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Browse our HRAS (HRAS) ELISA Kits

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V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog ELISA Kits (HRAS)
On are 13 V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog (HRAS) ELISA Kits from 3 different suppliers available. Additionally we are shipping HRAS Antibodies (138) and HRAS Proteins (20) and many more products for this protein. A total of 180 HRAS products are currently listed.
AI929937, C-BAS/HAS, c-H-ras, c-Ha-ras, C-HA-RAS1, c-rasHa, CTLO, H-ras, H-RASIDX, Ha-ras, HAMSV, Harvey-ras, hras, Hras-1, HRAS1, K-ras, Ki-ras, Kras-2, Kras2, N-RAS, p21B, ras, RASH1, zgc:110250

More ELISA Kits for HRAS Interaction Partners

Cow (Bovine) V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog (HRAS) interaction partners

  1. Data demonstrate that H-Ras activation is important in the activation of the specific signaling events leading to the accelerated retinal capillary cell apoptosis in hyperglycemic conditions.

  2. Activation of H-Ras and its downstream signaling pathway in the retina and its vasculature could be under the control of superoxide, and H-Ras activation in diabetes can be prevented by inhibiting superoxide accumulation.

  3. Thrombospondin 1 (show THBS1 ELISA Kits), fibronectin (show FN1 ELISA Kits), and vitronectin (show VTN ELISA Kits) are differentially dependent upon RAS, ERK1/2 (show MAPK1/3 ELISA Kits), and p38 (show MAPK14 ELISA Kits) for induction of vascular smooth muscle cell chemotaxis.

Zebrafish V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog (HRAS) interaction partners

  1. Kita driven expression of oncogenic HRAS leads to early onset and highly penetrant melanoma in zebrafish

Human V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog (HRAS) interaction partners

  1. herefore, our results also demonstrate that the phenotype in CS and somatic cancers is not only determined by the different transforming potentials of mutant HRAS proteins, but also by the efficiency of exon 2 inclusion resulting from the different HRAS mutations.

  2. Point mutations of HRAS is associated with thyroid cancer.

  3. The results show that mutant RAS is associated with an earlier and higher rate of local tumour progression in patients undergoing ablation of CLMs.

  4. A report of three cases with pediatric urothelial bladder cancer indicates that the tumors are characterized by a consistent H-RAS mutation status, whereas FGFR3 (show FGFR3 ELISA Kits) and p53 (show TP53 ELISA Kits) pathways are not involved in this tumor initiation.

  5. The coexistence of BRAF (show BRAF ELISA Kits) or RAS mutations enhanced the prognostic effects of telomerase reverse transcriptase (TERT (show TERT ELISA Kits)) promoter mutations. Furthermore, TERT (show TERT ELISA Kits) promoter mutations strengthened the predictions of mortality and recurrence by the ATA and TNM (show ODZ1 ELISA Kits) staging systems, particularly for high-risk patients with differentiated thyroid cancer.

  6. Overexpression of HRAS is associated with Breast Cancer Recurrence.

  7. Study acts as a further supplement of the genetic features of neuroendocrine tumors. Somatic mutations of three potential tumor-related genes (HRAS, PAK1 (show PAK1 ELISA Kits) and MEN1 (show MEN1 ELISA Kits)) might contribute to the tumorigenesis of thymic neuroendocrine tumors with EAS.

  8. Activating HRAS, KRAS and EGFR (show EGFR ELISA Kits) mutations play a major role in the pathogenesis of sporadic SGH. These results support the concept that SGH is a true benign neoplasm rather than a reactive hyperplasia.

  9. HRAS mutation is associated with liver cancer.

  10. findings support a role of HRAS mutations as a somatic driver event in benign PPGL without other known susceptibility gene mutations. HRAS mutated PPGL cluster together with NF1 (show NF1 ELISA Kits)- and RET (show RET ELISA Kits)-mutated tumors associated with activation of kinase-signaling pathways

Mouse (Murine) V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog (HRAS) interaction partners

  1. We establishment the transgenic mouse model of K-ras(G12D) p53 (show TP53 ELISA Kits)(loxP/loxP) and found that K-ras mutation and p53 (show TP53 ELISA Kits) deletion within the ovarian surface epithelium gave rise to ovarian lesions with a hyperproliferation and endometrioid glandular morphology

  2. K-Ras knockout (KO) mouse embryonic fibroblasts (K-ras(-/-) ) stimulated with transforming growth factor-beta1 (TGF-beta1 (show TGFB1 ELISA Kits)) exhibited reduced proliferation and impaired mobility than wild-type fibroblasts

  3. The abnormal expression of epidermal cytokeratins suggests that Ha-Ras and Bcl-2 (show BCL2 ELISA Kits) suppress the terminal differentiation and sustain the stem cell-like features in epidermal keratinocytes

  4. findings demonstrate that advanced gastric tumorigenesis requires oncogenic KRAS or BRAF (show BRAF ELISA Kits) in concert with aberrant STAT3 (show STAT3 ELISA Kits) activation in epithelial precursor cells of the glandular stomach

  5. Ras (G12V)-induced cyclin D1 (show CCND1 ELISA Kits) protein synthesis was markedly suppressed by the knockdown of IL-33 (show IL33 ELISA Kits).

  6. oncogenic KRas-induced increase in fluid-phase endocytosis has a key role during cellular transdifferentiation in pancreatic acinar cells. This result supports emerging evidence for endocytosis playing a crucial role in regulating the signaling output of the cells

  7. This study reveals KRAS(G12D) uniquely regulates tumor cells via heterotypic stromal cells. By exploiting heterocellularity, reciprocal signaling enables KRAS(G12D) to engage oncogenic signaling pathways beyond those regulated in a cell-autonomous manner.

