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Based on the ADEPT study data set, we report that airway mucosal expression of CCL26 was a robust discriminator of type 2 inflammation from healthy nonatopic subjects. Furthermore, airway mucosal CCL26 expression was best identified by using a panel of clinical biomarkers, including Feno values, bEOS counts, and expression of 2 novel markers, sCCL17 and sCCL26
hypoxia-inducible factors activate the transcription of chemokine (show CCL1 ELISA Kits) ligand 26 in cancer cells to recruit chemokine receptor 1 (show CMKLR1 ELISA Kits)-expressing myeloid-derived suppressor cells to the primary tumor
this study shows that expression of CCL26 is increased in eosinophilic myocarditis patients compared to chronic lymphocytic myocarditis patients
The reduction of circulating levels of MCP-4 (show CCL13 ELISA Kits), eotaxin-3, and MIP-1beta (show CCL4 ELISA Kits) could be one of the mechanisms by which bariatric surgery contributes to the reduction of cardiovascular risk in these patients.
Review: eotaxins (CCL11 (show CCL11 ELISA Kits), CCL24 (show CCL24 ELISA Kits), and CCL26) play key role(s) during symptomatic inflammatory responses raised in response to allergic crisis of allergic asthma and atopic dermatitis
Data show that the interleukins IL-4 (show IL4 ELISA Kits) and IL-13 (show IL13 ELISA Kits) are critical factors for the induction of eotaxin-3/CCL26 in the pancreas.
These results show a relation between CCL26 production by IL-13 (show IL13 ELISA Kits)-stimulated bronchial epithelial cells , sputum eosinophil counts, and asthma severity. They also suggest a role for CCL26 in the sustained inflammation observed in patients with severe eosinophilic asthma and reveal CCL26 as a potential target for treating patients with eosinophilic asthma that are refractory
The most potent inhibition of RANTES (show CCL5 ELISA Kits), eotaxin (show CCL11 ELISA Kits) and eotaxin3 as well as MMP9 (show MMP9 ELISA Kits) activity.
These findings indicate that eotaxin-3 is expressed in Chronic subdural hematoma fluid
these data suggest that CCL26 and CCL24 (show CCL24 ELISA Kits) are likely involved in the pathogenesis of chronic nasal hypereosinophilia, with a complex cooperation and different involvement of the various members of eotaxin (show CCL11 ELISA Kits) family.
Data indicate that puerarin attenuates airway inflammation via eotaxin-3.
Distinct acidic and basic residues within CCR3 (show CCR3 ELISA Kits) determine both receptor expression and activation by the eotaxins.
This gene is one of two Cys-Cys (CC) cytokine genes clustered on the q arm of chromosome 7. Cytokines are a family of secreted proteins involved in immunoregulatory and inflammatory processes. The CC cytokines are proteins characterized by two adjacent cysteines. The cytokine encoded by this gene displays chemotactic activity for normal peripheral blood eosinophils and basophils. The product of this gene is one of three related chemokines that specifically activate chemokine receptor CCR3. This chemokine may contribute to the eosinophil accumulation in atopic diseases.
chemokine (C-C motif) ligand 26
, C-C motif chemokine 26
, CC chemokine IMAC
, chemokine N1
, macrophage inflammatory protein 4-alpha
, small inducible cytokine A26
, small inducible cytokine subfamily A (Cys-Cys), member 26
, small-inducible cytokine A26
, thymic stroma chemokine-1
, chemokine (C-C motif) ligand 26-like
, eotaxin 3
, small chemokine ligand 26