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Resistance induced by Paenibacillus alvei K165 depends on salicylate and jasmonate-dependent defense pathways, as evidenced by an increased transient accumulation of PR1 and PDF1.2 transcripts in the aerial parts of infected plants treated with K165.
Di19, a new type of transcription factor, directly up-regulates the expressions of PR1, PR2, and PR5 in response to drought stress.
Dtta indicate that the expression of marker genes PR1 and BGL2 for the salicylic acid (SA) and the PDF1.2 for the jasmonic acid-ethylene (JA-ET) signalling pathways was enhanced in 10 muM Cd-treated non-infected plants.
It is reported that salicylic acid (SA)-treated tir1 afb2 mutant shows enhanced transcript level of a pathogenesis related gene, PR1.
Natural variation in salicylic acid-induced PR-1 expression is associated with basal resistance against the hemi-biotrophic pathogen Pst (show SULT1A1 Proteins) DC3000.
In the absence of LS5 and LS7, NPR1 activates the PR-1 promoter through a mechanism that requires LS4
Lipooligosaccharide and the lipid A and core oligosaccharides derived from it were all able to induce the defense-related gene PR1 in Arabidopsis and to prevent the hypersensitive response caused by avirulent bacteria.
The transcript level of PR1, and PR2 were increased in ascorbate-deficient mutants.
The cloning and characterization of pr1 completes the molecular identification of all genes encoding structural enzymes of the anthocyanin pathway of maize.
Partially responsible for acquired pathogen resistance.
, pathogenesis related protein-1
, voltage-dependent calcium channel gamma-like subunit
, transmembrane protein 37
, neuronal voltage-gated calcium channel gamma-like subunit
, voltage-dependent calcium channel gamma subunit-like protein
, protein distantly related to to the gamma subunit family