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Human TLR9 Primary Antibody for ELISA - ABIN414673
Yang, Xie, Deng, Qin: Expression of soluble Toll-like receptors in pleural effusions. in Chinese medical journal 2010
Show all 3 references for ABIN414673
Rat (Rattus) TLR9 Primary Antibody for ELISA - ABIN368321
Tunc, Aydemir, Karaoglu, Cekmez, Kul, Aydinoz, Babacan, Yaman, Sarici: Toll-like receptor levels and caffeine responsiveness in rat pups during perinatal period. in Regulatory peptides 2013
The data of this study demonstrated that TLR9 mRNAs is notably expressed in glioma tissues than in normal tissues.
analysis of TLR2 (show TLR2 ELISA Kits), TLR4 (show TLR4 ELISA Kits) and TLR9 genes and their significance as biological markers in patients with B-cell chronic lymphocytic leukemia
We found lower expression levels of TLR1 (show TLR1 ELISA Kits), TLR3 (show TLR3 ELISA Kits), TLR4 (show TLR4 ELISA Kits), TLR7 (show TLR7 ELISA Kits) and TLR9 in PBMCs from patients with ALL compared with those from control patients. We also observed that the PBMCs from patients with Pre-B and B ALL had lower TLR4 (show TLR4 ELISA Kits) expression than controls
TLR-stimulated pancreatic cancer cells were specifically investigated for activated signaling pathways of VEGF (show VEGFA ELISA Kits)/PDGF (show PDGFA ELISA Kits) and anti-apoptotic Bcl-xL (show BCL2L1 ELISA Kits) expression as well as tumor cell growth.
The results suggest that high TLR9 expression in pancreatic ductal adenocarcinoma indicates improved prognosis
interferon alpha (IFN-alpha (show IFNA ELISA Kits)) secretion induced by Toll-like receptor 7 (show TLR7 ELISA Kits) and Toll-like receptor 8 (show TLR8 ELISA Kits) (TLR7 (show TLR7 ELISA Kits)/TLR8 (show TLR8 ELISA Kits)) activation was observed in common variable immunodeficiency (CVID (show TNFRSF13B ELISA Kits)), which was recovered with Toll-like receptor 9 (TLR9) signaling.
Data show that the heterozygous genotypes of toll like receptor 9 (TLR9)-1486T/C and 2848C/T single nucleotide polymorphisms (SNPs) as well as the homozygous AA genotype of the intronic 1174G/A SNP were prevalent in infants with human cytomegalovirus (HCMV) infection.
On stimulation of TLR9, CDK8 (show CDK8 ELISA Kits)/19 positively regulates inflammatory gene transcription in cooperation with NF-kappaB (show NFKB1 ELISA Kits) and C/EBPbeta (show CEBPB ELISA Kits).
The results showed that the TLR9 expression in the mRNA and the protein level was significantly higher in PBMCs from Systemic lupus erythematosus (SLE) patients. However, IFN-alpha (show IFNA ELISA Kits) concentration in patients and controls significantly increased in response to CpG stimulation but this increase was significantly higher in healthy controls compared with SLE patients
individuals with the AA genotype were associated with an increased risk of bacterial meningitis compared with those with the GG genotype.the AA genotype was correlated with an elevated risk of bacterial meningitis compared with the GG+GA genotype.the results of our study suggest an association between the TLR9+TLR9+2848 polymorphism and a reduced risk of bacterial meningitis in the codominant and recessive models
this paper shows that endosomal retention of TLR9 via the interaction of IRAP (show IL1RN ELISA Kits) with the actin cytoskeleton is a mechanism that prevents hyper-activation of TLR9 in dendritic cells
data demonstrated for the first time that NK cells directly recognize Baculovirus via TLR9, which provides opportunities for the use of this technique as an effective tool for Baculovirus-based immunotherapies against malignancies.
3-Hydroxy-4,7-megastigmadien-9-one, isolated from Ulva pertusa, attenuates TLR9-mediated inflammatory response by down-regulating mitogen-activated protein kinase (show MAPK1 ELISA Kits) and NF-kappaB (show NFKB1 ELISA Kits) pathways.
Results demonstrate that nerve stimulation of TLR9 knock-out muscles produces weaker response than when the muscle is stimulated directly; TLR9 knock-out mice have functional and morphological alterations of the neuromuscular junction
Munc13-4 (show UNC13D ELISA Kits) regulates endosome-initiated, TLR9-dependent signaling and TLR9-specific cellular functions in neutrophils.
TLR9 expression showed dynamic changes for a long period of time and microglias were the main brain cells to express TLR9 after cerebral ischemia and reperfusion.
These findings demonstrate differential reaction of cholinesterase-targeting miRNAs to distinct TLR9 challenges, indicating upstream miRNA co-regulation of the intestinal alternative NFkappaB pathway and cholinergic signaling.
activated ARF3 (show ARF3 ELISA Kits) is associated with Unc93B1 (show UNC93B1 ELISA Kits) and TLR9, suggesting that ARF3 (show ARF3 ELISA Kits) conducts TLR9 trafficking by forming the TLR9-Unc93B1 (show UNC93B1 ELISA Kits)-ARF3 (show ARF3 ELISA Kits) complex.
In mice, nonalcoholic steatohepatitis development in response to a high-fat diet required TLR9 on lysozyme (show LYZ ELISA Kits)-expressing cells
TLR9 activation at high and low CpG ODN concentrations has roles in acute and chronic vascular injury in mice
TLR9 immunoreactivity is mainly detected in epithelial cells and antigen presenting cells, namely dendritic and macrophage-like cells, within the range of tissues examined. The pattern of TLR9 expression was similar in pigs of 3 weeks and 3 months of age.
These data demonstrated that TLR2 (show TLR2 ELISA Kits), TLR3 (show TLR3 ELISA Kits) and TLR9 contribute to NF-kappaB (show NFKB1 ELISA Kits) activation in response to porcine epidemic diarrhea virus infection, but not RIG-I (show DDX58 ELISA Kits).
expression of TLR3 (show TLR3 ELISA Kits), TLR7 (show TLR7 ELISA Kits) and TLR9 in alveolar macrophages infected with different genotype 1 PRRSV strains
we studied TLR9 expression in lung extracts from pigs, dogs, and cattle.
Moreover, the TLR9 transfectant demonstrated its usefulness for evaluation of immunostimulation by bacterial DNA through the detection of T(H)-1, T(H)-2 type cytokine induction via TLR9 signaling.
variants in the TLR1 (show TLR1 ELISA Kits) gene are associated with susceptibility to bovine tuberculosis, whereas no significant association can be inferred from the polymorphisms in the TLR9 gene
mRNA abundances of TLR9, TLR2 (show TLR2 ELISA Kits), and TLR4 (show TLR4 ELISA Kits) together with those of beta-defensin 5 (BNBD5 (show DEFB105A ELISA Kits)), an early bactericidal effector molecule of the innate system, in healthy and infected mammary glands
substantial conservation of TLR9 structure and TLR9 function in blood leukocytes
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns (PAMPs) that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This gene is preferentially expressed in immune cell rich tissues, such as spleen, lymph node, bone marrow and peripheral blood leukocytes. Studies in mice and human indicate that this receptor mediates cellular response to unmethylated CpG dinucleotides in bacterial DNA to mount an innate immune response.
Toll-like receptor 9 protein