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Human TLR9 ELISA Kit for Sandwich ELISA - ABIN414673
Yang, Xie, Deng, Qin: Expression of soluble Toll-like receptors in pleural effusions. in Chinese medical journal 2010
Show all 3 Pubmed References
Rat (Rattus) TLR9 ELISA Kit - ABIN368321
Tunc, Aydemir, Karaoglu, Cekmez, Kul, Aydinoz, Babacan, Yaman, Sarici: Toll-like receptor levels and caffeine responsiveness in rat pups during perinatal period. in Regulatory peptides 2013
Tumor TLR9 expression is not associated with prognosis in African American triple negative breast cancer . Significant differences were detected in TLR9 variant MAFs between European Americans and African American . They may affect TLR9 expression and function.
Early endosomal TLR9 activation is important for MR1 (show MR1 ELISA Kits)-mediated bacterial antigen presentation in B cells.
This study provides new insight into the mechanism that mediates TLR9 upregulation in response to cellular stresses. In addition, it shows that HPV38 E6 and E7 are able to interfere with this mechanism.
In conclusion, our data provide strong indirect evidence that TLR9 might play a greater role in HCV infection than previously expected. We identified an association of the polymorphism rs187084 within the TLR9 gene with the natural course of HCV infection in women.
this study shows that the double CpG motif sequence-specificity of human TLR9 results in decreased activation by oligodeoxyribonucleotides
Patients with low TLR9 expression have longer survival rates with supratentorial glioblastoma compared to patients with higher expression rates.
The effect could be blocked by chloroquine (CQ), a TLR9 inhibitor.
mechanism integrating BCR, TLR9, and cytokine signals provides a peripheral checkpoint for DNA-containing antigens that, if circumvented by survival and differentiative cues, yields B cells with the autoimmune-associated T-bet+ phenotype.
These results indicate that TLR9 activation via the cag island may modify the risk for malignancy within the context of H. pylori infection and provide an important framework for future studies investigating the microbial-epithelial interface in gastric carcinogenesis.
TLR9 2848 G/A single nucleotide polymorphism may be associated with human cytomegalovirus infection in pregnant women.
High-glucose induces tau hyperphosphorylation through activation of TLR9-P38 MAPK (show MAPK14 ELISA Kits) pathway.
proposed that TLR9 regulates the NF-kappaB (show NFKB1 ELISA Kits)-NLRP3 (show NLRP3 ELISA Kits)-IL-1beta (show IL1B ELISA Kits) pathway negatively in Salmonella-induced NKG2D (show KLRK1 ELISA Kits)-mediated intestinal inflammation and plays a critical role in defense against S. typhimurium infection and in the protection of intestinal integrity.
we demonstrated for the first time that the cross-talk of TLR2 (show TLR2 ELISA Kits) and TLR9 triggered Th1 (show HAND1 ELISA Kits) activation collaboratively
the cooperative role of TLR9 with TLR2 (show TLR2 ELISA Kits) or TLR6 (show TLR6 ELISA Kits) receptors in sensing Brucella, was determined.
accelerating effects of Tlr9 deficiency
Increased circulating mtDNA combined with upregulated TLR9 expression may corporately play a role in EAM as well as TLR4 (show TLR4 ELISA Kits) activation mediated cardiac inflammation and injury
Data show that inoculating mice deficient in toll (show TLR4 ELISA Kits)-like receptors TLR4 (show TLR4 ELISA Kits) or TLR9 with LPS (show TLR4 ELISA Kits) or CpG-stimulated eosinophils, NK cell recruitment was observed alongside cytotoxicity.
Significantly lower levels of interferon (show IFNA ELISA Kits)-stimulated gene expression in response to purified curli-DNA in TLR2 (show TLR2 ELISA Kits) and TLR9 deficient mice compared to wild-type mice.
these data provide a new perspective on the complexity of TLR9 regulation by proteolytic cleavage
TLR9 immunoreactivity is mainly detected in epithelial cells and antigen presenting cells, namely dendritic and macrophage-like cells, within the range of tissues examined. The pattern of TLR9 expression was similar in pigs of 3 weeks and 3 months of age.
These data demonstrated that TLR2 (show TLR2 ELISA Kits), TLR3 (show TLR3 ELISA Kits) and TLR9 contribute to NF-kappaB (show NFKB1 ELISA Kits) activation in response to porcine epidemic diarrhea virus infection, but not RIG-I (show DDX58 ELISA Kits).
expression of TLR3 (show TLR3 ELISA Kits), TLR7 (show TLR7 ELISA Kits) and TLR9 in alveolar macrophages infected with different genotype 1 PRRSV strains
we studied TLR9 expression in lung extracts from pigs, dogs, and cattle.
Moreover, the TLR9 transfectant demonstrated its usefulness for evaluation of immunostimulation by bacterial DNA through the detection of T(H)-1, T(H)-2 type cytokine induction via TLR9 signaling.
variants in the TLR1 (show TLR1 ELISA Kits) gene are associated with susceptibility to bovine tuberculosis, whereas no significant association can be inferred from the polymorphisms in the TLR9 gene
mRNA abundances of TLR9, TLR2 (show TLR2 ELISA Kits), and TLR4 (show TLR4 ELISA Kits) together with those of beta-defensin 5 (BNBD5 (show DEFB105A ELISA Kits)), an early bactericidal effector molecule of the innate system, in healthy and infected mammary glands
substantial conservation of TLR9 structure and TLR9 function in blood leukocytes
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns (PAMPs) that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This gene is preferentially expressed in immune cell rich tissues, such as spleen, lymph node, bone marrow and peripheral blood leukocytes. Studies in mice and human indicate that this receptor mediates cellular response to unmethylated CpG dinucleotides in bacterial DNA to mount an innate immune response.
Toll-like receptor 9 protein