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sLZIP is a novel co-repressor of ERalpha (show ESR1 ELISA Kits), and plays a negative role in ERalpha (show ESR1 ELISA Kits)-mediated cell proliferation in breast cancer
These findings indicate that LZIP is a novel modulator of APOA4 (show APOA4 ELISA Kits) expression and hepatic lipid metabolism.
The authors found that the CREB3/Herp (show HERPUD1 ELISA Kits) pathway limited the increase in cytosolic Ca2 (show CA2 ELISA Kits)+ concentration and apoptosis early in poliovirus infection and this may reduce the extent of poliovirus-induced damage to the central nervous system during poliomyelitis.
The essential parts of the Golgi stress response from the perspective of the organelle autoregulation. The pathways of the mammalian Golgi stress response have been identified, specifically the CREB3 pathway.
These results indicate that sLZIP plays a role in expression of c-Jun (show JUN ELISA Kits), and migration and invasion of cervical cancer cells via regulation of MMP-9 (show MMP9 ELISA Kits) transcription.
INHA (show INHA ELISA Kits) gene expression is upregulated by cAMP via CRE in human trophoblasts, and TFAP2 (show TFAP2A ELISA Kits) regulates this expression by interacting with CRE.
Findings indicate that sLZIP negatively regulates AR transactivation in androgen-dependent PCa (show FLVCR1 ELISA Kits) cells and functions as a positive regulator in tumor progression of androgen-independent PCa (show FLVCR1 ELISA Kits). sLZIP contributes to the malignant phenotype of PCa (show FLVCR1 ELISA Kits).
A CREB3-ARF4 (show ARF4 ELISA Kits) signalling cascade may be part of a Golgi stress response set in motion by stimuli compromising Golgi capacity.
propose that JAB1 (show COPS5 ELISA Kits) is a novel binding partner of Luman, which negatively regulates the activity of Luman by promoting its degradation
GSK3beta (show GSK3b ELISA Kits) was downregulate in all samples and CREB3 did not show a significant decrease or increase in its mRNA expression, but the results were significant in mucoepidermoid carcinoma and salivary duct carcinoma.
Tissue transcription analysis revealed that both porcine CREB2 (show ATF2 ELISA Kits) and CREB3 mRNA were ubiquitously detected in all examined tissues.
Tisp40 (show CREB3L4 ELISA Kits) aggravates tubular cells apoptosis in renal ischemia reperfusion injury.
LUMAN is a key regulator of glucocorticoid receptor (show NR3C1 ELISA Kits)-mediated signaling.
these findings suggest that Tisp40 (show CREB3L4 ELISA Kits) plays a critical role in the TGF-beta (show TGFB1 ELISA Kits)/ Smads pathway involved in this process. Hence, Tisp40 (show CREB3L4 ELISA Kits) could be a useful therapeutic target in the fight against renal tubulointerstitial fibrosis
Luman regulates mouse granulosa cell modulation of steroid synthesis, cell cycle activity and other regulators of folliculogenesis
LRF (show ZBTB7A ELISA Kits) seems to be involved in the regulation of decidualization during pregnancy.
These results suggest that Luman regulates the multinucleation of osteoclasts by promoting cell fusion of mononuclear osteoclasts through DC-STAMP (show TM7SF4 ELISA Kits) induction and intracellular distribution during osteoclastogenesis.
Creb3l4 (show CREB3L4 ELISA Kits) is an essential negative regulator of adipogenesis.
sLZIP is a novel PPARgamma2 (show PPARG ELISA Kits) modulator for control of the balance between adipogenesis and osteogenesis during Mesenchymal stem cell differentiation
sLZIP negatively regulates skeletal muscle differentiation via interaction with ACTN4 (show ACTN4 ELISA Kits).
Luman might have important roles in embryo implantation and decidualization.
This gene encodes a transcription factor that is a member of the leucine zipper family of DNA binding proteins. This protein binds to the cAMP-response element and regulates cell proliferation. The protein interacts with host cell factor C1, which also associates with the herpes simplex virus (HSV) protein VP16 that induces transcription of HSV immediate-early genes. This protein and VP16 both bind to the same site on host cell factor C1. It is thought that the interaction between this protein and host cell factor C1 plays a role in the establishment of latency during HSV infection. This protein also plays a role in leukocyte migration, tumor suppression, and endoplasmic reticulum stress-associated protein degradation. Additional transcript variants have been identified, but their biological validity has not been determined.
cAMP responsive element binding protein 3
, leucine zipper gene 8
, basic leucine zipper protein
, cAMP-responsive element-binding protein 3
, cyclic AMP response element (CRE)-binding protein/activating transcription factor 1
, cyclic AMP-responsive element-binding protein 3
, leucin zipper proitein
, leucine zipper protein
, transcription factor LZIP-alpha
, cAMP responsive element-binding protein 3
, cAMP responsive element binding protein 3 (luman)
, transcription factor LZIP
, (attaching to CRE-like 1
, androgen-induced basic leucine zipper
, attaching to CRE-like 1
, cAMP-responsive element-binding protein 3-like protein 4
, cyclic AMP-responsive element-binding protein 3-like protein 4
, transcript induced in spermiogenesis protein 40