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Browse our HRAS (HRAS) ELISA Kits

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V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog ELISA Kits (HRAS)
On are 13 V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog (HRAS) ELISA Kits from 3 different suppliers available. Additionally we are shipping HRAS Antibodies (130) and HRAS Proteins (20) and many more products for this protein. A total of 172 HRAS products are currently listed.
AI929937, C-BAS/HAS, c-H-ras, c-Ha-ras, C-HA-RAS1, c-rasHa, CTLO, H-ras, H-RASIDX, Ha-ras, HAMSV, Harvey-ras, hras, Hras-1, HRAS1, K-ras, Ki-ras, Kras-2, Kras2, N-RAS, p21B, ras, RASH1, zgc:110250

More ELISA Kits for HRAS Interaction Partners

Cow (Bovine) V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog (HRAS) interaction partners

  1. Data demonstrate that H-Ras activation is important in the activation of the specific signaling events leading to the accelerated retinal capillary cell apoptosis in hyperglycemic conditions.

  2. Activation of H-Ras and its downstream signaling pathway in the retina and its vasculature could be under the control of superoxide, and H-Ras activation in diabetes can be prevented by inhibiting superoxide accumulation.

  3. Thrombospondin 1 (show THBS1 ELISA Kits), fibronectin (show FN1 ELISA Kits), and vitronectin (show VTN ELISA Kits) are differentially dependent upon RAS, ERK1/2 (show MAPK1/3 ELISA Kits), and p38 (show MAPK14 ELISA Kits) for induction of vascular smooth muscle cell chemotaxis.

Zebrafish V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog (HRAS) interaction partners

  1. Kita driven expression of oncogenic HRAS leads to early onset and highly penetrant melanoma in zebrafish

Human V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog (HRAS) interaction partners

  1. Activating HRAS, KRAS and EGFR (show EGFR ELISA Kits) mutations play a major role in the pathogenesis of sporadic SGH. These results support the concept that SGH is a true benign neoplasm rather than a reactive hyperplasia.

  2. HRAS mutation is associated with liver cancer.

  3. findings support a role of HRAS mutations as a somatic driver event in benign PPGL without other known susceptibility gene mutations. HRAS mutated PPGL cluster together with NF1 (show NF1 ELISA Kits)- and RET (show RET ELISA Kits)-mutated tumors associated with activation of kinase-signaling pathways

  4. Ras activity fine tunes the period length and modulates photoentrainment of the circadian clock.

  5. mTOR kinase inhibitor pp242 causes mitophagy terminated by apoptotic cell death in E1A-Ras transformed cells.

  6. Data show that heterogeneous nuclear ribonucleoprotein A1 (hnRNP A1 (show HNRNPA1 ELISA Kits)) unfolds the HRAS protein i (show ANXA2 ELISA Kits)-motif.

  7. Data show that Ba/F3 cells transformed with mutant HRAS protien indicated equal sensitivity towards Map kinase (show MAPK1 ELISA Kits) kinase (MEK (show MAP2K1 ELISA Kits)) and mTOR (show FRAP1 ELISA Kits) serine-threonine kinase (show TLK2 ELISA Kits) (mTOR (show FRAP1 ELISA Kits)) inhibition.

  8. HRAS differentially influences iTBS- and PAS (show PASK ELISA Kits)-induced LTP (show SCP2 ELISA Kits).

  9. our data suggest roles for HRAS and EGFR (show EGFR ELISA Kits) as drivers in a subset of poroma and porocarcinoma.

  10. Endocytosis separates EGF (show EGF ELISA Kits) receptors from endogenous fluorescently labeled HRas and diminishes receptor signaling to MAP kinases in endosomes.

Mouse (Murine) V-Ha-Ras Harvey Rat Sarcoma Viral Oncogene Homolog (HRAS) interaction partners

  1. This study reveals KRAS(G12D) uniquely regulates tumor cells via heterotypic stromal cells. By exploiting heterocellularity, reciprocal signaling enables KRAS(G12D) to engage oncogenic signaling pathways beyond those regulated in a cell-autonomous manner.

  2. We also uncovered that Prox1-heterozygosis markedly increases the formation of acinar-to-ductal-metaplasias and early neoplasias, and enhances features associated with inflammation, in mouse pancreatic tissues expressing oncogenic Kras.

  3. The mutant KRas can initiate the transcription of the epidermal growth factor (show EGF ELISA Kits) receptors and its ligands by increasing mitochondrial generation of ROS (show ROS1 ELISA Kits) and activation of NF-kB signaling through protein kinase D1 (show PRKD1 ELISA Kits).

  4. Findings suggest that COX-2 (show COX2 ELISA Kits) deletion contributes to the repression of K-ras-induced lung tumorigenesis by reducing tumor cell proliferation, decreasing the production of PGE2, and increasing the production of 13,14-dihydro-15-keto-PGE2, possibly via the MAPK (show MAPK1 ELISA Kits) pathway.

  5. Data indicate that KrasG12D;Pdx1 (show PDX1 ELISA Kits)-Cre mouse model enables one to monitor the progression of pancreatic cancer (PC) from the onset of precursor lesions to cancer development.

