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Human TLR9 ELISA Kit for Sandwich ELISA - ABIN414673
Yang, Xie, Deng, Qin: Expression of soluble Toll-like receptors in pleural effusions. in Chinese medical journal 2010
Show all 3 references for ABIN414673
Rat (Rattus) TLR9 ELISA Kit - ABIN368321
Tunc, Aydemir, Karaoglu, Cekmez, Kul, Aydinoz, Babacan, Yaman, Sarici: Toll-like receptor levels and caffeine responsiveness in rat pups during perinatal period. in Regulatory peptides 2013
The G + 1174A single nucleotide polymorphism in the toll receptor 9 gene may contribute to the genetic susceptibility and manifictation of Systemic lupus erythematosus in Egyptian patients
this study shows no changes at epidermal expression of TLR9 in pemphigus and bullous pemphigoid (show DST ELISA Kits) lesions
The presence of a variant T allele in a common SNP (rs352140) in the TLR9 gene whose product recognizes bacterial DNA is associated with increased Placental inflammation.
The TLR9 rs352140 polymorphism may be associated with the susceptibility of female PD in Chinese Han population.
High expression of Toll-like receptor 9 is associated with the progression of cervical neoplasia.
de novo expressed TLR9 may utilize endogenous mtDNA as the ligand to facilitate podocyte apoptosis, a novel mechanism underlying podocyte injury in glomerular diseases.
Our meta-analysis suggests that TLR7 (show TLR7 ELISA Kits), TLR8 (show TLR8 ELISA Kits), and TLR9 polymorphisms are associated with the development of systemic lupus erythematosus in Caucasian, Asian, and African populations
Review/Meta-analysis: TLR9 rs187084 polymorphism may increase the risk of systemic lupus erythematosus in Asians.
TLR9 expression seems lower in individuals with deep venous thrombosis (DVT) and residual thrombosis (RT), albeit not significant. Interestingly, TLR9 might play a role in recurrent DVT, as the TLR9 expression was significantly higher in patients with recurrent DVT.
Increased expression of TLR7 (show TLR7 ELISA Kits) and TLR9 in myasthenia gravis thymus is accompanied by active Epstein-Barr virus infection.
Results demonstrate that nerve stimulation of TLR9 knock-out muscles produces weaker response than when the muscle is stimulated directly; TLR9 knock-out mice have functional and morphological alterations of the neuromuscular junction
Munc13-4 (show UNC13D ELISA Kits) regulates endosome-initiated, TLR9-dependent signaling and TLR9-specific cellular functions in neutrophils.
TLR9 expression showed dynamic changes for a long period of time and microglias were the main brain cells to express TLR9 after cerebral ischemia and reperfusion.
These findings demonstrate differential reaction of cholinesterase-targeting miRNAs to distinct TLR9 challenges, indicating upstream miRNA co-regulation of the intestinal alternative NFkappaB pathway and cholinergic signaling.
activated ARF3 (show ARF3 ELISA Kits) is associated with Unc93B1 (show UNC93B1 ELISA Kits) and TLR9, suggesting that ARF3 (show ARF3 ELISA Kits) conducts TLR9 trafficking by forming the TLR9-Unc93B1 (show UNC93B1 ELISA Kits)-ARF3 (show ARF3 ELISA Kits) complex.
In mice, nonalcoholic steatohepatitis development in response to a high-fat diet required TLR9 on lysozyme (show LYZ ELISA Kits)-expressing cells
TLR9 activation at high and low CpG ODN concentrations has roles in acute and chronic vascular injury in mice
The current study demonstrates for the first time an immunomodulatory role for TLR9 in acute kidney injury by enhancing the capacity of Tregs to migrate to damaged tissue in a model of common and relevant kidney disease.
recognition of beta-1,3 glucan by Dectin-1 (show CLEC7A ELISA Kits) triggers TLR9 trafficking to beta-1,3 glucan-containing phagosomes, which may be critical in coordinating innate antifungal defense.
Sustained activation of Toll-like receptor 9 causes cardiac and systemic inflammation, and deterioration of SERCA2a (show ATP2A2 ELISA Kits) depletion-mediated diastolic heart failure.
TLR9 immunoreactivity is mainly detected in epithelial cells and antigen presenting cells, namely dendritic and macrophage-like cells, within the range of tissues examined. The pattern of TLR9 expression was similar in pigs of 3 weeks and 3 months of age.
These data demonstrated that TLR2 (show TLR2 ELISA Kits), TLR3 (show TLR3 ELISA Kits) and TLR9 contribute to NF-kappaB (show NFKB1 ELISA Kits) activation in response to porcine epidemic diarrhea virus infection, but not RIG-I (show DDX58 ELISA Kits).
expression of TLR3 (show TLR3 ELISA Kits), TLR7 (show TLR7 ELISA Kits) and TLR9 in alveolar macrophages infected with different genotype 1 PRRSV strains
we studied TLR9 expression in lung extracts from pigs, dogs, and cattle.
Moreover, the TLR9 transfectant demonstrated its usefulness for evaluation of immunostimulation by bacterial DNA through the detection of T(H)-1, T(H)-2 type cytokine induction via TLR9 signaling.
variants in the TLR1 (show TLR1 ELISA Kits) gene are associated with susceptibility to bovine tuberculosis, whereas no significant association can be inferred from the polymorphisms in the TLR9 gene
mRNA abundances of TLR9, TLR2 (show TLR2 ELISA Kits), and TLR4 (show TLR4 ELISA Kits) together with those of beta-defensin 5 (BNBD5 (show DEFB105A ELISA Kits)), an early bactericidal effector molecule of the innate system, in healthy and infected mammary glands
substantial conservation of TLR9 structure and TLR9 function in blood leukocytes
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns (PAMPs) that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This gene is preferentially expressed in immune cell rich tissues, such as spleen, lymph node, bone marrow and peripheral blood leukocytes. Studies in mice and human indicate that this receptor mediates cellular response to unmethylated CpG dinucleotides in bacterial DNA to mount an innate immune response.
Toll-like receptor 9 protein