Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species
This study reveled that DRD5 are up regulation in CD4 (show CD4 ELISA Kits)+ T effector and regulatory cells in patient with multiple sclerosis.
DRD5 gene expression reduction in breast cancer patients after spiritual intervention
Conserved residues in intracellular loop 1 and transmembrane region 2 of DRD1 (show DRD1 ELISA Kits) and DRD5 are essential in ligand binding and signal transduction.
D1R and D5R colocalize in renal proximal tubule cells and physically interact in second messenger coupling pathways and heterologous receptor interaction between the two receptors.
This study shown DRD5 to be the risk factor for attention deficit/hyperactivity disorder.
LRs are essential not only for the proper membrane distribution and maintenance of AC5/6 activity but also for the regulation of D1R- and D5R-mediated AC signaling.
We found significant negative correlations regarding the expression of the genes COMT, MAOB, DRD4, DRD5 and FOS, indicating that increased schizotypy coincides with higher levels of dopaminergic dysregulation on the mRNA-level.
This study demonistrated that Lymphocyte DR D5 is reduced in MS and IFN-beta (show IFNB1 ELISA Kits) restores their expression and responsiveness.
Constitutive D5R signalling up-regulated expression of Na,K-ATPase (show ATP1A1 ELISA Kits)-alpha2 and NHE-2 (show SLC9A2 ELISA Kits), increasing glucose metabolism. Agonist treatment increased this and also upregulated NHE-3 (show SLC9A3 ELISA Kits).
Our find-ings suggest that common genetic variations of DRD5 are likely to con-tribute to genetic susceptibility to paranoid schizophrenia in Han Chinese
Findings demonstrate the relevant contribution of dopamine receptor D5 (D5R) in memory and suggest a functional interaction of D5R with hippocampal glutamatergic pathways.
Data provides evidence that forebrain D5R activation plays a unique role in spatial learning and memory in conjunction with D1R (show DRD1 ELISA Kits) activation
CCKBR and D5R synergistically interact in the kidney, which may contribute to the maintenance of normal sodium balance following an increase in sodium intake
We suggest that the ability of the dopamine D5 receptor to negatively regulate the renal NADPH oxidase (show NOX4 ELISA Kits) activity and AT1R (show AGTRAP ELISA Kits) function may have important implications in the pathogenesis of salt-sensitive blood pressure.
This study demonstrated that D5R stimulation contributes to an efficient CD4 (show CD4 ELISA Kits)(+)T-cell-induced early ERK1/2 (show MAPK1/3 ELISA Kits) phosphorylation, and differentiation.
The renal D5R protein upregulation was not caused by increased transcription because renal mRNA expression of D5R was similar in D3(-/-) and D3(+/+) mice.
This study demonistrated that the dopamine D5 receptor as a regulator of BDNF (show BDNF ELISA Kits) and Akt (show AKT1 ELISA Kits) signalling in rodent prefrontal cortex.
SNX1 (show SNX1 ELISA Kits) has a crucial role in D(5)R trafficking and SNX1 (show SNX1 ELISA Kits) depletion results in D(5)R dysfunction and thus may represent a novel mechanism for the pathogenesis of essential hypertension
By contributing to CD4(+) T cell activation and differentiation to Th17 phenotype, D5R expressed on DCs is able to modulate the development of an autoimmune response in vivo.
We propose that ciliary DR5 (show TNFRSF10B ELISA Kits) has functional chemo- and mechano-sensory roles in endothelial cells.
There were significant differences in the allelic frequencies among Bos taurus and Bos indicus breeds of different temperament for the DRD1 (show DRD1 ELISA Kits), DRD4 (show DRD4 ELISA Kits), and DRD5 markers.
these data provide evidence that functional D1/D5 receptors are expressed in the internal globus pallidus and the substantia nigra pars (show EPRS ELISA Kits) reticulata in both normal and parkinsonian states in monkeys
In the basolateral amygdala, the D1R (show DRD1 ELISA Kits) subtypes colocalize in dendritic spines and terminals, with D(5) predominant in terminals. There were similarities between the primates and rats, such as more prominent D(5) localization to presynaptic structures.
This gene encodes the D5 subtype of the dopamine receptor. The D5 subtype is a G-protein coupled receptor which stimulates adenylyl cyclase. This receptor is expressed in neurons in the limbic regions of the brain. It has a 10-fold higher affinity for dopamine than the D1 subtype. Pseudogenes related to this gene reside on chromosomes 1 and 2.
dopamine receptor D5
, D(1B) dopamine receptor-like
, D(1B) dopamine receptor
, D1beta dopamine receptor
, d(5) dopamine receptor
, dopamine D5 receptor
, dopamine receptor D1B
, dopamine receptor 5
, D(5) dopamine receptor
, D1b dopamine receptor
, D5 dopamine receptor
, dopamine D1B receptor