Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species and application
anti-Human TCF4 Antibodies:
anti-Mouse (Murine) TCF4 Antibodies:
anti-Rat (Rattus) TCF4 Antibodies:
Go to our pre-filtered search.
Human Monoclonal TCF4 Primary Antibody for IF, ELISA - ABIN520754
Tanaka, Itoh, Nishiyama, Takezawa, Kurihara, Itoh, Kato: Inhibition of endothelial cell activation by bHLH protein E2-2 and its impairment of angiogenesis. in Blood 2010
Show all 3 Pubmed References
Human Monoclonal TCF4 Primary Antibody for IHC (p), IP - ABIN264393
Barker, Huls, Korinek, Clevers: Restricted high level expression of Tcf-4 protein in intestinal and mammary gland epithelium. in The American journal of pathology 1999
Show all 5 Pubmed References
Human Monoclonal TCF4 Primary Antibody for EMSA, IHC (p) - ABIN264394
Denys, Jadidizadeh, Amini Nik, Van Dam, Aerts, Alman, Cassiman, Tejpar: Identification of IGFBP-6 as a significantly downregulated gene by beta-catenin in desmoid tumors. in Oncogene 2004
Show all 2 Pubmed References
Human Polyclonal TCF4 Primary Antibody for IF, IP - ABIN2476691
Valenta, Lukas, Doubravska, Fafilek, Korinek: HIC1 attenuates Wnt signaling by recruitment of TCF-4 and beta-catenin to the nuclear bodies. in The EMBO journal 2006
Human Monoclonal TCF4 Primary Antibody for IF, WB - ABIN393964
Eichhoff, Weeraratna, Zipser, Denat, Widmer, Xu, Kriegl, Kirchner, Larue, Dummer, Hoek: Differential LEF1 and TCF4 expression is involved in melanoma cell phenotype switching. in Pigment cell & melanoma research 2011
Human Polyclonal TCF4 Primary Antibody for IHC, IHC (p) - ABIN4358095
Saegusa, Hashimura, Kuwata: Sox4 functions as a positive regulator of β-catenin signaling through upregulation of TCF4 during morular differentiation of endometrial carcinomas. in Laboratory investigation; a journal of technical methods and pathology 2012
GRG5/AES (show AES Antibodies) interacts with TCF4 (show TCF7L2 Antibodies) and represses Wnt (show WNT2 Antibodies)-mediated transcription both in human cells and zebrafish embryos.
Data indicte that Tcf-1 (show HNF1A Antibodies) and Lef-1 (show LEF1 Antibodies) exhibit a function in the axis induction assay, which is lacking in Tcf-3 (show TCF3 Antibodies) and -4.
Tcf4 (show TCF7L2 Antibodies) transcription factor cooperates in patterning the Xenopus brain.
The rs7903146 (C/T) polymorphism of the TCF7L2 (show TCF7L2 Antibodies) gene might not be associated with obesity in the Cameroonian population.
These findings suggest that the rs7903146 locus might exert its enhancer function by interacting with HMGB1 (show HMGB1 Antibodies) in an allele dependent manner.
TCF7L2 (show TCF7L2 Antibodies) polymorphism is associated with colorectal cancer.
Data show that FERM domain-containing protein 5 (FRMD5) is regulated by both beta-catenin and transcription factor 7-Like 2 protein (TCF7L2) in colon cancer cells.
Altered DNA methylation (show HELLS Antibodies) in TCF4 involves in the etiology of Bipolar disorder and Major Depressive disorder .
We found that the genotype "AG" of rs9320010 and "GA" of rs7235757 decreased SCZ risk. In the genetic model analysis, we also observed that the allele "A" of rs9320010 and "G" of rs7235757 were inversely related with the risk of SCZ in the dominant model. Our study indicated that rs9320010, rs7235757, and rs1452787 were prominently associated with SCZ.
