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Human IL7 ELISA Kit for Sandwich ELISA - ABIN625028
Meltzer, Standley: Modeled repetitive motion strain and indirect osteopathic manipulative techniques in regulation of human fibroblast proliferation and interleukin secretion. in The Journal of the American Osteopathic Association 2008
Show all 13 references for ABIN625028
Rat (Rattus) IL7 ELISA Kit for Sandwich ELISA - ABIN1115680
Dai, Wu, Li, Chen, Chen, Hu, Shen: Effects and mechanisms of Geniposide on rats with adjuvant arthritis. in International immunopharmacology 2014
Mouse (Murine) IL7 ELISA Kit for Sandwich ELISA - ABIN625152
Jung, Han, Kim, Kim, Lee, Seo, Youn, Lee: Stimulatory effect of HGF-overexpressing adipose tissue-derived mesenchymal stem cells on thymus regeneration in a rat thymus involution model. in Cell biology international 2014
increased IL-7 protein production in situ (lung tissue) from non-human primates who received rBCG vaccination, which was associated with increased survival.
The results from this study suggested for the first time that miR (show MLXIP ELISA Kits)-181c was able to negatively regulate the production of proinflammatory cytokines IL-7 and IL-17 (show IL17A ELISA Kits) in myasthenia gravis patients.
Increase in serum IL-7 is associated with adenoma.
provides negative feedback on its own signaling in T cells via endocytosis and degradation of its receptor, CD127 (show IL7R ELISA Kits)
Observed highly significant reductions in the concentration of circulating interleukin (IL)-16 (show IL16 ELISA Kits), IL-7, and Vascular Endothelial Growth Factor A (VEGF-A (show VEGFA ELISA Kits)) in encephalomyelitis/chronic fatigue syndrome patients.
The observations suggest that IL-7 may play a role in the pathogenesis of Graves' disease and may be associated with its clinical activity.
IL-7 and SCF (show KITLG ELISA Kits) are elevated for a prolonged period after double umbilical cord blood transplantation and persistently high levels of these cytokines may correlate with worse clinical outcomes.
Common gamma-chain (show IL2RG ELISA Kits) cytokines IL-2 (show IL2 ELISA Kits), IL-7, and IL-15 (show IL15 ELISA Kits) prevent differentiation of naive T cells in vitro and limit activation of primed T cells in the absence of antigenic stimulus, which can contribute to the formation of cytokine imbalance.
IL7 was expressed primarily in the infundibulum and s (show CD8A ELISA Kits)uprabulb (show CD4 ELISA Kits)of the hair follicle. IL7 expression was increased in cutaneous T-cell lymphoma.
this study identified that IL-7, as well as the Akt (show AKT1 ELISA Kits)/ mTOR (show FRAP1 ELISA Kits) signaling pathway, effectively modulates human Double-Negative T Cell- mediated suppression of allogeneic T cell responses.
IL-7/IL-17 (show IL17A ELISA Kits) axis mediates chronic pelvic pain in experimental autoimmune prostatitis and in patients.
findings support a redundant role for adaptive Th17 cell- and innate gammadeltaT17 cell-derived IL17 (show IL17A ELISA Kits) in bacteria-induced colon carcinogenesis, stressing the importance of therapeutically targeting the cytokine itself rather than its cellular sources
functional Gimap5 is required for optimal signaling through TCR and IL-7R in T cells.
continuous IL7 signaling was not required for tumor regression, although LIP of naive CD8 (show CD8A ELISA Kits)+ T cells is usually regulated by IL7
IL7 represses the follicular helper T cell gene program.
This study uncovers the metabolic mechanisms by which IL-7 tailors the metabolism of memory T cells to promote their longevity and fast response to rechallenge.
IL-7 reduced IRAK-M (show IRAK3 ELISA Kits) expression and attenuated immune tolerance induced by either LPS (show TLR4 ELISA Kits) or CpGA
Poly I:C induces IL-7 production, early inflammatory responses, and characteristic pathologies of SS-like dacryoadenitis in non-autoimmune-prone C57BL/6 mice.
Data show that interleukin 7 (IL-7) signaling is a prerequisite for optimal CD4 (show CD4 ELISA Kits)(+) T cell activation.
reduced surface expression of IL-7R and concomitant limited responsiveness to IL-7 signals as a common mechanism resulting in reduced cell survival of common lymphoid progenitors and thymocytes at the double-negative to double-positive transition
Hair follicle expression of IL-7 was required for CD8 (show CD8A ELISA Kits)+ and CD4 (show CD4 ELISA Kits)+ skin-resident memory T (TRM) cells to exert tropism for the epidermis. In a cutaneous T cell lymphoma model, CD4 (show CD4 ELISA Kits)+ TRM lymphoma cell localization depended on hair follicle-derived IL-7.
The protein encoded by this gene is a cytokine important for B and T cell development. This cytokine and the hepatocyte growth factor (HGF) form a heterodimer that functions as a pre-pro-B cell growth-stimulating factor. This cytokine is found to be a cofactor for V(D)J rearrangement of the T cell receptor beta (TCRB) during early T cell development. This cytokine can be produced locally by intestinal epithelial and epithelial goblet cells, and may serve as a regulatory factor for intestinal mucosal lymphocytes. Knockout studies in mice suggested that this cytokine plays an essential role in lymphoid cell survival. Alternative splicing results in multiple transcript variants encoding distinct isoforms. Additional splice variants have been described but their presence in normal tissues has not been confirmed.