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Dog (Canine) Monoclonal TLR9 Primary Antibody for ELISA - ABIN4360433
Abel, Wang, Fritts, Sanchez, Chung, Fitzgerald-Bocarsly, Krieg, Miller et al.: Deoxycytidyl-deoxyguanosine oligonucleotide classes A, B, and C induce distinct cytokine gene expression patterns in rhesus monkey peripheral blood mononuclear cells and distinct alpha interferon ... in Clinical and diagnostic laboratory immunology 2005
Show all 49 Pubmed References
Dog (Canine) Monoclonal TLR9 Primary Antibody for CyTOF, ELISA - ABIN4360430
Gantner, Hermann, Nakashima, Matsukawa, Sakai, Bacon: CD40-dependent and -independent activation of human tonsil B cells by CpG oligodeoxynucleotides. in European journal of immunology 2003
Show all 116 Pubmed References
Dog (Canine) Monoclonal TLR9 Primary Antibody for FACS, ICC - ABIN4360434
Dillmann, Ringel, Ringleb, Mora, Olesch, Fink, Roberts, Brüne, Weigert: S1PR4 Signaling Attenuates ILT 7 Internalization To Limit IFN-α Production by Human Plasmacytoid Dendritic Cells. in Journal of immunology (Baltimore, Md. : 1950) 2016
Show all 56 Pubmed References
Dog (Canine) Monoclonal TLR9 Primary Antibody for FACS - ABIN4360435
Wang, Nicholas, Conway, Sen, Diz, Tisch, Clarke: EBV latent membrane protein 2A induces autoreactive B cell activation and TLR hypersensitivity. in Journal of immunology (Baltimore, Md. : 1950) 2006
Show all 57 Pubmed References
Dog (Canine) Monoclonal TLR9 Primary Antibody for CyTOF, ELISA - ABIN4360432
Damiano, Caputo, Bianco, DArmiento, Leonardi, De Placido, Bianco, Agrawal, Ciardiello, Tortora: Novel toll-like receptor 9 agonist induces epidermal growth factor receptor (EGFR) inhibition and synergistic antitumor activity with EGFR inhibitors. in Clinical cancer research : an official journal of the American Association for Cancer Research 2006
Show all 52 Pubmed References
Human Monoclonal TLR9 Primary Antibody for FACS, IF - ABIN2191963
Ahmad-Nejad, Häcker, Rutz, Bauer, Vabulas, Wagner: Bacterial CpG-DNA and lipopolysaccharides activate Toll-like receptors at distinct cellular compartments. in European journal of immunology 2002
Show all 8 Pubmed References
Human Monoclonal TLR9 Primary Antibody for FACS, IF - ABIN2191962
Rumio, Besusso, Palazzo, Selleri, Sfondrini, Dubini, Ménard, Balsari: Degranulation of paneth cells via toll-like receptor 9. in The American journal of pathology 2004
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Human Monoclonal TLR9 Primary Antibody for FACS, IF - ABIN2191964
Rutz, Metzger, Gellert, Luppa, Lipford, Wagner, Bauer: Toll-like receptor 9 binds single-stranded CpG-DNA in a sequence- and pH-dependent manner. in European journal of immunology 2004
Show all 8 Pubmed References
Human Monoclonal TLR9 Primary Antibody for FACS - ABIN4360413
Leifer, Kennedy, Mazzoni, Lee, Kruhlak, Segal: TLR9 is localized in the endoplasmic reticulum prior to stimulation. in Journal of immunology (Baltimore, Md. : 1950) 2004
Show all 5 Pubmed References
Mouse (Murine) Polyclonal TLR9 Primary Antibody for IHC - ABIN967189
Hemmi, Takeuchi, Kawai, Kaisho, Sato, Sanjo, Matsumoto, Hoshino, Wagner, Takeda, Akira: A Toll-like receptor recognizes bacterial DNA. in Nature 2000
Show all 5 Pubmed References
sickle red blood cell induced TLR9, NLRP3 (show NLRP3 Antibodies), Caspase-1 (show CASP1 Antibodies), IL-1beta (show IL1B Antibodies) and IL-18 (show IL18 Antibodies) expression and induced IL-1beta (show IL1B Antibodies), LTB4 (show PTGR1 Antibodies) and nitrite production in PBMC cultures.
Findings indicate that genotype TT in the -1486 locus of the TLR9 gene could comprise a risk genotype for HPV infection, squamous intraepithelial cervical lesion, and uterine cervical neoplasm in Mexican female population.
data show that TLR-5 (show TLR5 Antibodies) and TLR-9 are susceptible genes to lupus nephritis (LN) and that their expression is dysregulated in LN patients' kidneys, supporting a role of these mediators in the pathogenesis of LN.
