (IL-7) signaling is a prerequisite for optimal CD4 (show CD4 ELISA Kits)(+) T cell activation.
reduced surface expression of IL-7R and concomitant limited responsiveness to IL-7 signals as a common mechanism resulting in reduced cell survival of common lymphoid progenitors and thymocytes at the double-negative to double-positive transition
Hair follicle expression of IL-7 was required for CD8 (show CD8A ELISA Kits)+ and CD4 (show CD4 ELISA Kits)+ skin-resident memory T (TRM) cells to exert tropism for the epidermis. In a cutaneous T cell lymphoma model, CD4 (show CD4 ELISA Kits)+ TRM lymphoma cell localization depended on hair follicle-derived IL-7.
Late IL-2 (show IL2 ELISA Kits) promotes survival through acute downregulation of apoptotic pathways in effector T cells and by permanently upregulating their IL-7 receptor expression, enabling IL-7 to sustain them as memory T cells.
The protein encoded by this gene is a cytokine important for B and T cell development. This cytokine and the hepatocyte growth factor (HGF) form a heterodimer that functions as a pre-pro-B cell growth-stimulating factor. This cytokine is found to be a cofactor for V(D)J rearrangement of the T cell receptor beta (TCRB) during early T cell development. This cytokine can be produced locally by intestinal epithelial and epithelial goblet cells, and may serve as a regulatory factor for intestinal mucosal lymphocytes. Knockout studies in mice suggested that this cytokine plays an essential role in lymphoid cell survival. Alternative splicing results in multiple transcript variants encoding distinct isoforms. Additional splice variants have been described but their presence in normal tissues has not been confirmed.