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Human IL17A ELISA Kit for Sandwich ELISA - ABIN411314
Tan, Liu, Yang, Wang, Qin: An inactivated Pseudomonas aeruginosa medicament inhibits airway allergic inflammation and improves epithelial functions. in The journal of physiological sciences : JPS 2012
Show all 13 Pubmed References
Mouse (Murine) IL17A ELISA Kit for Sandwich ELISA - ABIN411315
Kotake, Udagawa, Takahashi, Matsuzaki, Itoh, Ishiyama, Saito, Inoue, Kamatani, Gillespie, Martin, Suda: IL-17 in synovial fluids from patients with rheumatoid arthritis is a potent stimulator of osteoclastogenesis. in The Journal of clinical investigation 1999
Show all 11 Pubmed References
Mouse (Murine) IL17A ELISA Kit for Sandwich ELISA - ABIN625138
Nakai, Yoneda, Hosokawa, Moriue, Presland, Fallon, Kabashima, Kosaka, Kubota: Reduced expression of epidermal growth factor receptor, E-cadherin, and occludin in the skin of flaky tail mice is due to filaggrin and loricrin deficiencies. in The American journal of pathology 2012
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Human IL17A ELISA Kit for Sandwich ELISA - ABIN365737
Deng, Chen, Zheng, Chen, Huang, Yin, Gao, Lin, Huang, Gao, Yu, Liu: Association of BAFF and IL-17A with subphenotypes of primary Sjögren's syndrome. in International journal of rheumatic diseases 2015
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Human IL17A ELISA Kit for Sandwich ELISA - ABIN1446085
Lu, Nossent: Thrombopoietin levels in systemic lupus erythematosus are linked to inflammatory cytokines, but unrelated to thrombocytopenia or thrombosis. in Lupus 2014
Mouse (Murine) IL17A ELISA Kit for Sandwich ELISA - ABIN1446148
Rosillo, Sánchez-Hidalgo, Sánchez-Fidalgo, Aparicio-Soto, Villegas, Alarcón-de-la-Lastra: Dietary extra-virgin olive oil prevents inflammatory response and cartilage matrix degradation in murine collagen-induced arthritis. in European journal of nutrition 2015
Data indicate that IL-17/22-producing cells play an important role in maintenance of intestinal mucosa in gluten-sensitive primates.
IL17A and IL17F (show IL17F ELISA Kits) gene polymorphism are not the important factors associated with susceptibility and some clinical parameters of rheumatoid arthritis in a Polish population.
IL-17A may limit the extent of bone formation at inflamed periosteal sites in spondyloarthritis
These findings indicate that IL-17 directly acts on bronchial smooth muscle (BSM) cells and up-regulates RhoA protein probably via a down-regulation of miR-133a-3p, resulting in an induction of BSM hyperresponsiveness.
serum levels determined of both T cell polarizing (IL- 33 (show IL33 ELISA Kits) and IL-12 (show IL12A ELISA Kits)) and T cell effector (IFN-gamma (show IFNG ELISA Kits), IL-4 (show IL4 ELISA Kits), IL-10 (show IL10 ELISA Kits), IL-17 and IL-9 (show IL9 ELISA Kits)) cytokines in T1DM (type-1 Diabetes Mellitus) subjects with and without microvascular complications (MVC). All the tested cytokines were significantly elevated in T1DM subjects except for IFN-gamma (show IFNG ELISA Kits) with no significant difference between those with and without MVC.
IL-17 can induce A549 alveolar epithelial cells to undergo epithelial-mesenchymal transition via the TGF-beta1 (show TGFB1 ELISA Kits) mediated Smad2 (show SMAD2 ELISA Kits)/3 and ERK1/2 (show MAPK1/3 ELISA Kits) activation
Th17 cells and IL-17 increased autophagy of RA FLSs by causing mitochondrial dysfunction. Our findings suggest that, in RA, interactions between RA FLSs and Th17 cells may be involved in the tumorous growth of FLSs and the formation of pannus in joints
high IL-17 expression was an indicator of poor prognosis for Chinese patients with lung cancer
This review demonstrates an understanding on how the pro- or antitumor function of Th17 cells and IL-17 may change cancer progression, leading to the appearance of complex and pivotal biologic activities in tumor. [review]
An increase in neutrophils and an upregulation of interleukin-17 responses have been documented in periodontitis.
Rhinovirus inhibits IL-17A and the downstream immune responses in allergic asthma.
this study shows that local production of IL-17A by innate lymphoid cells is crucial for the development of intestinal IR injury
These data thus reveal that HDAC6 (show HDAC6 ELISA Kits) represses IL-17 production in T cells, providing novel insights into the role of HDAC6 (show HDAC6 ELISA Kits) in the immune system.
