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Human BAX ELISA Kit for Sandwich ELISA - ABIN365025
Zhou, Zhao, Ma, Ju, Li, Li, Sun, Shi, Zhang, Lu: The anticancer efficacy of paclitaxel liposomes modified with mitochondrial targeting conjugate in resistant lung cancer. in Biomaterials 2013
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Rat (Rattus) BAX ELISA Kit for Sandwich ELISA - ABIN416240
Fang, Yang, Yang, Chen: Chitosan oligosaccharides attenuates oxidative-stress related retinal degeneration in rats. in PLoS ONE 2013
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Mouse (Murine) BAX ELISA Kit for Sandwich ELISA - ABIN424766
Nakano, Marui, Muranaka, Masumoto, Noma, Tabata, Ido, Tsubouchi, Ikeda, Sakata: Effects of hepatocyte growth factor in myocarditis rats induced by immunization with porcine cardiac myosin. in Interactive cardiovascular and thoracic surgery 2014
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Rat (Rattus) BAX ELISA Kit for Sandwich ELISA - ABIN583629
Zhang, Feng, Gao, Sun, Wang, Li, Wang, Zhao, Hu, Sun, Wei, Sun: Role of pigment epithelium-derived factor (PEDF) in arsenic-induced cell apoptosis of liver and brain in a rat model. in Biological trace element research 2013
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Mouse (Murine) BAX ELISA Kit for Sandwich ELISA - ABIN832356
Hao, Huang, Gu, Liu, Li, Liu, Ren, Yang, Peng, Wang, Li: Metallothionein deficiency aggravates depleted uranium-induced nephrotoxicity. in Toxicology and applied pharmacology 2015
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Human BAX ELISA Kit for Sandwich ELISA - ABIN414428
Gornowicz, Bielawska, Czarnomysy, Gabryel-Porowska, Muszyńska, Bielawski: The combined treatment with novel platinum(II) complex and anti-MUC1 increases apoptotic response in MDA-MB-231 breast cancer cells. in Molecular and cellular biochemistry 2015
Bid (show BID ELISA Kits) and Bax are signal transduction factors in granulosa cells and play proapoptotic roles.
The data demonstrate that severe hypocapnia results in increased Bax expression, DNA fragmentation, and membrane lipid peroxidation in mitochondria of cerebral cortical neurons of newborn piglets, and may result in apoptotic cell death.
BoHV-5 replication apparently modulates BCL-2 (show BCL2 ELISA Kits) expression and gene transcription, enhancing production of virus progeny, but does not increase Bax expression.
It was concluded that the Fas (show FAS ELISA Kits)-FasL (show FASL ELISA Kits) signaling pathway was involved in regulation of bovine oocyte apoptosis, perhaps related to B (show TDO2 ELISA Kits) cell lymphoma/leukemia-2 associated X upregulation.
apoptosis-inducing factor (show AIFM1 ELISA Kits) was expressed in luminal alveolar cells and, in concert with a change in bax protein to bcl-2 (show BCL2 ELISA Kits) protein ratio, might contribute to signalling of a change in the dynamic balance of the cell population as lactation progresses
mRNA expression of Bax, Bcl-2 (show BCL2 ELISA Kits), caspase-3 (show CASP3 ELISA Kits) and-7 cannot be used as a reliable apoptosis detection method.
The effect of culture conditions on expression of heat shock protein 70 (show HSP70 ELISA Kits) and Bax protein in bovine blastocysts is reported.
xlbax is a regulator of muscle fiber death in the regressing tail during metamorphosis.
These results suggest a role for mitochondrial p53 (show TP53 ELISA Kits) activity in promoting hair cell death due to aminoglycosides, likely upstream of Bax and Bcl2 (show BCL2 ELISA Kits).
YY1 (show YY1 ELISA Kits) promotes apoptosis via upregulating Bax transcription and subsequent activation of Bax by translocation from the cytosol to the mitochondrial membrane.
Immunohistochemical analysis showed that STAT3 (show STAT3 ELISA Kits), GRP78 (show HSPA5 ELISA Kits) and BAX protein levels in the combination group were significantly higher than those in STAT3 (show STAT3 ELISA Kits) group and CDDP group (P<0.05). Exogenous STAT3 (show STAT3 ELISA Kits) and CDDP may synergistically inhibit the xenograft tumour growth through up-regulation of BAX protein via GRP78 (show HSPA5 ELISA Kits).
This is the first study evaluating the potential relationship between BCL2 (show BCL2 ELISA Kits) and BAX gene polymorphisms and RRD (show DHRS4 ELISA Kits) in a Greek population, showing a significant association between BAX rs4645878 polymorphism and RRD (show DHRS4 ELISA Kits) susceptibility. This finding suggests that an apoptotic mechanism is implicated in the pathogenesis of RRD (show DHRS4 ELISA Kits)
Heavy ion irradiation could induce p53 (show TP53 ELISA Kits)(-/-) hepatoma cells to undergo apoptosis via E2F1 (show E2F1 ELISA Kits)/Bax/Casp3 (show CASP3 ELISA Kits) signaling pathway.
Bax effects were dependent on its oligomeric state. Monomeric Bax did not affect the membrane, oligomeric Bax lowered the breakthrough force of the membrane, which in the context of pore formation, implies a lowering of the line tension at the edge of the pore.