  8. We also uncovered that Prox1-heterozygosis markedly increases the formation of acinar-to-ductal-metaplasias and early neoplasias, and enhances features associated with inflammation, in mouse pancreatic tissues expressing oncogenic Kras.

  9. The mutant KRas can initiate the transcription of the epidermal growth factor (show EGF ELISA Kits) receptors and its ligands by increasing mitochondrial generation of ROS (show ROS1 ELISA Kits) and activation of NF-kB signaling through protein kinase D1 (show PRKD1 ELISA Kits).

  10. Findings suggest that COX-2 (show COX2 ELISA Kits) deletion contributes to the repression of K-ras-induced lung tumorigenesis by reducing tumor cell proliferation, decreasing the production of PGE2, and increasing the production of 13,14-dihydro-15-keto-PGE2, possibly via the MAPK (show MAPK1 ELISA Kits) pathway.

HRAS Antigen Profile

Antigen Summary

This gene belongs to the Ras oncogene family, whose members are related to the transforming genes of mammalian sarcoma retroviruses. The products encoded by these genes function in signal transduction pathways. These proteins can bind GTP and GDP, and they have intrinsic GTPase activity. This protein undergoes a continuous cycle of de- and re-palmitoylation, which regulates its rapid exchange between the plasma membrane and the Golgi apparatus. Mutations in this gene cause Costello syndrome, a disease characterized by increased growth at the prenatal stage, growth deficiency at the postnatal stage, predisposition to tumor formation, mental retardation, skin and musculoskeletal abnormalities, distinctive facial appearance and cardiovascular abnormalities. Defects in this gene are implicated in a variety of cancers, including bladder cancer, follicular thyroid cancer, and oral squamous cell carcinoma. Multiple transcript variants, which encode different isoforms, have been identified for this gene.

Alternative names and synonyms associated with HRAS

  • Harvey rat sarcoma viral oncogene homolog (hras) Elisa Kit
  • v-Ha-ras Harvey rat sarcoma viral oncogene homolog (HRAS) Elisa Kit
  • v-Ha-ras Harvey rat sarcoma viral oncogene homolog a (hrasa) Elisa Kit
  • neuroblastoma RAS viral (v-ras) oncogene homolog (NRAS) Elisa Kit
  • v-Ki-ras2 Kirsten rat sarcoma viral oncogene homolog (KRAS) Elisa Kit
  • Harvey rat sarcoma viral oncogene homolog (HRAS) Elisa Kit
  • Harvey rat sarcoma virus oncogene (Hras) Elisa Kit
  • Harvey rat sarcoma virus oncogene 1 (Hras1) Elisa Kit
  • v-Ha-ras Harvey rat sarcoma viral oncogene homolog (Hras) Elisa Kit
  • v-Ki-ras2 Kirsten rat sarcoma viral oncogene homolog (Kras) Elisa Kit
  • AI929937 Elisa Kit
  • C-BAS/HAS Elisa Kit
  • c-H-ras Elisa Kit
  • c-Ha-ras Elisa Kit
  • C-HA-RAS1 Elisa Kit
  • c-rasHa Elisa Kit
  • CTLO Elisa Kit
  • H-ras Elisa Kit
  • H-RASIDX Elisa Kit
  • Ha-ras Elisa Kit
  • HAMSV Elisa Kit
  • Harvey-ras Elisa Kit
  • hras Elisa Kit
  • Hras-1 Elisa Kit
  • HRAS1 Elisa Kit
  • K-ras Elisa Kit
  • Ki-ras Elisa Kit
  • Kras-2 Elisa Kit
  • Kras2 Elisa Kit
  • N-RAS Elisa Kit
  • p21B Elisa Kit
  • ras Elisa Kit
  • RASH1 Elisa Kit
  • zgc:110250 Elisa Kit

Protein level used designations for HRAS

v-Ha-ras Harvey rat sarcoma viral oncogene homolog , small G-protein H-Ras , Harvey ras 1 , p19 H-RasIDX protein , GTPase HRas (Transforming protein p21) (H-Ras-1) (c-H-ras) , xRAS1 , GTPase HRas , Transforming protein p21 , neuroblastoma ras oncogene , GTP- and GDP-binding peptide B , H-Ras-1 , Ha-Ras1 proto-oncoprotein , Ras family small GTP binding protein H-Ras , c-has/bas p21 protein , c-ras-Ki-2 activated oncogene , p21ras , transformation gene: oncogene HAMSV , transforming protein p21 , Harvey ras1 protein , Harvey rat sarcoma viral (v-Ha-ras) oncogene homolog , H-ras 1 protein , c-Ha-ras p21 protein , c-Ha-ras transgene , transforming protein P21 , GTPase NRas , transforming protein N-Ras , c-H-ras , v-Ha-ras Harvey rat sarcoma viral oncogene-like protein , ras p21 , GTPase KRas , K-Ras 2 , Kirsten rat sarcoma oncogene 2, expressed , c-K-ras , c-Ki-ras , p21 protein

399406 Xenopus laevis
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549757 Xenopus (Silurana) tropicalis
550286 Danio rerio
698830 Macaca mulatta
733587 Sus scrofa
100222058 Taeniopygia guttata
100227892 Taeniopygia guttata
3265 Homo sapiens
293621 Rattus norvegicus
15461 Mus musculus
100739349 Sus scrofa
100735038 Cavia porcellus
396229 Gallus gallus
403735 Canis lupus familiaris
100298939 Bos taurus
101104357 Ovis aries
16653 Mus musculus
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