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Human WNT3A ELISA Kit for Sandwich ELISA - ABIN572397
Lin, Ueng, Niu, Yuan, Yang, Chen, Lee, Chen: Effects of hyperbaric oxygen on the osteogenic differentiation of mesenchymal stem cells. in BMC musculoskeletal disorders 2014
Show all 7 Pubmed References
Human WNT3A ELISA Kit for Sandwich ELISA - ABIN837302
Shim, Park, Lee, Kang, Lee, Shin: Human dermal stem/progenitor cell-derived conditioned medium ameliorates ultraviolet a-induced damage of normal human dermal fibroblasts. in PLoS ONE 2013
These data define the mechanism responsible for the repressive effects of nitric oxide (NO) on the transcriptional activity of beta-catenin (show CTNNB1 ELISA Kits) and link eNOS (show NOS3 ELISA Kits)-derived NO to the modulation by VEGF (show VEGFA ELISA Kits) of Wnt (show WNT2 ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits)-induced endothelial cell proliferation.
These results indicate that PEDF (show SERPINF1 ELISA Kits) counters Wnt (show WNT2 ELISA Kits) signaling to allow for osteoblast differentiation and provides a mechanistic insight into how the PEDF (show SERPINF1 ELISA Kits) null state results in OI type VI.
These results suggest novel mechanisms for Wnt3a-induced osteoblast proliferation and cell survival via Npnt (show NPNT ELISA Kits) gene expression
this study shows that the Wnt3a expression levels in dendritic cells influences the generation of memory T cells after 5 days in co-culture with naive T cells through activation of the Wnt (show WNT2 ELISA Kits) canonical pathway
Wnt3a induces Osx (show SP7 ELISA Kits) expression via p38 MAPK (show MAPK14 ELISA Kits) signaling in dental follicle cells. Wnt3a-induced Osx (show SP7 ELISA Kits) expression was inhibited in the presence of p38 mitogen-activated protein kinase (show MAPK14 ELISA Kits) (MAPK (show MAPK1 ELISA Kits)) inhibitors (SB203580 and SB202190) at gene and protein levels, as assessed by real-time PCR and immunocytohistochemistry, respectively.
Furthermore, pigment epithelium-derived factor (PEDF (show SERPINF1 ELISA Kits)), a secreted glycoprotein known for its anti-tumor properties, blocked Wnt3a-directed induction of autophagy proteins. Autophagy inhibition was complemented by reciprocal regulation of the oxidative stress enzymes, superoxide dismutase 2 (SOD2 (show SOD2 ELISA Kits)) and catalase (show CAT ELISA Kits).
Ginkgo biloba exocarp extract inhibits tumor angiogenesis, which may be closely relevant to its effect in blockage of Wnt /beta-catenin-VEGF signaling pathway in Lewis lung carcinoma.
Western blot analysis demonstrated that following BMP2 (show BMP2 ELISA Kits) and BMP7 (show BMP7 ELISA Kits) cotransfection of MC3T3E1 cells, the protein expression levels of BMP2 (show BMP2 ELISA Kits), BMP4 (show BMP4 ELISA Kits), BMP6 (show BMP6 ELISA Kits), BMP7 (show BMP7 ELISA Kits), BMP9 (show GDF2 ELISA Kits) and Wnt3a were increased compared with control cells
Wnt3a promotes macrophage-mediated bacterial killing by elevating CRAMP and BD1 (show DEFB1 ELISA Kits) levels.
Wnt3a acutely reduces nuclear acetyl-CoA (show LPCAT1 ELISA Kits), the necessary substrate for histone acetyltransferases, resulting in a global decrease in histone acetylation.
PEGylated Wnt3A liposomes associated with skeletal stem cell populations in human bone marrow and promoted osteogenesis.
The AChE plays a role in osteoblastic differentiation and is regulated by both Wnt3a and Runx2 (show RUNX2 ELISA Kits).
In promoting the self-renewal symmetric division of hTERT(high) prostate cancer cells, WNT3a dramatically decreased the ratio of hTERT(high) prostate cancer cells undergoing asymmetric division. Increased WNT (show WNT2 ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits) signal activation was also detected in hTERT(high) prostate cancer cells. hTERT-mediated CSC properties were at least partially dependent on beta-catenin (show CTNNB1 ELISA Kits).
the results indicate neighboring structural elements within full-length Wnt3a affect saposin-like subdomain (SLD) conformational stability. Moreover, SLD function(s) in Wnt (show WNT2 ELISA Kits) proteins appear to have evolved away from those commonly attributed to SAPLIP family members.
