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EAE induced by weak activation of innate immunity requires the NLRP3 inflamma (show CXCR4 ELISA Kits)some and (show CXCR2 ELISA Kits)is sensitive to interferon-beta treatment
lymphatic vessel expansion occurs in two distinct phases; the first wave of expansion is dependent on IL-7 (show IL7 ELISA Kits); the second phase, responsible for leukocyte exit from the glands, is regulated by lymphotoxin (show LTB ELISA Kits) (LT)betaR signaling
LTbetaR is essential for efficient liver regeneration and cooperates with TNFRp55 (show TNFRSF1A ELISA Kits) in this process. Differences in survival kinetics strongly suggest distinct functions for these two cytokine receptors in liver regeneration.
DC-derived LTBR ligands are critical mediators of lymph node cells survival, and LTBR signaling on reticular stromal cells is mediated by Pdpn (show PDPN ELISA Kits) expression.
LTbR plays a role in macrophage-driven inflammation in atherosclerotic lesions from ApoE (show APOE ELISA Kits) knockout mice, probably by augmenting the Ccl5 (show CCL5 ELISA Kits)-mediated recruitment of monocytes.
M1 macrophages as inducer cells that trigger the expression of chemokines by vascular smooth muscle cell independently of lymphotoxin beta receptor signalling
lymphotoxin beta receptor regulates the development of the CCL21 (show CCL21 ELISA Kits)-expressing subset rather than the Aire (show AIRE ELISA Kits)-expressing subset of postnatal Medullary thymic epithelial cells.
LTbetaR activation on macrophages by T cell-derived lymphotoxin alpha(1)beta(2) controls proinflammatory responses by activation of a TRIM30alpha-controlled, counterregulatory signaling pathway to protect against exacerbating inflammatory reactions.
LTbetaR signaling in gut (show GUSB ELISA Kits) lymphoid follicles regulates IL-22 (show IL22 ELISA Kits) production by innate lymphoid cells in response to mucosal pathogen challenge.
expression of tumor necrosis factor (show TNF ELISA Kits) superfamily member 14 and lymphotoxin-beta receptor by motoneurons in vivo correlates with the preferential expression of interferon-gamma (show IFNG ELISA Kits) at disease onset and symptomatic stage in amyotrophic lateral sclerosis mice
membrane-bound lymphotoxin-beta receptor (LTbetaR) and CXC chemokine receptor (show CXCR4 ELISA Kits) 2 (CXCR2 (show CXCR2 ELISA Kits)), is involved in type B EAE development
LIGHT and LTBR interaction increases the survival and proliferation of human bone marrow-derived mesenchymal stem cells, and therefore, LIGHT might play an important role in stem cell therapy.
We found that LTbetaR polymorphisms caused severe BPH (show GLI3 ELISA Kits). Thus, LTbetaR may contribute to the risk of BPH (show GLI3 ELISA Kits) development.
activation enhances the LPS (show IRF6 ELISA Kits)-induced expression of IL-8 (show IL8 ELISA Kits) through NF-kappaB (show NFKB1 ELISA Kits) and IRF-1 (show IRF1 ELISA Kits)
LIGHT, via LTbetaR signaling, may contribute to exacerbation of airway neutrophilic inflammation through cytokine and chemokine (show CCL1 ELISA Kits) production by bronchial epithelial cells.
Relative expression of HVEM (show TNFRSF14 ELISA Kits) and LTbetaR modulates canonical NF-kappaB (show NFKB1 ELISA Kits) and pro-apoptotic signals stimulated by LIGHT.
lymphotoxin-B (show LTB ELISA Kits)-specific receptor.
LTBR gene polymorphisms may be associated with risk of IgA nephropathy in Korean children
Kidney-Tonifying plus Blood-Promoting Recipe regulates CD11b/CD18 (show ITGAM ELISA Kits) and Bcl-2 (show BCL2 ELISA Kits)/Bax (show BAX ELISA Kits) expression in blood leukocytes and improves microcirculatory status in aged patients with kidney deficiency and blood stasis syndrome.
show that a cognate interaction between LTalphabeta on CD4 (show CD4 ELISA Kits)(+) helper T cells and LTbeta (show LTB ELISA Kits) receptor on DCs results in unique signals that are necessary for optimal CD8 (show CD8A ELISA Kits)(+) T-cell expansion via a type I IFN-dependent mechanism
This gene encodes a member of the tumor necrosis factor receptor superfamily. The major ligands of this receptor include lymphotoxin alpha/beta and tumor necrosis factor ligand superfamily member 14. The encoded protein plays a role in signalling during the development of lymphoid and other organs, lipid metabolism, immune response, and programmed cell death. Activity of this receptor has also been linked to carcinogenesis. Alternatively spliced transcript variants encoding multiple isoforms have been observed.
lymphotoxin B receptor
, tumor necrosis factor receptor superfamily member 3
, LT beta-R
, LT-beta receptor
, TNF receptor-related protein
, lymphotoxin-beta receptor
, tumor necrosis factor receptor superfamily, member 3
, tumor necrosis factor C receptor
, tumor necrosis factor receptor 2-related protein
, tumor necrosis factor receptor type III