  6. in this overview are the features of various KRAS-driven genetically engineered mouse models (GEMMs) of non-small cell lung cancer--{REVIEW}

  7. Binase-KRAS interaction interferes with the function of GEFs and stabilizes the inactive GDP-bound conformation of RAS thereby inhibiting MAPK/ERK (show MAPK1 ELISA Kits) signaling and oncogenic activity.

  8. Data show that in both GTP- and GDP-bound states of the K-Ras, the catalytic domain exhibited similar membrane orientations.

  9. our results highlight the importance of Pas1 as a tumor-modifier locus, attribute to it a novel role as a major regulator of transcription in lung tumor nodules and strengthen the candidacy of the Kras gene as the effector of this locus.

  10. demonstrated that despite having both Kras and Pik3ca (show PIK3CA ELISA Kits) mutations at the time of tumor initiation from loss of APC (show APC ELISA Kits)

HRAS Antigen Profile

Antigen Summary

This gene belongs to the Ras oncogene family, whose members are related to the transforming genes of mammalian sarcoma retroviruses. The products encoded by these genes function in signal transduction pathways. These proteins can bind GTP and GDP, and they have intrinsic GTPase activity. This protein undergoes a continuous cycle of de- and re-palmitoylation, which regulates its rapid exchange between the plasma membrane and the Golgi apparatus. Mutations in this gene cause Costello syndrome, a disease characterized by increased growth at the prenatal stage, growth deficiency at the postnatal stage, predisposition to tumor formation, mental retardation, skin and musculoskeletal abnormalities, distinctive facial appearance and cardiovascular abnormalities. Defects in this gene are implicated in a variety of cancers, including bladder cancer, follicular thyroid cancer, and oral squamous cell carcinoma. Multiple transcript variants, which encode different isoforms, have been identified for this gene.

Alternative names and synonyms associated with HRAS

  • Harvey rat sarcoma viral oncogene homolog (hras) Elisa Kit
  • v-Ha-ras Harvey rat sarcoma viral oncogene homolog (HRAS) Elisa Kit
  • v-Ha-ras Harvey rat sarcoma viral oncogene homolog a (hrasa) Elisa Kit
  • neuroblastoma RAS viral (v-ras) oncogene homolog (NRAS) Elisa Kit
  • v-Ki-ras2 Kirsten rat sarcoma viral oncogene homolog (KRAS) Elisa Kit
  • Harvey rat sarcoma viral oncogene homolog (HRAS) Elisa Kit
  • Harvey rat sarcoma virus oncogene (Hras) Elisa Kit
  • Harvey rat sarcoma virus oncogene 1 (Hras1) Elisa Kit
  • v-Ha-ras Harvey rat sarcoma viral oncogene homolog (Hras) Elisa Kit
  • v-Ki-ras2 Kirsten rat sarcoma viral oncogene homolog (Kras) Elisa Kit
  • AI929937 Elisa Kit
  • C-BAS/HAS Elisa Kit
  • c-H-ras Elisa Kit
  • c-Ha-ras Elisa Kit
  • C-HA-RAS1 Elisa Kit
  • c-rasHa Elisa Kit
  • CTLO Elisa Kit
  • H-ras Elisa Kit
  • H-RASIDX Elisa Kit
  • Ha-ras Elisa Kit
  • HAMSV Elisa Kit
  • Harvey-ras Elisa Kit
  • hras Elisa Kit
  • Hras-1 Elisa Kit
  • HRAS1 Elisa Kit
  • K-ras Elisa Kit
  • Ki-ras Elisa Kit
  • Kras-2 Elisa Kit
  • Kras2 Elisa Kit
  • N-RAS Elisa Kit
  • p21B Elisa Kit
  • ras Elisa Kit
  • RASH1 Elisa Kit
  • zgc:110250 Elisa Kit

Protein level used designations for HRAS

v-Ha-ras Harvey rat sarcoma viral oncogene homolog , small G-protein H-Ras , Harvey ras 1 , p19 H-RasIDX protein , GTPase HRas (Transforming protein p21) (H-Ras-1) (c-H-ras) , xRAS1 , GTPase HRas , Transforming protein p21 , neuroblastoma ras oncogene , GTP- and GDP-binding peptide B , H-Ras-1 , Ha-Ras1 proto-oncoprotein , Ras family small GTP binding protein H-Ras , c-has/bas p21 protein , c-ras-Ki-2 activated oncogene , p21ras , transformation gene: oncogene HAMSV , transforming protein p21 , Harvey ras1 protein , Harvey rat sarcoma viral (v-Ha-ras) oncogene homolog , H-ras 1 protein , c-Ha-ras p21 protein , c-Ha-ras transgene , transforming protein P21 , GTPase NRas , transforming protein N-Ras , c-H-ras , v-Ha-ras Harvey rat sarcoma viral oncogene-like protein , ras p21 , GTPase KRas , K-Ras 2 , Kirsten rat sarcoma oncogene 2, expressed , c-K-ras , c-Ki-ras , p21 protein

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3265 Homo sapiens
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100735038 Cavia porcellus
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100298939 Bos taurus
101104357 Ovis aries
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