Our work suggests that DM1 (show DMPK Antibodies) patients are at risk for Fuchs' endothelial corneal dystrophy (FECD (show COL8a2 Antibodies)). DMPK (show DMPK Antibodies) mutations contribute to the genetic burden of FECD (show COL8a2 Antibodies) but are uncommon. We establish a connection between two repeat expansion disorders converging upon RNA-MBNL1 (show MBNL1 Antibodies) foci and FECD (show COL8a2 Antibodies).
the variation of the TCF7L2 (show TCF7L2 Antibodies) gene in three Amerindian populations
These data define the mechanism responsible for the repressive effects of nitric oxide (NO) on the transcriptional activity of beta-catenin (show CTNNB1 Antibodies) and link eNOS (show NOS3 Antibodies)-derived NO to the modulation by VEGF (show VEGFA Antibodies) of Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies)-induced endothelial cell proliferation.
we show that TopoIIalpha forms protein-protein interactions with beta-catentin and TCF4 and interacts with Wnt (show WNT2 Antibodies) response elements (WREs) and promoters of direct target genes of TCF (show HNF4A Antibodies) transcription, including: MYC (show MYC Antibodies), vimentin (show VIM Antibodies), AXIN2 (show AXIN2 Antibodies) and LEF1 (show LEF1 Antibodies)
We report that TCF4 comprises two transcriptional isoforms, both of which are required for optimal plasmacytoid DC development in vitro
these data identified E2A (show TCF3 Antibodies) and E2-2 as central regulators of B cell immunity.
down-regulation of Id3 (show ID3 Antibodies) in B cells is essential for releasing E2A (show TCF3 Antibodies) and E2-2, which in a redundant manner are required for antigen-induced B cell differentiation.
TCF7L2 (show TCF7L2 Antibodies) mediates canonic Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) signaling and c-Myc (show MYC Antibodies) upregulation during abnormal cardiac remodeling in heart failure and suppression of Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) to c-Myc (show MYC Antibodies) axis can be explored for preventing and treating heart failure.
TCF-4 as a co-activator of p65 (show NFkBP65 Antibodies) in the potentiation of proinflammatory cytokine production in macrophages and aggravation of high-fat diet induced chronic inflammation and insulin (show INS Antibodies) resistance in mice.
These findings suggest a unique role for Tcf7l2 (show TCF7L2 Antibodies) in generating distinct neuronal phenotypes from homogeneous progenitor population.
Tcf7l2 (show TCF7L2 Antibodies) may be involved in maintenance of stem/progenitor cells properties.
Data indicate that upregulation of E2-2 protein markedly attenuated the inhibitor of DNA-binding 1 (ID1 (show ID1 Antibodies))-mediated increase in endothelial progenitor cells (EPCs) proliferation and migration.
results indicate that miR (show MLXIP Antibodies)-181a-5p promotes 3T3-L1 preadipocyte differentiation and adipogenesis through regulating TGFbeta (show TGFB1 Antibodies)/Smad (show SMAD1 Antibodies) and Wnt (show WNT2 Antibodies) signaling pathway by directly targeting Smad7 (show SMAD7 Antibodies) and Tcf7l2 (show TCF7L2 Antibodies)
Tcf7l2 (show TCF7L2 Antibodies) protein levels decline upon initiation of endocrine differentiation in vivo, disclosing the downregulation of this factor in the developing endocrine compartment.
This gene encodes a high mobility group (HMG) box-containing transcription factor that plays a key role in the Wnt signaling pathway. The protein has been implicated in blood glucose homeostasis. Genetic variants of this gene are associated with increased risk of type 2 diabetes. Several transcript variants encoding multiple different isoforms have been found for this gene.
transcription factor 7-like 2 (T-cell specific, HMG-box)
, transcription factor 4
, transcription factor 4-like
, helix-loop-helix transcription factor
, SL3-3 enhancer factor 2
, class B basic helix-loop-helix protein 19
, immunoglobulin transcription factor 2
, thyroglobulin promoter transcription factor TFE
, transcriptional factor 4
, Transcription factor 4 (Immunoglobulin transcription factor 2) (ITF-2) (MITF-2) (SL3-3 enhancer factor 2) (SEF-2) (Class A helix-loop-helix transcription factor ME2)
, class A helix-loop-helix transcription factor ME2
, immunoglobulin transcription factor-2
, R8f DNA-binding protein
, HMG box transcription factor 4
, T-cell factor 4
, T-cell-specific transcription factor 4
, transcription factor 7-like 2
, transcription factor 7-like 2, T-cell specific, HMG-box
, transcription factor Tcf4
, T-cell factor-4 variant A
, T-cell factor-4 variant B
, T-cell factor-4 variant C
, T-cell factor-4 variant D
, T-cell factor-4 variant E
, T-cell factor-4 variant F
, T-cell factor-4 variant G
, T-cell factor-4 variant H
, T-cell factor-4 variant I
, T-cell factor-4 variant J
, T-cell factor-4 variant K
, T-cell factor-4 variant L
, T-cell factor-4 variant M
, T-cell factor-4 variant X2