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ERG (show ERG ELISA Kits) signaling in prostate cancer is driven through PRMT5 (show PRMT5 ELISA Kits)-dependent methylation of the androgen receptor.
AP-1 (show FOSB ELISA Kits) likely plays a more important role in the AR cistrome in fibroblasts.
Results suggest that relief of AR-mediated suppression of BRN2 is a consequence of enzalutamide treatment in castration-resistant prostate cancer that may facilitate the progression of neuroendocrine prostate cancer, especially in men with "non-AR driven" disease.
ID4 (show ID4 ELISA Kits) selectively regulates AR activity through direct interaction with FKBP52 (show FKBP4 ELISA Kits).
Our findings provide evidence for a novel crosstalk and a crossregulation between ING1 and ING2 in regulating androgen receptor-mediated transactivation and suggest that ING2 acts as a novel corepressor that inhibits AR signaling, prostate cancer cell growth, and migration.
Androgen Receptor mRNA Splice Variants are associated with Prostate Cancer.
Our result demonstrated that a shorter GGN (show GGN ELISA Kits) repeat polymorphism cannot increase the risk of prostate cancer compared to the longer GGN (show GGN ELISA Kits) repeats. That's different with previous meta-analysis.
miR (show MLXIP ELISA Kits)-221/-222 are AR-repressed genes and their expression and oncogenic function are associated with AR status in PCa (show FLVCR1 ELISA Kits) cells. The findings provide an explanation of why miR (show MLXIP ELISA Kits)-221/-222 act as oncogenes in the development of CRPC, but their overexpression is not observed in CRPC tumors.
Glyoxalase 2 expression in prostate cancer was dependent on androgen receptor and was aimed at stimulating cell proliferation and eluding apoptosis through a mechanism involving the p53 (show TP53 ELISA Kits)-p21 (show CDKN1A ELISA Kits) axis
Silencing of androgen receptor rescues the muscular spinal atrophy transgenic mice.
Taken together, our findings provide evidence for a novel crosstalk and a crossregulation between ING1 and ING2 in regulating androgen receptor-mediated transactivation and suggest that ING2 acts as a novel corepressor that inhibits AR signaling, prostate cancer cell growth, and migration.
Prostate 5aR1 levels were positively correlated with adjusted prostate most severe lesion scores. Downregulation of androgen receptor and 5alpha-reductase 2, along with upregulation of 5a-reductase 1 in tumors may promote prostatic intraepithelial neoplasia and prostate cancer development in TRAMP (show DPT ELISA Kits) mice
Bisphenol A inhibits Sertoli cell proliferation by interfering with androgen receptor signaling pathway.
Findings indicate that targeting androgen receptor (AR) with ASC (show STS ELISA Kits)-J9 in experimental autoimmune myocarditis (EAM) resulted in an attenuation in the severity of disease and cardiac injury.
The findings suggest that Cyp3a deficiency stimulated the expression of Scap via activation of the AR, which elevated cholesterogenic gene expression levels through activation of SREBP2 (show SREBF2 ELISA Kits) and increased total cholesterol contents in the prostate.
Gli1 and Gli2 exhibited different functions in the regulation of p63 expression or proliferation of p63(+) cells in Kras-AR driven tumors.
Therefore, BA321 is a novel selective androgen receptor modulator (SARM) that may offer a new therapy option for osteoporosis in the male.
We show that Foxp1 (show FOXP1 ELISA Kits) and the androgen receptor are co-expressed in striatal medium spiny neurons and that brain-specific (show CALY ELISA Kits) androgen receptor KO (ArNesCre) mice exhibit reduced Foxp1 (show FOXP1 ELISA Kits) expression in the striatum at E17.5 and P7.5 and an increased Foxp2 (show FOXP2 ELISA Kits) level in the cortex at P7.5. Thus, androgens may contribute to sex-specific differences in Foxp1 (show FOXP1 ELISA Kits) and Foxp2 (show FOXP2 ELISA Kits) expression and ultrasonic vocalizations
Pharmacologic and genetic androgen receptor blockade causes attenuation of abdominal aortic aneurysm formation.
substantial up-regulation of androgen receptor expression during trophoblast giant cell differentiation suggests that androgens may be related to this process and are active products of bovine placental steroidogenesis
no association between the AR CAG polymorphism and the relative risk of prostate cancer in white Brazilian individuals with a CAG repeat (show CELF3 ELISA Kits)
FSHR (show FSHR ELISA Kits) is specifically regulated through androgen receptor in granulosa cells
Roles of IGF-I (show IGF1 ELISA Kits) and the estrogen, androgen and IGF-I (show IGF1 ELISA Kits) receptors in estradiol-17beta- and trenbolone acetate-stimulated proliferation of cultured bovine satellite cells.
In GD20 and PD2 (show PAF1 ELISA Kits) males we found the reduction of the luminal compartment, inflammatory changes, decreased androgen receptor and increased Cx43 (show GJA1 ELISA Kits) expression
Data suggest that signal transduction involving androgen receptor is involved in apoptosis of granulosa cells (as seen in follicular atresia).
Pig ejaculated spermatozoa express androgen receptor.
Sertoli cell maturation during puberty in the stallion was accompanied by a reduced expression of anti-Mullerian hormone (show AMH ELISA Kits) and its receptor, arrest of cell proliferation, increased expression of androgen receptor
The vesicular gland of castrated goats showed significantly lower AR and COX-2 (show COX2 ELISA Kits) immuno-expression than intact goats indicating that both AR and COX-2 (show COX2 ELISA Kits) are androgen dependent.
Androgen receptor (AR) and Wilms' tumor gene 1 expression dramatically decreased after heat treatment in Sertoli cells
The androgen receptor gene is more than 90 kb long and codes for a protein that has 3 major functional domains: the N-terminal domain, DNA-binding domain, and androgen-binding domain. The protein functions as a steroid-hormone activated transcription factor. Upon binding the hormone ligand, the receptor dissociates from accessory proteins, translocates into the nucleus, dimerizes, and then stimulates transcription of androgen responsive genes. This gene contains 2 polymorphic trinucleotide repeat segments that encode polyglutamine and polyglycine tracts in the N-terminal transactivation domain of its protein. Expansion of the polyglutamine tract causes spinal bulbar muscular atrophy (Kennedy disease). Mutations in this gene are also associated with complete androgen insensitivity (CAIS). Two alternatively spliced variants encoding distinct isoforms have been described.
androgen nuclear receptor variant 2
, dihydrotestosterone receptor
, nuclear receptor subfamily 3 group C member 4
, testicular feminization
, androgen receptor (Testicular feminization), same as Tfm
, androgen receptor (dihydrotestosterone receptor; testicular feminization; spinal and bulbar muscular atrophy; Kennedy disease)
, androgen receptor AR
, Ar beta
, androgen receptor
, prostate androgen receptor