The results strongly suggest that the direct apoptosis-activation activities of Bid (show BID ELISA Kits), Bim (show BCL2L11 ELISA Kits), Puma (show BBC3 ELISA Kits), and p53 (show TP53 ELISA Kits) are not essential for activating Bax/Bak (show BAK1 ELISA Kits) once the anti-apoptotic Bcl-2 (show BCL2 ELISA Kits) proteins are neutralized.
The C-terminal helical conformation of Bax, not its primary sequence, appears to be critical for CASP8 (show CASP8 ELISA Kits) recruitment and activation culminating in cell death.
An autoinhibited dimeric form of BAX regulates the cytosolic BAX activation pathway.
Although TP53 (show TP53 ELISA Kits) and BAX immunoreactivity levels were associated with some clinicopathological parameters of the patients, the expression of EP300 (show EP300 ELISA Kits), TP53 (show TP53 ELISA Kits) and BAX did not reveal any prognostic significance in ccRCC
Cells expressing mitoCERT import ceramides into mitochondria and undergo Bax-dependent apoptosis.
although all CR subtypes undergo cell death, septum, but not hem, CRs (show CARS ELISA Kits) die in a Bax-dependent manner. Bax-inactivated rescued septum-CRs (show CARS ELISA Kits) maintain immature electrophysiological properties
Postnatal synaptic rearrangement needed for acquisition of skilled behaviors requires the activity-dependent, non-apoptotic Bax/Bak (show BAK1 ELISA Kits)-caspase (show CASP3 ELISA Kits) signaling cascade. Adult Bax/Bak (show BAK1 ELISA Kits) mutant mice exhibit aberrant co-activation of antagonistic muscle pairs and skilled grasping deficits but normal reaching and retrieval behaviors.
Study found that emphysema occurred in ku70(-/-) mice at the age of three-months old, and that Bax deficiency was able to suppress it. These results suggest that Bax-mediated apoptosis induces emphysema in ku70(-/-) mice.
The polyglutamine embedded in the ER membrane was observed at the same time as Bax insertion. These results demonstrated that the ER membrane may be a target of polyglutamine, which triggers cell death through Bax.
These results suggest that Bax mediates beta-cell apoptosis in Pdx1 (show PDX1 ELISA Kits)-deficient diabetes.
Bim (show BCL2L11 ELISA Kits) and Puma (show BBC3 ELISA Kits) overlapped apoptosis only partially during physiological apoptotic stage and they were present in non-apoptotic parts of the follicles.
This study demonstated that Bax KO mice show Hyperactivity and depression.
The influence of chronic ethanol consumption on the expression of the Bdnf (show BDNF ELISA Kits), Bax, Bcl-xL (show BCL2L1 ELISA Kits), and CASP3 (show CASP3 ELISA Kits) genes was studied in the brain structures of B6-1473C (C/C) and B6-1473G (G/G) mice that had been obtained on the base of the C57BL/6 strain.
These data indicate that the rapid and robust microglial activation and PIF (show DCD ELISA Kits) expression observed in situ following acute EtOH exposure is largely driven indirectly by BAX-dependent apoptotic cell death, rather than directly by the EtOH.
The content of Bax protein in the cardiomyocyte cytoplasm decreased, thus indicating that the mitochondrial pathway was not involved in the realization of the apoptotic program in a model of ventricular overload.
These results indicate that RI-PostC can ameliorate myocardial ischemia-reperfusion injury and increase the Bcl-2 (show BCL2 ELISA Kits)/Bax ratio through a mechanism involving protein kinase C (show PKC ELISA Kits).
Cinobufagin can remarkably inhibit the proliferation and induce the apoptosis of lens epithelial cells by increasing expression of bax and decreasing expression of bcl2 (show BCL2 ELISA Kits).
Elevated local temperature of the testis buried in the inguinal pocket increases the apoptosis of spermatogenic cells, and the spermatogenic cell apoptosis is highly correlated with the decreased expression of Bcl-2 (show BCL2 ELISA Kits) and increased expression of Bax.
In this study evidence is provided that exogenous PGF2alpha differentially modulates luteal expression of BCL2-associated X protein (BAX) transcripts and protein depending on luteal stage.
bcl-2 (show BCL2 ELISA Kits) and bax were expressed strongly in denervated guinea-pig facial muscle. [bcl-2 (show BCL2 ELISA Kits); bax]
goat oocytes based on G6PDH (show G6PD ELISA Kits)-activity through the BCB test improves their developmental competence, increases intracellular GSH content, and affects the expression of the apoptosis-related genes9 Bax and Bcl-2 (show BCL2 ELISA Kits) ).
The protein encoded by this gene belongs to the BCL2 protein family. BCL2 family members form hetero- or homodimers and act as anti- or pro-apoptotic regulators that are involved in a wide variety of cellular activities. This protein forms a heterodimer with BCL2, and functions as an apoptotic activator. This protein is reported to interact with, and increase the opening of, the mitochondrial voltage-dependent anion channel (VDAC), which leads to the loss in membrane potential and the release of cytochrome c. The expression of this gene is regulated by the tumor suppressor P53 and has been shown to be involved in P53-mediated apoptosis. Multiple alternatively spliced transcript variants, which encode different isoforms, have been reported for this gene.
BCL2-associated X protein
, BCL2-associated protein
, apoptosis regulator BAX
, bax zeta
, BCL2-associated X protein omega
, BCL2-associated X protein transcript variant delta2
, bcl-2-like protein 4