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anti-Human TLR9 Antibodies:
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Dog (Canine) Monoclonal TLR9 Primary Antibody for EIA, IHC (fro) - ABIN1106225
Ahmad-Nejad, Häcker, Rutz, Bauer, Vabulas, Wagner: Bacterial CpG-DNA and lipopolysaccharides activate Toll-like receptors at distinct cellular compartments. in European journal of immunology 2002
Show all 7 references for ABIN1106225
Dog (Canine) Monoclonal TLR9 Primary Antibody for EIA, IHC (fro) - ABIN1106226
Rutz, Metzger, Gellert, Luppa, Lipford, Wagner, Bauer: Toll-like receptor 9 binds single-stranded CpG-DNA in a sequence- and pH-dependent manner. in European journal of immunology 2004
Show all 7 references for ABIN1106226
Human Polyclonal TLR9 Primary Antibody for IHC (fro), WB - ABIN951281
Takeda, Kaisho, Akira: Toll-like receptors. in Annual review of immunology 2003
Show all 5 references for ABIN951281
Human Monoclonal TLR9 Primary Antibody for IF, WB - ABIN393403
Selvaraj, Harishankar, Singh, Jawahar, Banurekha: Toll-like receptor and TIRAP gene polymorphisms in pulmonary tuberculosis patients of South India. in Tuberculosis (Edinburgh, Scotland) 2010
Show all 5 references for ABIN393403
Human Monoclonal TLR9 Primary Antibody for WB - ABIN2668281
Kitzinger: Letter from England--birth plans. in Birth (Berkeley, Calif.) 1992
Human Polyclonal TLR9 Primary Antibody for FACS, WB - ABIN657643
Fiola, Gosselin, Takada, Gosselin: TLR9 contributes to the recognition of EBV by primary monocytes and plasmacytoid dendritic cells. in Journal of immunology (Baltimore, Md. : 1950) 2010
Cow (Bovine) Polyclonal TLR9 Primary Antibody for WB - ABIN2777826
Ramirez-Ortiz, Specht, Wang, Lee, Bartholomeu, Gazzinelli, Levitz: Toll-like receptor 9-dependent immune activation by unmethylated CpG motifs in Aspergillus fumigatus DNA. in Infection and immunity 2008
These findings demonstrate differential reaction of cholinesterase-targeting miRNAs to distinct TLR9 challenges, indicating upstream miRNA co-regulation of the intestinal alternative NFkappaB pathway and cholinergic signaling.
Toll-like receptor 9 mediates OTSCC invasion and migration in vitro and is an independent prognostic factor of OTSCC.
no association between genetic polymorphism the hyper-IgE syndrome phenotype
Report increased TLR9 expression in endosomes from patients with metabolic syndrome.
clinical outcome in malaria was observed to be apparently modulated by LD between TLR9 promoter variants.
IRF5 (show IRF5 Antibodies) and IRF8 (show IRF8 Antibodies), two transcription factors with opposing functions, control TLR9 signaling in human plasmacytoid dendritic cells.
activated ARF3 (show ARF3 Antibodies) is associated with Unc93B1 (show UNC93B1 Antibodies) and TLR9, suggesting that ARF3 (show ARF3 Antibodies) conducts TLR9 trafficking by forming the TLR9-Unc93B1 (show UNC93B1 Antibodies)-ARF3 (show ARF3 Antibodies) complex.
NADPH oxidase (show NOX1 Antibodies)-deficient patient-derived B cells also expressed enhanced levels of TLR7 (show TLR7 Antibodies) and TLR9 mRNA and protein compared with the same cells reconstituted to restore oxidase activity.
Data show that differences in codon bias limit Toll-like receptor 7 (TLR7 (show TLR7 Antibodies)) expression relative to Toll-like receptor 9 (TLR9).
Results indicate that heterodimerization of GCA (show NPR1 Antibodies) and TLR9 is important for TLR9-mediated downstream signaling and might serve to fine tune processes against viral infection.
In mice, nonalcoholic steatohepatitis development in response to a high-fat diet required TLR9 on lysozyme (show LYZ Antibodies)-expressing cells
TLR9 activation at high and low CpG ODN concentrations has roles in acute and chronic vascular injury in mice
The current study demonstrates for the first time an immunomodulatory role for TLR9 in acute kidney injury by enhancing the capacity of Tregs to migrate to damaged tissue in a model of common and relevant kidney disease.
recognition of beta-1,3 glucan by Dectin-1 (show CLEC7A Antibodies) triggers TLR9 trafficking to beta-1,3 glucan-containing phagosomes, which may be critical in coordinating innate antifungal defense.
Sustained activation of Toll-like receptor 9 causes cardiac and systemic inflammation, and deterioration of SERCA2a (show ATP2A2 Antibodies) depletion-mediated diastolic heart failure.
TLR9 signaling is involved in regulating adipose tissue inflammation and controlling obesity and the metabolic syndrome.
Genetic deletion of MyD88 (show MYD88 Antibodies), TLR2 (show TLR2 Antibodies) or TLR9 effectively controlled the signs of intestinal injury when compared with irinotecan-administered WT controls
AP-3 (show AP3B1 Antibodies) recruitment to TLR9 endosomes was impaired by PIKfyve (show PIKFYVE Antibodies) inhibition.
TLR9 immunoreactivity is mainly detected in epithelial cells and antigen presenting cells, namely dendritic and macrophage-like cells, within the range of tissues examined. The pattern of TLR9 expression was similar in pigs of 3 weeks and 3 months of age.
These data demonstrated that TLR2 (show TLR2 Antibodies), TLR3 (show TLR3 Antibodies) and TLR9 contribute to NF-kappaB (show NFKB1 Antibodies) activation in response to porcine epidemic diarrhea virus infection, but not RIG-I (show DDX58 Antibodies).
expression of TLR3 (show TLR3 Antibodies), TLR7 (show TLR7 Antibodies) and TLR9 in alveolar macrophages infected with different genotype 1 PRRSV strains
we studied TLR9 expression in lung extracts from pigs, dogs, and cattle.
Moreover, the TLR9 transfectant demonstrated its usefulness for evaluation of immunostimulation by bacterial DNA through the detection of T(H)-1, T(H)-2 type cytokine induction via TLR9 signaling.
variants in the TLR1 (show TLR1 Antibodies) gene are associated with susceptibility to bovine tuberculosis, whereas no significant association can be inferred from the polymorphisms in the TLR9 gene
mRNA abundances of TLR9, TLR2, and TLR4 together with those of beta-defensin 5 (BNBD5), an early bactericidal effector molecule of the innate system, in healthy and infected mammary glands
substantial conservation of TLR9 structure and TLR9 function in blood leukocytes
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns (PAMPs) that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This gene is preferentially expressed in immune cell rich tissues, such as spleen, lymph node, bone marrow and peripheral blood leukocytes. Studies in mice and human indicate that this receptor mediates cellular response to unmethylated CpG dinucleotides in bacterial DNA to mount an innate immune response.
Toll-like receptor 9 protein