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Human AMH Primary Antibody for ELISA - ABIN414951
Lin, Wang, Weng, Sheng, Jiang, Yang et al.: Influence of gonadotropin-releasing hormone agonist on the effect of chemotherapy upon ovarian cancer and the prevention of chemotherapy-induced ovarian damage: an experimental study with nu/nu ... in Journal of Zhejiang University. Science. B 2012
Show all 11 references for ABIN414951
Rat (Rattus) AMH Primary Antibody for ELISA - ABIN367547
Ozler, Turgut, Soydinç, Sak, Evsen, Alabalik, Basarali, Deveci: The biochemical and histologic effects of adnexal torsion and early surgical intervention to unwind detorsion on ovarian reserve: an experimental study. in Reproductive sciences (Thousand Oaks, Calif.) 2013
Show all 4 references for ABIN367547
Cow (Bovine) AMH Primary Antibody for ELISA - ABIN413999
Nfon, Marszal, Zhang, Weingartl: Innate immune response to Rift Valley fever virus in goats. in PLoS neglected tropical diseases 2012
Show all 2 references for ABIN413999
Mouse (Murine) AMH Primary Antibody for ELISA - ABIN415733
Wang, Qu, Dong, Huang, Kumar, Ji, Wang, Yao, Yang, Wu, Zhang: Long-term effects of methamphetamine exposure in adolescent mice on the future ovarian reserve in adulthood. in Toxicology letters 2016
nuclear receptor subfamily 5, group A, member 1b is a new candidate for sex determination and differentiation in a way similar to steroidogenic factor 1 (show NR5A1 ELISA Kits), possibly involving AMH
zebrafish Anti-Mullerian hormone (Amh) is regulated by sox9a, sox9b, and cyp19a1a during gonad development
amh is a candidate gene down-regulating cyp19a1a, leading to "juvenile ovary-to-testis" transformation.
It was shown that the male-to-female sex reversal phenotype in hotei medaka mutants is not a direct consequence of anti-Mullerian hormone signaling in supporting cells, but is instead mediated by germ cells.
Anti-Mullerian hormone (AMH) and inhibin-A (show INHA ELISA Kits) (INH-A) levels were found to be significantly higher in the polycystic ovary syndrome (PCOS) group compared to the controls.
a significant positive correlation between serum asymmetric dimethylarginine and AMH levels in primary dysmenorrhea, is reported.
Data suggest that the serum AMH level (a biomarker of ovarian reserve) in adolescent girls newly diagnosed with Hashimoto's thyroiditis (HT) is up-regulated compared to age- and BMI-matched control subjects; serum AMH levels are negatively correlated with serum biomarkers of oxidative stress in HT.
Significantly lower serum AMH concentration was found in the regularly menstruating CKD women on hemodialysis in comparison with the healthy controls. 2. Serum AMH decreased significantly after successful kidney transplantation.
AMH levels in serum and seminal plasma in healthy men and men with sperm pathology
Genotyping of the AMH c.146G>T and AMHR2 (show AMHR2 ELISA Kits) -482A>G polymorphisms does not provide additional useful information as a predictor of ovarian reserve or ovarian response and treatment outcomes.
The aim of this study was to investigate the density and distribution of single nucleotide polymorphisms (SNPs) anti-Mullerian hormone (AMH) and AMHRII receptors in cryptorchid patients.
Different phenotypes for polycystic ovary syndrome were identified with each related to different levels of anti-mullerian hormone.
Data from a longitudinal, observational study suggest that, among women with type 1 diabetes, AMH levels in serum decline in manner similar to that previously reported in women without diabetes; thus, it is possible that AMH may be used to risk-stratify women with type 1 diabetes at risk for diminished ovarian reserve and poor reproductive outcomes in a similar manner as used in healthy women.
Ile(49)Ser (show SIGLEC1 ELISA Kits) genotype not associated with estradiol levels, ovarian parameters, menstrual cycle length, or pregnancy outcomes in healthy Singapore women
AMH increases GnRH-dependent LH pulsatility and secretion, supporting a central action of AMH on GnRH neurons.
AMH and FOXL2 (show FOXL2 ELISA Kits) collaboratively work to reserve ovarian follicles. AMH is an endogenous target gene of FOXL2 (show FOXL2 ELISA Kits).
Up-regulation of SOX9 (show SOX9 ELISA Kits) in sertoli cells from testiculopathic patients accounts for increasing anti-mullerian hormone expression via impaired androgen receptor (show AR ELISA Kits) signaling.
Male mice require AMH to undergo normal social development.
Data show that Purkinje cells express receptors for Mullerian inhibiting substance (MIS), and that MIS(-/-) male mice have female-like numbers of Purkinje cells and a female-like size to other parts of their cerebellum.
MIS may be involved in anterograde rather than autocrine or retrograde regulation of neurons.
FSH (show BRD2 ELISA Kits) and cAMP stimulate AMH transcription by granulosa cells. FSH (show BRD2 ELISA Kits) and LH have an additive effect, which may be important in polycystic ovary syndrome.
This suggests that MIS is one of the determinants of "boy"-specific behavior.
Role of anti-Mullerian hormone (AMH) as a regulator and marker of ovarian function.
Administration of MIS to male mice induced IEX-1S mRNA in the prostate in vivo, suggesting that MIS may function as an endogenous hormonal regulator of NF-kappaB (show NFKB1 ELISA Kits) signaling and growth in the prostate gland.
Results from these studies indicate that AMH signaling plays a role in both regulating granulosa cell proliferation and preventing granulosa cells from 5- to 8-mm follicles from undergoing premature differentiation before follicle selection.
These findings indicate the followings: AMH mRNA levels decrease in both dominant and secondary follicles during follicular deviation; granulosa cells from heathy follicles express more AMH mRNA compared to subordinate follicles undergoing atresia and FSH (show BRD2 ELISA Kits) stimulates AMH and AMHR2 (show AMHR2 ELISA Kits) mRNA expression in granulosa cells of co-dominant follicles.
Measurement of AMH concentration in the plasma of cows can help to predict their capacity for embryo production in response to gonadotrophin treatment.
In first study to investigate the blood profile and immunohuistochemistry of anti-Mullerian hormone in bovine granulosa-theca cell tumors, the findings indicated that anti-Mullerian hormone is a novel biomarker for granulosa-theca cell tumors in cattle.
Regulation of anti-Mullerian hormone production in the cow.
Intrafollicular AMH was not a marker of cystic development in the cow, but low AMH concentrations in cysts were associated with luteinization.
AMH expression is modulated by androgens in bovine granulosa cells from small follicles.
Anti-Mullerian hormone is a member of the transforming growth factor-beta gene family which mediates male sexual differentiation. Anti-Mullerian hormone causes the regression of Mullerian ducts which would otherwise differentiate into the uterus and fallopian tubes. Some mutations in the anti-Mullerian hormone result in persistent Mullerian duct syndrome.
, anti-Mullerian hormone
, anti-mullerian hormone
, Mullerian inhibiting factor
, Mullerian inhibiting substance
, anti-Muellerian hormone
, muellerian-inhibiting substance
, Mullerian inhibitory substance
, Anti - Mullerian hormone (Mulerian inhibiting substance)