Data indicate that IL-2 (show IL2 ELISA Kits) can augment the gammadeltaT-17 response in favor of short-lived effectors with limited plasticity, particularly in the presence of IL-1beta (show IL1B ELISA Kits) and IL-23 (show IL23A ELISA Kits). In this way, IL-2 (show IL2 ELISA Kits) may act to curtail the innate-like response of gammadeltaT-17 cells upon arrival of IL-2 (show IL2 ELISA Kits)-producing adaptive immune cells at the site of inflammation.
These data support the conclusion that IL-17A contributes to middle ear injury through the p38 MAPK (show MAPK14 ELISA Kits) signaling pathway.
this study describes regulation of IL-17A expression in mice following subacute ozone exposure
impact of IL-6 on the Th9/Th17 balance depending on the predominant cytokine milieu; data suggest that the IL-6-mediated reduction of Th2-related IL-4 leads to a decline of the Th9 immune response and allows Th17 differentiation
these results identify a distinct pathway in which IL-17 production by splenic LSK (show LCK ELISA Kits)- cells supports their differentiation into B cells in response to acute Plasmodium infection
this study shows that an ocular commensal protects against corneal infection by driving an IL-17a response from mucosal TCR gamma delta T Cells
At week 15, skin cell infiltrates from pigs with crusted scabies had significantly higher CD8 (show CD8A ELISA Kits)+ T cell, gammadelta T cell and IL-17+ cell numbers than those with ordinary scabies. Peripheral IL-17 levels were not increased, suggesting that localized skin IL-17-secreting T cells may play a critical role in the pathogenesis of crusted scabies development.
Alterations in the IL-17 regulated pathway of porcine colon in response to dietary supplementation with seaweeds and yeast are reported.
conclude that only two subpopulations of porcine WBCs are sources of IL-17 after non-specific stimulation: CD3 (show CD3 ELISA Kits)(+)CD4 (show CD4 ELISA Kits)(+) and CD3 (show CD3 ELISA Kits)(+)gammadeltaTCR(+).
Cloning and expression of interleukin-17.
calves, like humans, mount a robust IL-17 response during respiratory syncytial virus infection.
Data show that CD4 (show CD4 ELISA Kits)(+) and WC1(+) gammadelta T-cells were induced to produce IL-17 termed Th17 and gammadelta17 cells.
IL-17A was shown to be upregulated, supporting its investigation as a potential biomarker of bTB.
IL-17A and IL-17F (show IL17F ELISA Kits) have a potential to modulate the mammary gland immune response to mastitis-causing pathogens.
The results of this study provide evidence of the role of IL-17 in the immunopathology of tuberculosis and support the use of IL-17 as a potential biomarker with predictive value of prognosis in bTB.
Data show increased interleukin 17 (IL-17) staining intensity in mediastinal lymph node from chronic recurrent airway obstruction (RAO (show AOC2 ELISA Kits)) horses.
cloning and characterization of interleukin-17 expressed gene sequence from mRNA obtained from intestinal tissue and interleukin-23 expressed gene sequence from mRNA obtained from peripheral blood mononuclear cells
This study examined effects of in vitro exposure to solutions of hay (show GTF2H5 ELISA Kits) dust, lipopolysaccharides, or beta-glucan on cytokine expression in pulmonary mononuclear cells isolated from healthy horses and horses with recurrent airway obstruction.
molecular cloning and expression of recombinant IL-17A; homology modeling revealed that the three-dimensional structure resembles that of human IL-17A. The secondary structure predicted showed the presence of one extra helix in the N-terminal region.
one of the interaction sites between IL17A and its receptor IL17RA (show IL17RA ELISA Kits)
The protein encoded by this gene is a proinflammatory cytokine produced by activated T cells. This cytokine regulates the activities of NF-kappaB and mitogen-activated protein kinases. This cytokine can stimulate the expression of IL6 and cyclooxygenase-2 (PTGS2/COX-2), as well as enhance the production of nitric oxide (NO). High levels of this cytokine are associated with several chronic inflammatory diseases including rheumatoid arthritis, psoriasis and multiple sclerosis.
, cytotoxic T-lymphocyte-associated antigen 8
, cytotoxic T-lymphocyte-associated protein 8
, interleukin 17 (cytotoxic T-lymphocyte-associated serine esterase 8)
, interleukin 17
, interleukin 17A
, Interleukin 17 (cytotoxic T-lymphocyte-associated serine esterase 8)