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Human MET ELISA Kit for Sandwich ELISA - ABIN1672788
Bin, Ma, Xu, Shi: Embryonic hepatocyte transplantation for hepatic cirrhosis: efficacy and mechanism of action. in World journal of gastroenterology : WJG 2012
Show all 3 references for ABIN1672788
our data provide evidence for the critical involvement of c-Met signaling in cholesterol and bile acids toxicity
Data show that genes coding for epidermal growth factor receptor (EGFR) and hepatocyte growth factor receptor (c-MET) were directly downregulated by both microRNAs miR-1 and miR-206.
Study provides evidence that AXL (show AXL ELISA Kits) and MET are associated with cell proliferation and metastasis in lung cancer, and the crosstalk between these receptors affects tumor progression.
overexpression of HGF resulted in resistance to c-MET tyrosine kinase inhibitors through an autocrine manner in gastric cancer cells
CMET overexpression and CMET amplification are commonly found in non-small cell lung cancer (NSCLC) brain metastases and may represent a promising therapeutic target.
Study shows that MET was directly regulated by miR (show MLXIP ELISA Kits)-23b and miR (show MLXIP ELISA Kits)-27b. Moreover, downregulating the MET gene by use of siRNA significantly inhibited cell migration and invasion by oral squamous cell carcinoma cells.
Activation of the HGF (show HGF ELISA Kits)-mediated MET pathway is involved in escape to selective VEGFR (show KDR ELISA Kits) inhibition in neuroblastoma (show ARHGEF16 ELISA Kits) suggesting combined inhibition of MET and VEGFR (show KDR ELISA Kits) signaling to reduce secondary resistance and enhanced invasiveness.
miR (show MLXIP ELISA Kits)-27b was frequently downregulated in NSCLC tissues and cell lines and could inhibit the malignant phenotypes of NSCLC cells by directly targeting the MET oncogene (show RAB1A ELISA Kits). The identification of miR (show MLXIP ELISA Kits)-27b-mediated novel signaling pathways may help reveal the molecular mechanism underlying the development and malignant progression of this disease.
Therefore, our findings suggested miR (show MLXIP ELISA Kits)-34a could modulate human gastric cancer(GC) cell Cisplatin sensitivity by regulation of cell proliferation and apoptosis via targeting MET, potentially benefiting human GC treatment in the future.
miR1 (show FSD1 ELISA Kits) expression inversely correlated with MET, cyclin D1 (show CCND1 ELISA Kits) and CDK4 (show CDK4 ELISA Kits) expression in esophageal squamous cell carcinoma cells.
study demonstrates that co-activation of AKT (show AKT1 ELISA Kits) and c-Met induces hepatocellular carcinoma development that depends on the mTORC1/FASN (show FASN ELISA Kits) pathway.
Molecular and functional studies revealed that ectopic Met expression in limb mesenchyme does not alter HGF (show HGF ELISA Kits) expression patterns and levels, but impairs HGF (show HGF ELISA Kits) bioavailability
miR-206 suppressed c-Met expression in gastric cancer and could function as a potent tumor suppressor in c-Met overexpressing tumors.
Meg3 overexpression in insulinoma cells down-regulated the expression of the protooncogene c-met.
our results support the concept of the acrosome as a lysosome-related organelle and provide evidence for the identification of MET as a tyrosine kinase receptor (show KDR ELISA Kits) that may play a role in fertilization.
We show that a subset of adult calcitonin gene-related peptide (CGRP (show CALCA ELISA Kits))-expressing myenteric neurons produce MET, the receptor for hepatocyte growth factor (show HGF ELISA Kits).
HGF and c-met inhibition by means of a novel monoclonal neutralizing antibody inhibits tumor growth in prostate cancer cells and in mouse xenografts.
facilitates T cell recruitment to the heart via CCR5 (show CCR5 ELISA Kits) receptor by inducing autocrine CCR5 (show CCR5 ELISA Kits) ligand release
C-Met pathway is suggested to contribute to the lymphomagenesis in the MALT lymphoma after H. heilmannii infection.
c-Met targeting enhances the effect of irradiation and chemical agents against malignant colon cells harboring a KRAS mutation.
possible cooperative role of the EGF (show EGF ELISA Kits) and HGF (show HGF ELISA Kits) pathways and indicate that cross-talk between their respective receptors may modulate mammary gland development in the cow
The proto-oncogene MET product is the hepatocyte growth factor receptor and encodes tyrosine-kinase activity. The primary single chain precursor protein is post-translationally cleaved to produce the alpha and beta subunits, which are disulfide linked to form the mature receptor. Various mutations in the MET gene are associated with papillary renal carcinoma. Two transcript variants encoding different isoforms have been found for this gene.
, HGF/SF receptor
, SF receptor
, hepatocyte growth factor receptor
, met proto-oncogene tyrosine kinase
, proto-oncogene c-Met
, scatter factor receptor
, tyrosine-protein kinase Met
, HGF receptor c-Met