Data suggest that PORCN (show PORCN ELISA Kits) exhibits substrate specificity that includes a Wnt3a peptide fragment (residues 199-219, with disulfide bonds); recombinant PORCN (show PORCN ELISA Kits) containing a point mutation (R228C) associated with focal dermal hypoplasia exhibits impaired acylation activity toward Wnt3a peptide fragment. (PORCN (show PORCN ELISA Kits) = porcupine (show PORCN ELISA Kits) O-acyltransferase; Wnt3a = Wnt (show WNT2 ELISA Kits) family member 3A)
CPE (show CPE ELISA Kits) through its N'-terminal sequence, forms aggregates with Wnt3a and possible endoplasmic reticulum (ER) stress leading to its loss of function.
Our findings suggest that Pyk2 (show PTK2B ELISA Kits) plays an important role in the coordination of stabilization of beta-catenin (show CTNNB1 ELISA Kits) in the crosstalk between Wnt (show WNT2 ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits) and Wnt (show WNT2 ELISA Kits)/Ca(2 (show CA2 ELISA Kits)+) signaling pathways upon Wnt3a stimulation in differentiating hNPCs.
Macrophage polarization seems to have a key role in the progression of pediatric non-alcoholic fatty liver disease; the modulation of macrophage polarization could drive hepatic progenitor cell response by Wnt3a production and beta-catenin (show CTNNB1 ELISA Kits) phosphorylation.
this study shows that Wnt3a promotes differentiation of dendritic cells, but inhibits their maturation
Taken together, our results suggest that canonical Wnt signaling and its antagonist, sFRP1, regulate proliferation of human CSCs. Furthermore, excess sFRP1 in elderly patients causes CSC aging.
Data suggest that Wnt3, Wnt3a, and Wnt8a (show WNT8A ELISA Kits) bind to their respective receptors (Fz8 (show FZD2 ELISA Kits), Lrp6 (show LRP5 ELISA Kits), and Lypd6 (show LYPD6 ELISA Kits)) in ordered plasma membrane environments; ordered plasma membrane environments appear to be essential for binding of Wnt (show WNT2 ELISA Kits) proteins to their receptor complexes and stimulation of downstream signaling activity.
It was shown that Wnt3a-Wnt8a (show WNT8A ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits) signaling directly regulates ciliogenic transcription factor foxj1a expression and ciliogenesis in zebrafish Kupffer's vesicle.
In zebrafish embryos lacking Wnt3a, Wnt1 (show WNT1 ELISA Kits) and Wnt10b (show WNT10B ELISA Kits), the expression of engrailed orthologs, pax2a and fgf8 (show FGF8 ELISA Kits) is not maintained after mid-somitogenesis
data suggest a specific role for Wnt3a in the development of cardiac NCCs; propose that this function of wnt3a in r6 is partially mediated by crip2 (show CRIP2 ELISA Kits) expression in the premigratory cardiac NCCs, which subsequently affects cardiac function and PA patterning
Sulf1 (show SULF1 ELISA Kits) does not affect Wnt3a-mediated activation of canonical Wnt (show WNT2 ELISA Kits) signaling.
Wnt3a protein alone is sufficient to rescue the severe loss of inner ear structures resulting from dorsal but not ventral half ablations.
hindbrain-repressive Wnt3a/Meis3/Tsh1 circuit promotes neuronal differentiation and coordinates tissue maturation
Wnt3a thus acts downstream of FAK (show PTK2 ELISA Kits) to balance anterior-posterior cell fate specification in the developing neural plate
Data suggest a new model for neural anteroposterior patterning, in which Wnt3a from the paraxial mesoderm induces posterior cell fates via direct activation of a crucial transcription factor in the overlying neural plate.
Wnt3A secretion from tectal cells along with ephrin-B1 (show EFNB1 ELISA Kits) signaling are specifically responsible for enhanced neural responses in the developing optic tectum.
The WNT gene family consists of structurally related genes which encode secreted signaling proteins. These proteins have been implicated in oncogenesis and in several developmental processes, including regulation of cell fate and patterning during embryogenesis. This gene is a member of the WNT gene family. It encodes a protein which shows 96% amino acid identity to mouse Wnt3A protein, and 84% to human WNT3 protein, another WNT gene product. This gene is clustered with WNT14 gene, another family member, in chromosome 1q42 region.
wingless-type MMTV integration site family, member 3A
, protein Wnt-3a-like
, protein Wnt-3a
, vestigial tail
, wingless-type MMTV integration site family, member 3 like
, wnt3 like
, wingless-type MMTV integration site family, member 3
, Wnt-3a homolog
, Wnt3a variant 3
, wingless-type MMTV integration site family member 3a
, wingless-related MMTV integration site 3A