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Rat (Rattus) F3 ELISA Kit for Sandwich ELISA - ABIN431567
Li, Guo, Chen, Hu, Chen: Increased plasma level of asymmetric dimethylarginine in hypertensive rats facilitates platelet aggregation: role of plasma tissue factor. in Canadian journal of physiology and pharmacology 2011
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Human F3 ELISA Kit for Sandwich ELISA - ABIN415041
Tu, Hu, Chen: Endothelial gene expression and molecular changes in response to radiosurgery in in vitro and in vivo models of cerebral arteriovenous malformations. in BioMed research international 2013
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Mouse (Murine) F3 ELISA Kit for Sandwich ELISA - ABIN415624
Mizugishi, Inoue, Hatayama, Bielawski, Pierce, Sato, Takaori-Kondo, Konishi, Yamashita: Sphingolipid Pathway Regulates Innate Immune Responses at the Fetomaternal Interface during Pregnancy. in The Journal of biological chemistry 2015
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Mouse (Murine) F3 ELISA Kit for Sandwich ELISA - ABIN2859236
Wu, Ming, Zhang, Wu, Wu, Yao: Edaravone rescues the lung by inhibiting lipid peroxidation and pro-inflammatory cytokines in a rat model. in Chinese medical journal 2014
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Rat (Rattus) F3 ELISA Kit for Sandwich ELISA - ABIN368173
Milano, Dongiovanni, Artoni, Gatti, Rosso, Colombo, Bollati, Maggioni, Mannucci, Bertazzi, Fargion, Valenti: Particulate matter phagocytosis induces tissue factor in differentiating macrophages. in Journal of applied toxicology : JAT 2015
Ticagrelor, but not clopidogrel, reduces arterial thrombosis via endothelial tissue factor suppression. Ticagrelor reduced TNF-alpha (show TNF ELISA Kits)-induced TF expression via proteasomal degradation.
Macrophage tissue factor prothrombotic activity is regulated by integrin-alpha4/arf6 (show ARF6 ELISA Kits) trafficking.
Evening primrose oil and forskolin decreased the prothrombotic effect of celecoxib initiated by a LPS (show TLR4 ELISA Kits) challenge in mice, and this effect was, at least partly, mediated by mitigating TF expression and activity.
Thrombin (show F2 ELISA Kits)-independent contribution of tissue factor to inflammation and cardiac hypertrophy in a mouse model of sickle cell disease.
These findings reveal a novel biological function and mechanism of the protein C (show PROC ELISA Kits) pathway in which protein S and the aPC (show APC ELISA Kits)-cleaved form of fV are cofactors for anti-inflammatory cell signaling by aPC (show APC ELISA Kits) in the context of endotoxemia and infection
Lung epithelial tissue factor regulates alveolar procoagulant activity and permeability in acute lung injury.
PGE2 increases both TF expression and activity through the regulation of the EP1 (show PTGER1 ELISA Kits)/SIRT1 (show SIRT1 ELISA Kits) pathway.
Fas (show FAS ELISA Kits)-initiated, caspase-3 (show CASP3 ELISA Kits)-dependent hepatocyte apoptosis increases tissue factor procoagulant activity through a mechanism involving phosphatidylserine externalization.
Heme promotes tissue factor-dependent coagulation activation and induces tissue factor expression on leukocytes in vivo.
TF is essential for the development of pericyte coverage of tumor microvessels and aPL (show FASL ELISA Kits)-induced tumor cell expression of chemokine ligand 2 (show CXCL2 ELISA Kits), a mediator of pericyte recruitment
Platelet tissue factor activity and membrane cholesterol are increased in hypercholesterolemia and normalized by rosuvastatin, but not by atorvastatin.
the identification of platelet TF and TLR4 (show TLR4 ELISA Kits) as regulators of the effect of E. coli O111 might represent a novel therapeutic target to reduce the devastating consequences of the hemostatic disorder during sepsis.
A coagulation initiating pathway is revealed in which the TF-FVIIa-nascent FXa (show F10 ELISA Kits) complex activates FVIII (show F8 ELISA Kits) apart from thrombin (show F2 ELISA Kits) feedback.
TF-induced microvessel stabilization is regulated via PAR2 (show F2RL1 ELISA Kits)-SMAD3 (show SMAD3 ELISA Kits) that is indispensable for the maintenance of vascular integrity.
The aim of this study was to evaluate the concentration of TF and its inhibitor TFPI (show TFPI ELISA Kits) in blood plasma, the impact of traditional and non-traditional cardiovascular risk factors on their concentration and the impact of both markers of haemostasis on the severity of subclinical atherosclerosis.
Inhibition of the inflammatory signaling intermediate p38 MAPK (show MAPK14 ELISA Kits) reduced tissue factor (TF) mRNA by one third but increased tumor necrosis factor (TNF (show TNF ELISA Kits)) and interleukin-1 beta (IL-1beta (show IL1B ELISA Kits)) mRNA.
TF levels were significantly elevated in type 2 diabetes mellitus (both with and without cardiovascular complications) when compared to the controls. We suggest that pathologic plasma TF activity, as marker of increased propensity of clot (show TXNDC17 ELISA Kits) pathology, should be investigated.
this study shows that low levels of circulating tissue factor may contribute to the reduced coagulopathy reported in patients infected with Neisseria meningitidis lpxL1 mutants
Arterial (18)F-fluorodeoxyglucose uptake reflects balloon catheter-induced thrombus formation and tissue factor expression via nuclear factor-kappaB in rabbit atherosclerotic lesions.
Polycations could present a new class of anticoagulants with such unique upstream downregulation of blood coagulation, selectively blocking tissue factor-dependent factor VII (show TH ELISA Kits) activation.
Upregulated TF expression and increased plasma TF level during reperfusion period, reduced plasma TFPI-1 (show TFPI ELISA Kits) level during reperfusion period.
lectin-like oxidized LDL receptor (show OLR1 ELISA Kits) Oxidized low-density lipoprotein receptor (show LDLR ELISA Kits) 1expression appears to be closely associated with tissue factor expression, apoptotic events and morphological vulnerability in atherosclerotic lesions
Tissue factor expression on porcine neonatal islet cell clusters is an important initiator of instant blood-mediated inflammatory reaction in islet xenotransplantation.
Prolonged clopidogrel treatment reduced coronary tissue factor (TF) expression and tended to reduce the blood TF level post-PCI (show SERPINA5 ELISA Kits), thus possibly modulating the risk of late thrombosis.
Procoagulant porcine tissue factor is induced in primary pig aortic endothelial cells only by fresh human plasma, and not by heat-inactivated plasma.
myocardial infarction induced proinflammatory gene and protein expression in peripheral blood mononuclear cells of tissue factor cyclo-oxygenase-2, monocyte chemoattractant protein-1 (show CCL2 ELISA Kits) and CRP (show CRP ELISA Kits)
This gene encodes coagulation factor III which is a cell surface glycoprotein. This factor enables cells to initiate the blood coagulation cascades, and it functions as the high-affinity receptor for the coagulation factor VII. The resulting complex provides a catalytic event that is responsible for initiation of the coagulation protease cascades by specific limited proteolysis. Unlike the other cofactors of these protease cascades, which circulate as nonfunctional precursors, this factor is a potent initiator that is fully functional when expressed on cell surfaces. There are 3 distinct domains of this factor: extracellular, transmembrane, and cytoplasmic. This protein is the only one in the coagulation pathway for which a congenital deficiency has not been described. Alternate splicing results in multiple transcript variants.
, coagulation factor III
, tissue factor
, brain tissue factor
, coagulation factor 3