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Levels of protein carbonyls were moderately higher in liver tissue of APOE4 vs. APOE3 mice. Other biomarkers of oxidative stress were similar between the two genotypes. Under basal conditions, the stress-response pathways investigated appeared largely unaffected by the APOE (show APOE ELISA Kits) genotype
Mid-life socioeconomic status was positively associated with cognitive status among both Baby Boomers and pre-Boomers, even after adjusting for APOE (show APOE ELISA Kits) polymorphism.
Influence of APOE varepsilon4 genotype on phospholipid profiles associated with mTBI and/or PTSD diagnostic categories.
Rather, we hypothesize that PA may protect APOE (show APOE ELISA Kits)-epsilon4 allele carriers from selective neurodegeneration of individual fiber populations at locations of crossing fibers within projection and association WM fiber tracts.
ApoE (show APOE ELISA Kits) epsilon2 as a risk factor in FTD (show FTL ELISA Kits)-3 and epsilon4 plays a protective role in the disease.
The data indicate that the APOE (show APOE ELISA Kits) rs405509 T homozygote modulates the effect of APOE (show APOE ELISA Kits) epsilon4 on both cognitive performance and brain gray matter structure
ApoE2 and apoE4 increase the risk for heart disease; ApoE4 also increases the risk for neurodegenerative diseases. (Review)
in a population with low levels of cerebrovascular disease and a lower prevalence of SNAP than the general population, APOE (show APOE ELISA Kits) and known genetic drivers of AD do not appear to contribute to the neurodegeneration observed in SNAP
Data conclude that the ER-stress mediated reduction in apoA-I (show APOA1 ELISA Kits) transcription could be partly mediated via the inhibition of PPARalpha (show PPARA ELISA Kits) mRNA expression and activity. In addition, BET inhibition increased apoA-I (show APOA1 ELISA Kits) transcription, even if PPARalpha (show PPARA ELISA Kits) production and activity were decreased. Both BET inhibition and PPARalpha (show PPARA ELISA Kits) activation ameliorate the apoA-I (show APOA1 ELISA Kits) lowering effect of ER-stress and are therefore interesting targets to elev...
Elevated cholesterol and APOE (show APOE ELISA Kits) 4 genotype are independent risk factors for cognitive decline in ART-adherent HIV(+) men aged >50 years. Treatment of dyslipidemia may be an effective strategy to reduce cognitive decline in older HIV(+) individuals.
Chylomicron remnants and very low density lipoprotein (VLDL) remnants are rapidly removed from the circulation by receptor-mediated endocytosis in the liver. Apolipoprotein E, a main apoprotein of the chylomicron, binds to a specific receptor on liver cells and peripheral cells. ApoE is essential for the normal catabolism of triglyceride-rich lipoprotein constituents. The APOE gene is mapped to chromosome 19 in a cluster with APOC1 and APOC2. Defects in apolipoprotein E result in familial dysbetalipoproteinemia, or type III hyperlipoproteinemia (HLP III), in which increased plasma cholesterol and triglycerides are the consequence of impaired clearance of chylomicron and VLDL remnants.
, apolipoprotein E3