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anti-Rat (Rattus) GABRA4 Antibodies:
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Rat (Rattus) Polyclonal GABRA4 Primary Antibody for WB - ABIN361441
Diaz, Christian, Anderson, McCool: Chronic ethanol and withdrawal differentially modulate lateral/basolateral amygdala paracapsular and local GABAergic synapses. in The Journal of pharmacology and experimental therapeutics 2011
Show all 8 Pubmed References
Rat (Rattus) Polyclonal GABRA4 Primary Antibody for WB - ABIN152534
Pignataro, Miller, Ma, Midha, Protiva, Herrera, Harrison: Alcohol regulates gene expression in neurons via activation of heat shock factor 1. in The Journal of neuroscience : the official journal of the Society for Neuroscience 2007
Show all 2 Pubmed References
Rat (Rattus) Polyclonal GABRA4 Primary Antibody for WB - ABIN361442
Marowsky, Rudolph, Fritschy, Arand: Tonic inhibition in principal cells of the amygdala: a central role for ?3 subunit-containing GABAA receptors. in The Journal of neuroscience : the official journal of the Society for Neuroscience 2012
Show all 8 Pubmed References
Human Polyclonal GABRA4 Primary Antibody for IHC (p), IHC - ABIN451648
Ma, Whitehead, Menold, Martin, Ashley-Koch, Mei, Ritchie, Delong, Abramson, Wright, Cuccaro, Hussman, Gilbert, Pericak-Vance: Identification of significant association and gene-gene interaction of GABA receptor subunit genes in autism. in American journal of human genetics 2005
GABRA4 is associated with autism spectrum disorder in a dataset from Argentina.
Transgenic mice with the alpha4betadelta GABAA (show GABRg1 Antibodies) receptor subunit deletion show increased conditioned place preference, but not from delta-deficient or alpha4-deficient knockout mice.
The pathway by which GABRA2 (show GABRA2 Antibodies) initially confers risk for eventual alcohol problems begins with a predisposition to sensation-seeking early in adolescence.
Association between premenstrual symptom severity and two genes from the gamma-aminobutyric acid (GABA) pathway: steroid-5-alpha-reductase, alpha polypeptide (show ITGAE Antibodies) 1 (show CYP Antibodies) (SRD5A1 (show SRD5A1 Antibodies)) and gamma-aminobutyric acid receptor subunit alpha-4 (GABRA4).
single nucleotide polymorphisms studied in the GABRA4, GABRE (show GABRE Antibodies), and GABRQ (show GABRQ Antibodies) genes are not related to the risk for familial ET.
GABRA4 is involved in the etiology of autism and potentially increases autism risk through interaction with GABRB1 (show GABRB1 Antibodies).
These results confirmed our earlier findings, indicating GABRA4 and GABRB1 (show GABRB1 Antibodies) as genes contributing to autism susceptibility, extending the effect to multiple ethnic groups and suggesting seizures as a stratifying phenotype.
Lower delta mRNA levels in schizophrenia might reflect a reduced number of alpha(1)beta(x)delta (show DLL1 Antibodies) GABA(A) receptors that could contribute to deficient tonic inhibition and prefrontal cortical dysfunction in schizophrenia.
Gabra4 KO CD4 (show CD4 Antibodies)(+) cells produced increased cytokines.
This study addressed the effects of AW on changes in the expression of Gabra4 and related genes that encode other subunits of GABAARs
GABAA (show GABRg1 Antibodies) receptors in the mediodorsal thalamic nucleus (MD) modulate fear extinction.
Increase in GABRA4 activity promotes hippocampal neurogenesis via nuclear factor of activated T cell (show NFATC3 Antibodies) (NFATc)4 (show NFATC4 Antibodies).
This study demonistrated that alpha4-GABAARs contribute to nRT burst-mediated inhibition and that activity-dependent recruitment of eGABAARs dynamically influences the ventrobasal neuron tonic conductance during nRT tonic spike trains.
Data suggest that the availability of distinct GABA(A) R subtypes (Gabra2, -4, and Gabrd) provides a molecular mechanism endowing spatiotemporal specificity to GABAergic control of neuronal maturation in adult brain.
alpha-4 subunit of the GABAA (show GABRg1 Antibodies) receptor is transcriptionally regulated
Gamma-aminobutyric acid (GABA) is the major inhibitory neurotransmitter in the mammalian brain where it acts at GABA-A receptors, which are ligand-gated chloride channels. Chloride conductance of these channels can be modulated by agents such as benzodiazepines that bind to the GABA-A receptor. At least 16 distinct subunits of GABA-A receptors have been identified. This gene encodes subunit alpha-4, which is involved in the etiology of autism and eventually increases autism risk through interaction with another subunit, gamma-aminobutyric acid receptor beta-1 (GABRB1). Alternatively spliced transcript variants encoding different isoforms have been found in this gene.
gamma-aminobutyric acid (GABA) A receptor, alpha 4
, gamma-aminobutyric acid A receptor, alpha 4
, GABA(A) receptor subunit alpha-4
, gamma-aminobutyric acid receptor subunit alpha-4-like
, GABA-A receptor alpha-4 subunit
, gamma-aminobutyric acid (GABA-A) receptor, subunit alpha 4
, gamma-aminobutyric acid receptor subunit alpha-4
, GABA(A) receptor, alpha 4