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Human MET ELISA Kit for Sandwich ELISA - ABIN1672788
Bin, Ma, Xu, Shi: Embryonic hepatocyte transplantation for hepatic cirrhosis: efficacy and mechanism of action. in World journal of gastroenterology : WJG 2012
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Mouse (Murine) MET ELISA Kit for Sandwich ELISA - ABIN1672789
Yin, Liu, Li, Liu, Xie, Lv, Gao, Guang: The cyclooxygenase-2 inhibitor celecoxib attenuates hepatocellular carcinoma growth and c-Met expression in an orthotopic mouse model. in Oncology research 2011
Show all 3 references for ABIN1672789
Data indicate that oncogene (show RAB1A ELISA Kits) c-Met was a putative target of microRNA miR (show MLXIP ELISA Kits)-139-5p.
Study identified the G-quadruplex structure in MET promoter and showed its importance in binding to the Sp1 (show PSG1 ELISA Kits) activator promoting tumor growth.
Data show that silencing of fibroblast growth factor receptors FGFR1 or FGFR2 overcomes resistance to the proto-oncogene proteins c-met (MET) inhibitor.
Data show that inhibition of ATP binding cassette subfamily B member 1 (ABCB1) might be a novel therapeutic strategy for non-small cell lung cancer (NSCLC) patients with acquired resistance to proto-oncogene proteins c-met (MET) inhibitors.
Results indicate that proto-oncogene protein c-MET (MET) expression was significantly associated with MET amplification.
Report the application of advanced quantitative fluorescence microscopy techniques to study changes in the oligomerization state of the RTK Met in response to stimulation by its endogenous ligand hepatocyte growth factor (show HGF ELISA Kits).
Results show that overexpression of c-MET and its phosphorylation with up-regulation of VEGFR1 (show FLT1 ELISA Kits) are associated with chemo-resistance phenotype of colon cancer.
High c-Met expression is associated with small cell hypercalcemic ovarian carcinoma.
The findings suggest that the oncogenic activity of hepsin (show HPN ELISA Kits) arises not only from elevated expression level but also from depletion of HAI-1 (show SPINT1 ELISA Kits), events which together trigger gain-of-function activity impacting HGF (show HGF ELISA Kits)/MET signalling and epithelial cohesion
analysis of ARID1A (show ARID1A ELISA Kits), CDH1 (show CDH1 ELISA Kits), cMET and PIK3CA (show PIK3CA ELISA Kits) expression and target-related microRNA expression in gastric cancer
Molecular and functional studies revealed that ectopic Met expression in limb mesenchyme does not alter HGF (show HGF ELISA Kits) expression patterns and levels, but impairs HGF (show HGF ELISA Kits) bioavailability
miR-206 suppressed c-Met expression in gastric cancer and could function as a potent tumor suppressor in c-Met overexpressing tumors.
Meg3 overexpression in insulinoma cells down-regulated the expression of the protooncogene c-met.
our results support the concept of the acrosome as a lysosome-related organelle and provide evidence for the identification of MET as a tyrosine kinase receptor (show KDR ELISA Kits) that may play a role in fertilization.
We show that a subset of adult calcitonin gene-related peptide (CGRP (show CALCA ELISA Kits))-expressing myenteric neurons produce MET, the receptor for hepatocyte growth factor (show HGF ELISA Kits).
HGF and c-met inhibition by means of a novel monoclonal neutralizing antibody inhibits tumor growth in prostate cancer cells and in mouse xenografts.
facilitates T cell recruitment to the heart via CCR5 (show CCR5 ELISA Kits) receptor by inducing autocrine CCR5 (show CCR5 ELISA Kits) ligand release
C-Met pathway is suggested to contribute to the lymphomagenesis in the MALT lymphoma after H. heilmannii infection.
c-Met targeting enhances the effect of irradiation and chemical agents against malignant colon cells harboring a KRAS mutation.
HGFR activation is regulated by integrin beta-1 (show ITGB1 ELISA Kits). Decreased Itgb1 (show ITGB1 ELISA Kits) leads to decreased HGFR activation.
possible cooperative role of the EGF (show EGF ELISA Kits) and HGF (show HGF ELISA Kits) pathways and indicate that cross-talk between their respective receptors may modulate mammary gland development in the cow
The proto-oncogene MET product is the hepatocyte growth factor receptor and encodes tyrosine-kinase activity. The primary single chain precursor protein is post-translationally cleaved to produce the alpha and beta subunits, which are disulfide linked to form the mature receptor. Various mutations in the MET gene are associated with papillary renal carcinoma. Two transcript variants encoding different isoforms have been found for this gene.
, HGF/SF receptor
, SF receptor
, hepatocyte growth factor receptor
, met proto-oncogene tyrosine kinase
, proto-oncogene c-Met
, scatter factor receptor
, tyrosine-protein kinase Met
, HGF receptor c-Met