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Human Selectin E + CD62e ELISA Kit for Sandwich ELISA - ABIN624970
Ko, Kuo, Chang, Chen, Liu, Chen, Tsai, Lee, Chen, Wu, Chen: CXCL10/IP-10 is a biomarker and mediator for Kawasaki disease. in Circulation research 2015
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Rat (Rattus) Selectin E + CD62e ELISA Kit for Sandwich ELISA - ABIN416131
Shimazaki, Matsumoto, Ogura, Muroya, Kuwagata, Nakagawa, Yamakawa, Hosotsubo, Imamura, Shimazu: Systemic involvement of high-mobility group box 1 protein and therapeutic effect of anti-high-mobility group box 1 protein antibody in a rat model of crush injury. in Shock (Augusta, Ga.) 2012
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Human Selectin E + CD62e ELISA Kit for Sandwich ELISA - ABIN414367
Tu, Hu, Chen: Endothelial gene expression and molecular changes in response to radiosurgery in in vitro and in vivo models of cerebral arteriovenous malformations. in BioMed research international 2013
Human Selectin E + CD62e ELISA Kit for Sandwich ELISA - ABIN411350
Zhong, He, Yang, Bao, Zhang, Zhou, Chen, Li, Han, Yang, Sun, Cao, Li, Shi, Jiang, Zhang, Yan: L-selectin and P-selectin are novel biomarkers of cervicovaginal inflammation for preclinical mucosal safety assessment of anti-HIV-1 microbicide. in Antimicrobial agents and chemotherapy 2012
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High E-selectin expression is associated with breast cancer metastasis.
his study shows that two adhesion molecules, shed as soluble forms, are elevated during the acute phase of leptospirosis: E-selectin and s-ICAM1 (show ICAM1 ELISA Kits). These molecules may interfere with the process of immune cell recruitment to clear Leptospira at tissue levels.
ELAM-1 upregulation may promote disorder in adhesion, and migration of the cells leading to the endometriosis disorder.
Elevated endothelium-related mediators (vWF (show VWF ELISA Kits), E-selectin and EPCR (show PROCR ELISA Kits)) appear to participate in the development of pancreatic necrosis and may be a potential indicator of overall prognosis.
interplay between flow-dependent on-rate and off-rate of E-selectin-ligand bonds determine flow-enhanced cell rolling stability
High SELE expression is associated with Inflamed Atherosclerotic Plaque.
In this study, we addressed this question by analysing sLex expression together with two glycoproteins (BST-2 (show BST2 ELISA Kits) and LGALS3BP (show LGALS3BP ELISA Kits)), shown to interact with E-selectin in a carbohydrate-dependent manner, in a cohort of 249 invasive breast cancers
SELE expression is significantly upregulated in human masticatory mucosa during wound healing
E-selectin concentrations in this study were associated with hyperglycemia, possibly reflecting early endothelial damage. E-selectin was not useful to assess carotid intima-media thickness, a marker of subclinical atherosclerosis, which appeared to be determined by aging and male gender.
Plasma E-selectin is overexpressed in pediatric patients with mycoplasma pneumoniae pneumonia and myocardial injury.
Chitosan oligosaccharides downregulate the expression of E-selectin and ICAM-1 (show ICAM1 ELISA Kits) by inhibiting the phosphorylation of Mitogen-Activated Protein Kinases and the activation of NF-kappaB (show NFKB1 ELISA Kits) in lipopolysaccharides treated porcine iliac artery endothelial cells.
Increased E-selectin mRNA at end of 20 hours of cold ischemia time might indicate a preactivated state of endothelial cells potentially triggered by bacterial translocation or products.
The e-selectin/biliverdin reductase (show BLVRA ELISA Kits) pathway participates in the inhibitory process of hypoxia in pulmonary artery endothelial cell apoptosis which is mediated by the mitochondrial-dependent apoptosis pathway.
Five single nucleotide polymorphisms (SNPs) from different exons in selectin SELP (show SELP ELISA Kits), two in selectin SELL and one in selectin SELE were taken forward to genotyping in 337 Holstein Friesian heifers using PCR-RFLP.
Before the arrival of tumor cells, the lung-recruited monocytic myeloid-derived suppressor cells produced IL-1beta (show IL1B ELISA Kits), thereby increasing E-selectin expression and promoting tumor cell arrest on endothelial cells.
Inhibiting E-selectin attenuated inflammation in atherosclerotic plaques, likely by reducing leukocyte recruitment into plaques and by mitigating hematopoietic stem and progenitor cell activation in the spleen of mice with myocardial infarction.
In a model of corneal transplantation, P- and E-selectin mediate T cell recruitment to the graft, E-selectin mediates APC (show APC ELISA Kits) trafficking to lymphoid tissue, and blockade of E-selectin has a modest effect on improving long-term graft survival.
Social defeat induced an exposure-dependent increase in mRNA levels of E-selectin, CXCL1 (show CXCL1 ELISA Kits), and CXCL2 (show CXCL2 ELISA Kits) that increased with additional days of social defeat.
The histopathological modification of lupus nephritis by non-nephritogenic bystander IgM (show CD40LG ELISA Kits) antibodies is associated in part with glomerular E-selectin expression.
Data show that actin cytoskeleton regulated CD44 (show CD44 ELISA Kits) membrane mobility and interactions with E-selectin.
Regulatory T cells dynamically regulate P and E selectin ligand expression during multiple challenge contact hypersensitivity.
Systemic inflammation may increase the expression of E-selectin which mediated the lung metastasis of breast cancer in mouse model.
In the absence of either PSGL-1 (show SELPLG ELISA Kits) or E- and P-selectin (show SELP ELISA Kits).
Elevated E-selectin plays an important role in hepatic neutrophil infiltration and injury induced by chronic-binge feeding in mice and may also contribute to the pathogenesis of early stages of human alcoholic liver disease.
The protein encoded by this gene is found in cytokine-stimulated endothelial cells and is thought to be responsible for the accumulation of blood leukocytes at sites of inflammation by mediating the adhesion of cells to the vascular lining. It exhibits structural features such as the presence of lectin- and EGF-like domains followed by short consensus repeat (SCR) domains that contain 6 conserved cysteine residues. These proteins are part of the selectin family of cell adhesion molecules. Adhesion molecules participate in the interaction between leukocytes and the endothelium and appear to be involved in the pathogenesis of atherosclerosis.
, endothelial adhesion molecule 1
, selectin E (endothelial adhesion molecule 1)
, CD62 antigen-like family member E
, endothelial leukocyte adhesion molecule 1
, leukocyte endothelial cell adhesion molecule 2
, leukocyte-endothelial cell adhesion molecule 2
, selectin, endothelial cell