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Human MET ELISA Kit for Sandwich ELISA - ABIN2684958
Edelmann, Fahrner, Malinka, Song, Stroka, Mermod: Nuclear Factor I-C acts as a regulator of hepatocyte proliferation at the onset of liver regeneration. in Liver international : official journal of the International Association for the Study of the Liver 2015
Which control critical events for colonization such as HGF (show HGF ELISA Kits)/Met axis and Wwox (show WWOX ELISA Kits), as therapy of bone metastasis.
Data show that by reducing CD82, KSHV miR-K6-5p expedites cell invasion and angiogenesis by activating the c-Met pathway.
These results suggest MET overexpression is related to altered c-CBL (show CBL ELISA Kits) expression in head and neck squamous cell carcinoma, which may influence tumorigenesis
Authors characterized exosomes from GBM cells harbouring and not harbouring PTPRZ1 (show PTPRZ1 ELISA Kits)-MET fusion (ZM fusion).
Data demonstrated that c-Met activation increases miR (show MLXIP ELISA Kits)-130b levels, inhibits androgen receptor (show AR ELISA Kits) expression, promotes cancer spreading and resistance to hormone ablation therapy.
The role of the EGFR (show EGFR ELISA Kits) and Met interaction in glioblastoma.The role of MET in the glioblastoma resistance to treatment. [review]
NCTD suppressed not only the expression of the total EGFR and the phosphorylated EGFR but also the expression of the total c-Met.
Altered Met receptor phosphorylation and LRP1 (show LRP1 ELISA Kits)-mediated uptake in cells lacking carbohydrate-dependent lysosomal targeting
Dual MET and SMO Inhibitors are potent antiproliferative agents in EGFR (show EGFR ELISA Kits)-Tyrosine kinase (show TXK ELISA Kits) inhibitors resistant human non small cell lung cancer.
although the juxtamembrane mutations of MET do not affect its known proteolytic cleavages, the R970C MET variant favors calpain dependent proteolytic cleavage in lung cancer cells.
Results demonstrate a new mechanism for the modulation of synapse formation, whereby MET activation induces an alignment of presynaptic and postsynaptic elements that are necessary for assembly and formation of functional synapses by subsets of neocortical neurons that express MET/beta-catenin (show CTNNB1 ELISA Kits) complex.
MET signaling regulates intestinal homeostasis and regeneration, as well as adenoma formation. These activities of MET are promoted by the stem cell CD44 (show CD44 ELISA Kits) isoform CD44v4-10.
a c-Met/ETS-1 (show ETS1 ELISA Kits)/matrix metalloproteinase-14 (MMP-14 (show MMP14 ELISA Kits)) axis that controls VE-cadherin (show CDH5 ELISA Kits) degradation, endothelial mesenchymal transition, and vascular abnormality.
We found the roles of hepatocyte growth factor (HGF (show HGF ELISA Kits)) signaling in stria vascularis development for the first time and that lack of HGF (show HGF ELISA Kits) signaling in the inner ear leads to profound hearing loss in the mouse. Our findings reveal a novel mechanism that may underlie human deafness DFNB39 (show HGF ELISA Kits) and DFNB97. Our findings reveal an additional example of context-dependent c-MET signaling diversity, required here for proper cellular inva
c-Met has been found to attenuate lung injury and apoptosis in bronchial epithelial cells.
In this paper, we demonstrate a potential biochemical mechanism for such phenomena by showing that c-Met receptors promote the tyrosine phosphorylation of RalGDS (show RALGDS ELISA Kits) at Y752 in its RBD (show CACNA1D ELISA Kits), which blocks the binding of Ras to RalGDS (show RALGDS ELISA Kits).
study demonstrates that co-activation of AKT (show AKT1 ELISA Kits) and c-Met induces hepatocellular carcinoma development that depends on the mTORC1/FASN (show FASN ELISA Kits) pathway.
Molecular and functional studies revealed that ectopic Met expression in limb mesenchyme does not alter HGF (show HGF ELISA Kits) expression patterns and levels, but impairs HGF (show HGF ELISA Kits) bioavailability
miR-206 suppressed c-Met expression in gastric cancer and could function as a potent tumor suppressor in c-Met overexpressing tumors.
Meg3 overexpression in insulinoma cells down-regulated the expression of the protooncogene c-met.
possible cooperative role of the EGF (show EGF ELISA Kits) and HGF (show HGF ELISA Kits) pathways and indicate that cross-talk between their respective receptors may modulate mammary gland development in the cow
The proto-oncogene MET product is the hepatocyte growth factor receptor and encodes tyrosine-kinase activity. The primary single chain precursor protein is post-translationally cleaved to produce the alpha and beta subunits, which are disulfide linked to form the mature receptor. Various mutations in the MET gene are associated with papillary renal carcinoma. Two transcript variants encoding different isoforms have been found for this gene.
, HGF/SF receptor
, SF receptor
, hepatocyte growth factor receptor
, met proto-oncogene tyrosine kinase
, proto-oncogene c-Met
, scatter factor receptor
, tyrosine-protein kinase Met
, HGF receptor c-Met