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Human KISS1 ELISA Kit for Sandwich ELISA - ABIN418250
Özgen, Torun, Bayraktar-Tanyeri, Durmaz, Kılıç, Cesur: The relation of urinary bisphenol A with kisspeptin in girls diagnosed with central precocious puberty and premature thelarche. in Journal of pediatric endocrinology & metabolism : JPEM 2016
Show all 3 Pubmed References
Human KISS1 ELISA Kit for Sandwich ELISA - ABIN842975
Abac?, Çatl?, An?k, Küme, Çalan, Dündar, Böber: Significance of serum neurokinin B and kisspeptin levels in the differential diagnosis of premature thelarche and idiopathic central precocious puberty. in Peptides 2015
Mouse (Murine) KISS1 ELISA Kit for Competition ELISA - ABIN425747
Chen, Fu, Cui, Pan, Li, Zhang, Evans, Cui, Liu: LIM-homeodomain transcription factor Isl-1 mediates kisspeptin's effect on insulin secretion in mice. in Molecular endocrinology (Baltimore, Md.) 2014
Kisspeptin neuronal system plays an essential role as the gatekeeper of reproduction via stimulating GnRH secretion.(review)
kiss1 and ghrh3 double mutants retain reproductive capacity
results reveal the pattern of kisspeptin neurones and their connections with GnRH3 neurones in the brain, suggesting distinct mechanisms for Kiss1 and Kiss2 in regulating reproductive events in zebrafish.
These findings implicate that Kiss1 could modulate fear responses mediated by 5-HT1A and 5-HT2 receptors
These findings suggest that the autocrine-regulated habenular Kiss1 neurons indirectly regulate the serotonergic system in the raphe nuclei through the habenula-interpeduncular nucleus in the zebrafish.
Dada (show ADA ELISA Kits) show that kiss1-expressing neurons are only located in the habenular nucleus, while kiss2-expressing neurons are found in the dorsal and ventral hypothalamus.
The zebrafish kiss1 was expressed in brain, intestine, adipose tissue and testis.
Kiss1/Kiss1r (show KISS1R ELISA Kits) system may participate in puberty initiation in fish as well.
Kisspeptin-10 may contribute to accelerate the progression and instability of atheromatous plaques, leading to plaque rupture. The GPR54 (show KISS1R ELISA Kits) antagonist may be useful for prevention and treatment of atherosclerosis.
we conclude that KISS-1 can inhibit the proliferation of osteosarcoma in vitro by accelerating the processes of apoptosis and autophagy.
These findings suggest that infundibular kisspeptin neurons are sensitive to circulating sex steroid hormones throughout life and that the sex reversal observed in MTF transsexuals might reflect, at least partially, an atypical brain sexual differentiation.
Patients with ovarian epithelial cancer and low KISS1 mRNA expression had shorter survival time than those with high expression (P = 0.001).Preoperative KISS1 mRNA was a potential prognostic biomarker for EOC, and high preoperative KISS1 expression indicated a favorable prognosis.
Lower expression of KiSS1 was observed in metastatic Breast Cancer.
KAI1 (show CD82 ELISA Kits) and KISS1 are implicated in the pathogenesis and maintenance of endometriosis.
In human cumulus (show KISS1R ELISA Kits) GCs, kiss1r mRNA levels were positively correlated with age but not with BMI. There was no expression of kiss1 mRNA in either cumulus o (show KISS1R ELISA Kits)r mural GCs
Overexpression of KiSS-1 suppressed the invasiveness of CRC (show CALR ELISA Kits) cells, and the gene exerted its function by reducing the expression of MMP-9 (show MMP9 ELISA Kits) via blocking of tge (show TGM3 ELISA Kits) PI3K (show PIK3CA ELISA Kits)/Akt (show AKT1 ELISA Kits)/NF-kappaB (show NFKB1 ELISA Kits) pathway.
Data suggest that plasma kisspeptin and serum prolactin (show PRL ELISA Kits) levels may be involved in the physiopathology of breast enlargement in newborns.
KISS1 Low Expression Correlates with Colorectal Liver Metastasis.
loss of PGR (show PGR ELISA Kits) impairs kisspeptin secretory machinery and therefore that PGR (show PGR ELISA Kits) plays a critical role in regulating kisspeptin secretion.
data suggest the utility of the KISS1R (show KISS1R ELISA Kits) as a novel biomarker for multiple myeloma, capable of targeting both tumor cells and host cells of the tumor microenvironment
Neurons in the hypothalamus produce Kiss1 and can synchronize their activity and activate GnRH neurons thus coordinating reproduction and fertility.
Our data indicate the absence of the circadian input to Kiss1 in pregnancy, despite high gestational estradiol levels and normal clock gene expression, and may suggest a disruption of a kisspeptin-specific diurnal rhythm that operates in the nonpregnant state.
kiss1 and kiss1r (show KISS1R ELISA Kits) mRNA levels were significantly higher in old compared to reproductive-aged mice, and diet-induced obesity did not alter kiss1 or kiss1r (show KISS1R ELISA Kits) mRNA levels. Compared to young control mice, young MCP-1 (show CPT1B ELISA Kits) knockout mice had significantly lower ovarian kiss1 mRNA
In the anteroventral periventricular, ERalpha (show ESR1 ELISA Kits) is required but not ERbeta (show ESR2 ELISA Kits) or GPR30 (show GPER ELISA Kits), vs the arcuate nucleus Kiss-expressing cell lines that require GPR30 (show GPER ELISA Kits), and either ERalpha (show ESR1 ELISA Kits) and/or ERbeta (show ESR2 ELISA Kits).
Data show that estradiol has no effect on kisspeptin (Kiss1) mRNA expression in prepubertal Kiss1 cell-specific estrogen receptoralpha knockout (KERalphaKO) mice.
On D4 of pregnancy, the mouse uterus expresses a functional kisspeptin/KISS1R (show KISS1R ELISA Kits) signaling system.
The results suggest that the 5'-upstream region of Kiss1 locus functions as an enhancer for the arcuate nucleus Kiss1 gene expression in mice.
This gene is a metastasis suppressor gene that suppresses metastases of melanomas and breast carcinomas without affecting tumorigenicity. The encoded protein may inhibit chemotaxis and invasion and thereby attenuate metastasis in malignant melanomas. Studies suggest a putative role in the regulation of events downstream of cell-matrix adhesion, perhaps involving cytoskeletal reorganization. A protein product of this gene, kisspeptin, stimulates gonadotropin-releasing hormone (GnRH)-induced gonadotropin secretion and regulates the pubertal activation of GnRH nuerons. A polymorphism in the terminal exon of this mRNA results in two protein isoforms. An adenosine present at the polymorphic site represents the third position in a stop codon. When the adenosine is absent, a downstream stop codon is utilized and the encoded protein extends for an additional seven amino acid residues.
, kisspeptin 1
, malignant melanoma metastasis-suppressor
, metastasis-suppressor KiSS-1
, Kiss1 variant E1a-E2b-E3
, Kiss1 variant E1b-E2a-E3
, Kiss1 variant E1b-E2b-E3
, MLL septin-like fusion
, G protein-coupled receptor 54
, G-protein coupled receptor 54
, G-protein coupled receptor OT7T175
, kiSS-1 receptor
, kisspeptins receptor
, metastin receptor
, orphan G protein-coupled receptor 54