By CRISPR/Cas9-induced inactivation of TLR9, MyD88 (show MYD88 Antibodies), IRAK4 (show IRAK4 Antibodies) and IRAK1 (show IRAK1 Antibodies) we confirm that BZLF1 repression is dependent on functional TLR9 and MyD88 (show MYD88 Antibodies) signaling, and identify IRAK4 (show IRAK4 Antibodies) as an essential element for TLR9-induced repression of BZLF1 expression upon BCR (show BCR Antibodies) cross-linking
results imply that TLR9-mediated activation of B-cells not only promotes cell survival, but may via p53 (show TP53 Antibodies) provide cells with a barrier against harmful consequences of enhanced activation and proliferation
mutually exclusive transcriptional regulation by AP-1 (cjun (show JUN Antibodies)/cfos) and non-canonical NF-kappaB (show NFKB1 Antibodies) (RelB (show RELB Antibodies)/p52 (show FKBP4 Antibodies)) downstream of MEK (show MAP2K1 Antibodies)-ERK (show EPHB2 Antibodies) and NIK (show MAP3K14 Antibodies)-IKK-alpha (show CHUK Antibodies)-NF-kappaB2 (p100 (show CUX1 Antibodies)) phosphorylation, respectively was responsible for persistent Ccl20 (show CCL20 Antibodies) expression in the colonic cells.
This study evaluation the roles of SOCS1 (show SOCS1 Antibodies), the regulator of TLR9, RIG-I (show DDX58 Antibodies), and CD152 (show CTLA4 Antibodies) in patients with liver fibrosis/cirrhosis; the use of polymorphisms as markers for genetic risk is reported.
cathelicidin selectively modulated synthesis of TLR4 (show TLR4 Antibodies) and 9 in intestinal epithelium, but only when cells were exposed to virulence factors, mostly from apical surfaces.
Tumor TLR9 expression is not associated with prognosis in African American triple negative breast cancer . Significant differences were detected in TLR9 variant MAFs between European Americans and African American . They may affect TLR9 expression and function.
Early endosomal TLR9 activation is important for MR1 (show MR1 Antibodies)-mediated bacterial antigen presentation in B cells.
TLR9 expression on B1a cells is not critical for their IgM-dependent atheroprotection.
This study establishes a correlation between TLR-3 (show TLR3 Antibodies) and TLR-9 expression with the development of EAE. In addition, evidence of a role for the myelin peptide in targeting the innate inflammatory response to the CNS is presented.
these data demonstrate that TLR7 (show TLR7 Antibodies)/TLR9 ligands push the macrophage into a phagocytic long-lived cell, with a decreased capacity of antigen presentation and reminiscent of the M2 polarized state.
TLR9/MyD88 (show MYD88 Antibodies) signaling selectively in CD11c (show ITGAX Antibodies)(+) dendritic cells (DCs) strongly enhances murine cytomegalovirus clearance.
High-glucose induces tau hyperphosphorylation through activation of TLR9-P38 MAPK (show MAPK14 Antibodies) pathway.
proposed that TLR9 regulates the NF-kappaB (show NFKB1 Antibodies)-NLRP3 (show NLRP3 Antibodies)-IL-1beta (show IL1B Antibodies) pathway negatively in Salmonella-induced NKG2D (show KLRK1 Antibodies)-mediated intestinal inflammation and plays a critical role in defense against S. typhimurium infection and in the protection of intestinal integrity.
mechanism integrating BCR, TLR9, and cytokine signals provides a peripheral checkpoint for DNA-containing antigens that, if circumvented by survival and differentiative cues, yields B cells with the autoimmune-associated T-bet+ phenotype.
we demonstrated for the first time that the cross-talk of TLR2 (show TLR2 Antibodies) and TLR9 triggered Th1 (show HAND1 Antibodies) activation collaboratively
the cooperative role of TLR9 with TLR2 (show TLR2 Antibodies) or TLR6 (show TLR6 Antibodies) receptors in sensing Brucella, was determined.
accelerating effects of Tlr9 deficiency
TLR9 immunoreactivity is mainly detected in epithelial cells and antigen presenting cells, namely dendritic and macrophage-like cells, within the range of tissues examined. The pattern of TLR9 expression was similar in pigs of 3 weeks and 3 months of age.
These data demonstrated that TLR2 (show TLR2 Antibodies), TLR3 (show TLR3 Antibodies) and TLR9 contribute to NF-kappaB (show NFKB1 Antibodies) activation in response to porcine epidemic diarrhea virus infection, but not RIG-I (show DDX58 Antibodies).
expression of TLR3 (show TLR3 Antibodies), TLR7 (show TLR7 Antibodies) and TLR9 in alveolar macrophages infected with different genotype 1 PRRSV strains
we studied TLR9 expression in lung extracts from pigs, dogs, and cattle.
Moreover, the TLR9 transfectant demonstrated its usefulness for evaluation of immunostimulation by bacterial DNA through the detection of T(H)-1, T(H)-2 type cytokine induction via TLR9 signaling.
variants in the TLR1 (show TLR1 Antibodies) gene are associated with susceptibility to bovine tuberculosis, whereas no significant association can be inferred from the polymorphisms in the TLR9 gene
mRNA abundances of TLR9, TLR2, and TLR4 together with those of beta-defensin 5 (BNBD5), an early bactericidal effector molecule of the innate system, in healthy and infected mammary glands
substantial conservation of TLR9 structure and TLR9 function in blood leukocytes
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns (PAMPs) that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This gene is preferentially expressed in immune cell rich tissues, such as spleen, lymph node, bone marrow and peripheral blood leukocytes. Studies in mice and human indicate that this receptor mediates cellular response to unmethylated CpG dinucleotides in bacterial DNA to mount an innate immune response.
Toll-like receptor 9 protein